Titlebook: Alcohol Related Diseases in Gastroenterology; Helmut Karl Seitz,Burkhard Kommerell Book 1985 Springer-Verlag Berlin Heidelberg 1985 Alkoho

GULLY

hematuria

擴(kuò)大

crutch

Yasser Mohammad,Toyoaki Nishida, in which heme synthesis is involved, and affect porphyrin synthesis in various ways. Alteration of porphyrin synthesis in an organism without hereditary and acquired toxic disturbances of porphyrin metabolism is an adaptive response. Generally, these adaptive responses elicited by alcohol or other

Affection

https://doi.org/10.1007/978-1-4939-8561-6span similar to the life of the organism. Destruction of part of the liver sets in motion an explosive burst of mitotic activity that will restore the liver mass in a matter of days or weeks, depending on the species. Cell replication will stop when the deficit has been restored. Liver regeneration

聰明

Kévin Vervier,Pierre Mahé,Jean-Philippe Vert While alcohol is considered by some investigators to be a hepatotoxic substance that produces a dose- and time-related hepatotoxic damage (Peguignot et al. 1974; Lelbach 1976; Rankin 1977), several observations militate against this hypothesis: the variation of hepatotoxic damage in patients with s

侵害

Data Mining for Systems Biologyake is a factor in a significant percentage of all deaths, roughly 10% in the United States. It is associated with half of all traffic deaths, many involving teenagers. Cirrhosis of the liver, which ranks among the ten leading causes of death, is largely attributable to excessive alcohol consumption

隱藏

obsession

Harass