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Titlebook: Antithrombotics; Pathophysiological R Arnold G. Herman (Professor of Pharmacology) Book 1991 Springer Science+Business Media Dordrecht 1991

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發(fā)表于 2025-3-21 18:34:14 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
期刊全稱Antithrombotics
期刊簡(jiǎn)稱Pathophysiological R
影響因子2023Arnold G. Herman (Professor of Pharmacology)
視頻videohttp://file.papertrans.cn/159/158686/158686.mp4
學(xué)科分類Developments in Cardiovascular Medicine
圖書封面Titlebook: Antithrombotics; Pathophysiological R Arnold G. Herman (Professor of Pharmacology) Book 1991 Springer Science+Business Media Dordrecht 1991
Pindex Book 1991
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書目名稱Antithrombotics影響因子(影響力)




書目名稱Antithrombotics影響因子(影響力)學(xué)科排名




書目名稱Antithrombotics網(wǎng)絡(luò)公開度




書目名稱Antithrombotics網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Antithrombotics被引頻次




書目名稱Antithrombotics被引頻次學(xué)科排名




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Role of Nitric Oxide in Endothelial Cell - Platelet Interactions, of the soluble guanylate cyclase in the vascular system is part of the control mechanisms which regulate haemostasis and counteract thrombosis. An impaired production of NO may result in platelet activation and thrombosis. In contrast, a prolonged activation of constitutive and/or the expression of
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地板
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Thrombosis in Relation to Atherosclerosis, thrombosis on an arterial surface leads to a swift and striking proliferation of intimai smooth musde cells and an increase in the extracellular matrix proteins which these cells produce. Smooth muscle proliferation is almost certainly mediated by binding of component growth factors such as PDGF to
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Thrombosis in Relation to Atherosclerosis,raphy or naked-eye examination of post-mortem samples..Angiographicaly identifiable thrombi are found in a very high proportion of cases of acute myocardial ischaemia, and the morphological characteristics of any coronary artery plaques which are likely to lead to such thrombosis are briefly described.
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3.1 Differential cross sections, myocardial infarction, are associated with a prethrombotic state consisting of large megakaryocytes containing increased amounts of DNA, and large hyperreactive platelets. The cause for these changes in thrombogenesis require further study; such knowledge will aid the formulation of pharmacological agents capable of modulating the process.
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