Titlebook: Arterial Mesenchyme and Arteriosclerosis; William D. Wagner,Thomas B. Clarkson Book 1974 The Editor(s) (if applicable) and The Author(s),

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https://doi.org/10.1007/978-3-658-22398-4subintimal media below plaques. The lipid accumulations commonly occur intracellularly as well as extracellularly. As many morphological studies have shown, the increased amounts of intracellular lipids are deposited mainly in proliferated and modified smooth muscle cells (Constantinides, 1965; Scot

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https://doi.org/10.1007/978-3-476-04136-4in emphasis was on observation of cell growth and an overall fascination with the technique. During the second phase of 15 to 20 years the emphasis was on growing large numbers of cells, producing cell populations for quantitative work, and studying neoplastic transformation. Methods were developed

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https://doi.org/10.1007/978-3-658-05626-17; McGill and Geer, 1963; Wissler, 1967). Other cells, such as hematogenously derived macrophages, have been found within the lesions, but there is little evidence that they play a principal part in lesion development. Numerous observations of human lesions and of lesions produced in experimental an

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Einheiten und Symbole für Ingenieureharides, collagen, and elastic tissue. Varying amounts of intracellular and extracellular lipids are present. In the late phase of the disease the lesion is complicated by necrosis, hemorrhage, and calcification (Daoud et al., 1964; Haust et al., 1960; Geer et al., 1961). The role of the extracellul