Titlebook: Antirheumatic Therapy: Actions and Outcomes; Richard O. Day,Daniel E. Fürst,Barry Bresnihan Book 2005 Birkh?user Basel 2005 Arthritis.auto

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Tetracyclines,Although an infectious cause of RA has not been demonstrated, the role of the tetracycline family in treating RA has been re-examined due to the discovery of the anti-inflammatory and immunomodulatory effects of these agents.

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Medicinal chemistry of the disease modifying anti-rheumatic drugs,ll drugs, the chemical properties of the DMARD and corticosteroids are important aspects of their pharmacology. In this chapter, the medicinal chemistry of the various DMARD is discussed. The coverage includes the chemical factors that affect their handling by the body. In addition to the DMARD disc

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Targeting DMARD therapy,heir patients are faced with a variety of therapeutic choices in the management of RA and must make decisions about specific disease-modifying antirheumatic drugs (DMARDs), including potential combinations of different agents. A careful balance of short-term and long-term benefits and risks must be

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Hydroxychloroquine,e bark of the Peruvian cinchocha tree. Along with quinacrine, the two aminoquilones are labeled antimalarials after their long history in the treatment of that disease, highlighted by Pelletier and Caventou’s isolation of quinine and cinchonine as active antimalarial agents in 1820. Quinacrine, thou

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Sulfasalazine,c drugs based on notions of pathogenesis. She tried to link an antibacterial to an antiinflammatory agent in order to achieve simultaneous elimination of a putative infectious organism causing rheumatoid arthritis (RA) and suppression of inflammation. A number of compounds were developed and the one

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Parenteral gold, infections and, from the 1920s, also for rheumatoid arthritis (RA), believed to be caused by strep-tococcal infection. In 1935 Forestier reported over 550 cases, of whom 70–80% experienced remission or at least transient inactivation of the disease for 2–3 years, and Hartfall reported a clear impro

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