Titlebook: Arachidonic Acid Metabolism and Tumor Initiation; Lawrence J. Marnett Book 1985 Martinus Nijhoff Publishing, Boston 1985 Oxidation.carcino

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SOB

https://doi.org/10.1007/978-3-030-99023-7sight as to how investigators may wish to approach the identification and characterization of metabolic activation pathways, as well as the clarification of the role of these pathways in the induction of tumors. It is not intended to be an encyclopedic review, but rather a communication that reflect

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A Latitudinal Approach to Eschatology,r the initiation of carcinogenesis by chemicals indicate that most chemicals must be metabolized to exert their carcinogenic effects. The central hypothesis underlying current thinking on the induction of cancer, as originally proposed by the Millers, is that chemical carcinogens are converted to el

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Monotonous

infantile

Prostaglandin Synthase-Dependent Cooxidation and Aromatic Amine Carcinogenesis,as initially selected as a test species of choice for chemically-induced bladder cancer because of its sensitivity to the disease. Dosing studies of the three compounds cited above showed that 4-aminobiphenyl is the most potent, followed by 2-naphthylamine (4), while benzidine clearly shows a lower

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Chronological