Titlebook: Advances in the Immunopathogenesis of Multiple Sclerosis; D. Gambi,P. A. Muraro,U. Ecari Conference proceedings 1999 Springer-Verlag Itali

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Marta Sabou,Dietmar Winkler,Stefan Bifflles are virtually absent. Heterodimeric MHC molecules are essential for the initiation, propagation and effector phases of antigen-specific immune responses. Endogenous and exogenous antigenic peptides are presented via MHC molecules to T lymphocytes to enable cognate interactions. While MHC molecul

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Richard Torkar,Robert Feldt,Carlo A. Furiaow pathological specificity of conventional, T2-weighted MRI inevitably limits its potential for defining the pathophysiology of MS [1], although enhancement on T1-weighted scans after the injection of gadolinium-DTPA (Gd) can be used as a reliable marker of blood-brain barrier (BBB) dysfunction [1]

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Catrien Notermans,Maya Turolla,Willy Jansening to demyelination and axonal loss, and containing autoreactive T cells and pathogenic non-antigen-specific mononuclear cells [1]. It is currently believed that CNS antigen-reactive T cells provide the organ specificity of the pathogenic process. These cells regulate the recirculation within the C

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Can There Be Religion Without Gender?mmune diseases and in particular demyelinating disease of the central nervous system (CNS) such as multiple sclerosis (MS). EAE is characterized by an autoimmune response against myelin antigens mediated mostly by T lymphocytes but also by macrophages and B cells [2]. Activated CD4. T cells mediate

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29 October 1998. The high standard of presentations prompted us to report them in extended form, to highlight recent pro- gress in the understanding of basic mechanisms sustaining MS immuno- pathogenesis. A central role in the possible mechanisms leading to myelin destruc- tion has been attributed to T lympho978-88-470-0067-4978-88-470-2269-0