Titlebook: Angiotensin Receptors; Juan M. Saavedra,Pieter B. M. W. M. Timmermans Book 1994 Springer Science+Business Media New York 1994 Angiotensin

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Angiotensin Receptor Subtypes and Cerebral Blood Flow,n is secured by two separate vascular channels: the carotid arteries and the vertebrobasilar system These systems meet at the base of the brain in a vascular ring known as the circle of Willis.. Due to this complex anatomy, cerebral blood flow (CBF) can be maintained even if one of the vascular terr

Rankle

Angiotensin II Receptor Subtypes and Growth,blood pressure regulation and control of fluid volume.. Angiotensin II (ANG II) induces the expression of growth-related proto-oncogenes .. and .. Expression of growth factors like platelet-derived growth factor (PDGF). and extracellular matrix components like thrombospondin. and tenascin. is also i

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Lice692

Inhibiting the Effects of Angiotensin on Cardiovascular Hypertrophy,of ANG II increase blood pressure by a slow process, which may be due to the development of vascular hypertrophy.. Abnormal vascular smooth muscle cell growth, which consists of hypertrophy and/or hyperplasia, is a characteristic feature in arteries from hypertensive patients and animals.. This cond

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Angiotensin Antagonists in Models of Heart Failure,icular free wall.. The extent and location of the tissue loss is the major determinant in the subsequent depression of cardiac performance following the acute event.. If myocardial necrosis is extensive, i.e., encompassing greater than 40% of the ventricular free wall, left ventricular failure rapid

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Angiotensin II Receptor Antagonism in an Ovine Model of Heart Failure Comparison with ACE and Reninegard to the central role of the renin—angiotensin system (RAS), which is activated early in the course of heart failure. These findings are highlighted by recent studies that demonstrate improved survival in patients with heart failure and left ventricular dysfunction treated with angiotensin-conve

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Angiotensin Antagonists in Models of Heart Failure,ators with a new pharmacological tool to examine cardiovascular pathophysiology. Before the development of these compounds, saralasin was the only agent that could block the angiotensin receptor. The disadvantages of saralasin are that it is a partial agonist as well as an antagonist and that it is

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浮雕

Angiotensin Receptors and Renal Function,availability of specific receptor antagonists, a body of information is evolving to describe the specific function of these important receptors. Along with insights in the physiological actions of ANG II on these receptors, variability in the type and localization of ANG II receptors within the rena