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Titlebook: Age-associated Neurological Diseases; Lüder Deecke,Peter Dal-Bianco Book 1991 Springer-Verlag Wien 1991 Alzheimer.PET.Parkinson.SPECT.Synd

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發(fā)表于 2025-3-21 17:31:35 | 只看該作者 |倒序瀏覽 |閱讀模式
期刊全稱Age-associated Neurological Diseases
影響因子2023Lüder Deecke,Peter Dal-Bianco
視頻videohttp://file.papertrans.cn/152/151025/151025.mp4
學科分類Journal of Neural Transmission. Supplementa
圖書封面Titlebook: Age-associated Neurological Diseases;  Lüder Deecke,Peter Dal-Bianco Book 1991 Springer-Verlag Wien 1991 Alzheimer.PET.Parkinson.SPECT.Synd
影響因子The papers compiled in this supplementum are a selection of the best con- th tributions presented at the 19 Central-European Neurological Symposium (CNS 19) held on June 29 - July 1, 1989 in Vienna. The main topic of this conference was degenerative and age-associated neurological diseases. In recent decades life expectancy has dramatically increased, at least in the industrialized countries. This has led to extreme distortions of the so-called population pyramids that no longer look like such but begin to almost resemble cylinders. As a consequence of this "overaging" of the population, diseases that are associated with age have become much more common than before. It was thus more than reasonable to devote a congress of the CNS series to these important neurological diseases. The following fields of interest are covered: Age-associated memory impairment (AAMI), Alzheimer‘s and other dementias, Parkinson‘s disease and other movement disorders, stroke and others. Concerning the de men- tias, some papers deal with diagnosis employing neuro-imaging methods such as MRI, CT, PET and SPECT, others using electrophysiological methods. An important aspect in the early preclinical diagnosis
Pindex Book 1991
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Positron emission tomography in the differential diagnosis of organic dementias,metabolism are coupled, and since glucose is the dominant substrate of the brain’s energy metabolism, studies of glucose metabolism by PET of 2(18F)-fluoro-2-deoxy-D-glucose (FDG) are widely applied for investigating the participation of various brain systems in simple or complex stimulations and ta
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,Cytoskeleton pathology in Alzheimer’s disease and related disorders,ary it appears to be a general biochemical marker for disorders in the degradation of a variety of cytoskeletal and other cytoplasmic proteins. The pattern of affected cytoskeletal components is not specific of AD/SDAT tangles. Tau definitely is present also in PSP tangles and possibly in Pick bodie
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,Morphometry of the corpus callosum in normal aging and Alzheimer’s disease,tion program was implemented allowing objective, quantitative and reproducible data. The various parts of the corpus callosum showed a different pattern of changes in normal aging as compared to Alzheimer’s disease. In conclusion, in normal aging affects mostly the fronttemporal interhemispheric fib
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Clinical and epidemiological aspects of dementia in the elderly,f prevalence rates every 5 years. Analytic epidemiology so far failed to reliably detect risk factors for DAT other than age. This might depend on the difficulties encountered in the clinical diagnosis and differential diagnosis of dementia in the elderly, which are discussed with special reference
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Multiinfarct dementia,s current level of mental functioning and some higher level of intellectual function in the past. The recognition of early or mild cases is specially difficult. Out of epidemiological studies it has been shown that the incidence for multi-infarct dementia (MID) increases with age and is slightly hig
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,Galanthamine treatment in Alzheimer’s disease,nhibitor Galanthamine, 30 mg/day. Neuropsychological and social parameters were rated. This open clinical pilot-study showed no statistic significant change in neuropsychological test-results. However after 1 year treatment 6 patients are still taking the drug. According to their care-persons there
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