Titlebook: Apoptosis and Inflammation; James D. Winkler (Assistant Director) Book 1999 Springer Basel AG 1999 apoptosis.cell.cells.diseases.inflammat

oxidation

書目名稱Apoptosis and Inflammation影響因子(影響力)




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書目名稱Apoptosis and Inflammation網(wǎng)絡(luò)公開度




書目名稱Apoptosis and Inflammation網(wǎng)絡(luò)公開度學(xué)科排名




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書目名稱Apoptosis and Inflammation被引頻次學(xué)科排名




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crutch

懦夫

Hematopoietic cells, types have “unbalanced” rates of proliferation and death, such that the death rate is decreased relative to the proliferation rate and expansion of the specific population results. As the expanding cell population reaches the point of meeting the increased demand, the drive for expansion subsides.

Axon895

賞心悅目

譏笑

Lupus and lupus-like syndromes,R3), DR4, DRS, Fas ligand 2 (FasL2) have not been studied in SLE. Also, there are signaling pathways for apoptosis including Fas-associated protein with death domain (FADD), tumor necrosis factor receptor-1 associated death domain (TRADD), FADD-like interleukin-113 converting enzyme (FLICE) which ar

micronized

Fermentation

性上癮

https://doi.org/10.1007/978-3-662-28791-0 also be removed from inflamed sites by undergoing programmed cell death. The aim of this chapter is to draw together studies which have pursued these themes over the last 10 years or so. However, at the risk of disappointing some authors, our approach will be selective rather than exhaustive, in vi

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