Titlebook: Advances in Multiple Sclerosis and Experimental Demyelinating Diseases; Moses Rodriguez Book 2008 The Editor(s) (if applicable) and The Au

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雇傭兵

https://doi.org/10.1007/BFb0011119y and maintenance of axonal health. It occurs as a spontaneous regenerative response following demyelination in a range of pathologies including traumatic injury as well as primary demyelinating disease such as multiple sclerosis (MS). Experimental models of demyelination based on the use of toxins,

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Lecture Notes in Earth Sciencesfluence inflammation and neuron and glial function is being slowly unraveled. There is increasing evidence that estrogen, progesterone, and testosterone contain immune responses and influence damage repair in the nervous system. Hormones such as prolactin and vitamin D are being explored as immunomo

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Dick Hoogendoorn,Wim H. Rulkens Ph.D., and results in early permanent disability for most affected patients. A new autoantibody marker called neuromyelitis optica immunoglobulin G (NMO-IgG), which targets the water channel protein aquaporin-4, is highly specific for NMO. The marker has demonstrated that the NMO spectrum of disorders is

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