Titlebook: Amino Acid Neurotransmitters; Leslie L. Iversen,Susan D. Iversen,Solomon H. Snyd Book 1975 Springer Science+Business Media New York 1975 n

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Leslie L. Iversen,Susan D. Iversen,Solomon H. Snyd

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Jens Mae?e,Hanno Pahl,Jan Sparsam, Chap. 1, this volume). It was therefore surprising to find in the mid-1960s that the contention that GABA might fulfill the role of the inhibitory transmitter in the mammalian nervous system was still being hotly debated in reviews for by Krnjevi? (1964) and against by Curtis and Watkins (1965). L

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https://doi.org/10.1007/978-3-642-92412-5, but there is now both anatomical and physiological evidence that these “peptidergic” neurons participate in synaptic events in the CNS. However, direct proof that these peptide hormones are the transmitter substances released by neurosecretory cells has been limited.

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Biochemical Pharmacology of GABA in CNS,ossible role of this amino acid as an inhibitory synaptic transmitter in the CNS. The experimental approaches used have been essentially the following: (1) modifying the metabolism of GABA in the living animal by systemic, intracranial, or topical application of compounds and attempting to correlate

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FACT

Amino Acid Receptors in CNS. I. GABA and Glycine in Spinal Cord, extracted an inhibitory substance, factor I, from mammalian brain and subsequently (Bazemore ., 1957) showed that it contained .-aminobutyric acid (GABA). However, some active preparations of factor I do not contain GABA (McLennan, 1958). In some of the earliest studies (Honour and McLennan, 1960;

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Amino Acid Receptors in CNS. II. GABA in Supraspinal Regions, potentials and single cell recording techniques and by the application of drugs to single cells by microiontophoresis to have a powerful inhibitory action on the majority of neurons encountered in the more accessible parts of mammalian brain and spinal cord (Hayashi, 1956; Purpura ., 1959; Curtis a

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Excitatory Amino Acid Receptors in the Central Nervous System,ime that Hayashi (1954) first described the marked excitatory action of the latter compound on the general electrical activity of the cerebral cortex. Thereafter, the potent effect of these substances on single neurons of the spinal cord was demonstrated (Curtis ., 1960), and similar studies have no