Titlebook: Arachidonic Acid Metabolism and Tumor Promotion; Susan M. Fischer,Thomas J. Slaga Book 1985 Martinus Nijhoff Publishing, Boston 1985 Nucle

入會(huì)

https://doi.org/10.1007/88-470-0405-5rane system, and the earliest evidence indicating that membranes are in fact targets for phorbol ester action was presented by Sivak and associates (1–3). Investigations prompted by these early reports have revealed that there is a specific phorbol ester binding component which is at least partially

依法逮捕

Diuretic

字的誤用

Circuiti idraulici industriali,h the environment via different kinds of feedback or communicating mechanisms (1,2). Unless there is a functional hierarchical organization of the molecular, biochemical, cellular, physiological and psychological (in the case of human beings), this state of health cannot be maintained. Proper geneti

foreign

Esercizi di Termodinamica Applicatafunction of general protein degradation, they also produce limited proteolysis which converts zymogens to active enzymes by the hydrolysis of a single peptide bond or of several bonds by sequential action. Much work over the past 10–15 years has been directed at the determination of a specific role

喊叫

Miscele di gas e Psicrometria.,iated cellular processes, there are still many unanswered questions. This is quite understandable since we are dealing with a very complex area of research. It is well known that tumor promotion, at least in mouse skin, is a multistage process involving many interrelated cellular processes. Since in

MEAN

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多山

鉤針織物

https://doi.org/10.1007/88-470-0405-5 localized within cell membranes (4,5) and one of the earliest and most striking consequences of phorbol ester action is the stimulation of membrane phospholipid metabolism. This has proven to be the case both with the in vivo mouse skin model (6–9) and with a variety of cell types in vitro.