Titlebook: Anaesthesia, Pain, Intensive Care and Emergency Medicine — A.P.I.C.E.; Proceedings of the 1 Antonino Gullo (Head) Conference proceedings 20

Condescending

Grenzen der Untersuchung und Ausblick,pontaneous breathing. These effects are secondary to an altered mechanical behaviour of the respiratory system. In addition, artificial ventilation is mostly undertaken in humans who are either anaesthetised or suffering from respiratory failure. Thus, most observations on the effects of artificial

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Commentary

https://doi.org/10.1007/978-3-658-21639-9’s safety, as suggested by the Society of Critical Care Medicine [1–4]. However, only recently have technological developments resulted in the availability of capnography at the patient’s bedside, to measure variations in carbon dioxide concentration (CO.) and tidal volume simultaneously, during the

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https://doi.org/10.1007/978-3-658-21639-9ed cardiogenic and non-cardiogenic lung edema. Whereas the first is predominantly based on increased hydrostatic filtration pressure and will not be discussed in the following chapter, the second is due to increased vascular permeability. The prototypic example of non-cardiogenic pulmonary edema is

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Kontrastierender Vergleich der Einzelf?llessociated with sinus node dysfunction include symptomatic sinus bradycardia, sinus arrest, sinoatrial block, and paroxysmal supraventricular tachycardia alternating with periods of bradycardia or even asystole. The definition of bradycardia varies from institution to institution, but it generally includes a rate less than 40–50 beats/min.

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