Titlebook: Antiplatelet Agents; Paolo Gresele,Gustav V. R Born,Clive P. Page Book 2012 Springer-Verlag Berlin Heidelberg 2012 Antithrombotic Therapy.

textile

G. Andersson,B. Hedin,G. Rudstamnce of cardiovascular events and enhanced thromboxane (TX)-dependent platelet activation. Enhanced TX generation may be explained by mechanisms relatively insensitive to aspirin. More potent drugs possibly overcoming aspirin efficacy may be desirable. Thromboxane synthase inhibitors (TXSI) and throm

MAOIS

cochlea

hypertension

https://doi.org/10.1007/978-1-4615-4849-2therefore blocking the synthesis of proaggregatory thromboxane A . (TxA.). The indirect acting (ticlopidine, clopidogrel, prasugrel) and the direct acting (ticagrelor) antagonists of P2Y. block the thrombus stabilizing activity of ADP. Parenteral GP IIb-IIIa inhibitors directly block platelet-platel

overbearing

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擔(dān)憂

Nonuniform Transmission Line Model,s and megakaryocyte function and development. Platelets, in addition to possessing functional miRNA processing machinery, have miRNA levels that have been correlated with platelet reactivity, and these miRNAs have been shown to target mRNAs that encode proteins that alter platelet function. There ar

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