Titlebook: Advances in Hydrogen Sulfide Biology; Yi-Chun Zhu Book 2021 The Editor(s) (if applicable) and The Author(s), under exclusive license to Sp

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https://doi.org/10.1007/978-1-4471-6455-5ey physiological events including arterial blood flow, glomerular filtration, and electrolyte and water transport. Deficiency of H.S generation has been implicated in acute kidney injury brought on by ischemia, administration of nephrotoxic medications, and obstruction. A role for impaired H.S expre

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https://doi.org/10.1057/9781137385673diseases, particularly diabetes, obesity, metabolic syndrome, cardiovascular ailments, and cancers. Among numerous endogenous mediators, the gasotransmitter hydrogen sulfide (H.S) plays a central role in the maintenance of glucose and lipid homeostasis. Current evidence from both pharmacological stu

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Discourse Theory and Artistic Performances,ical effects in mammalian cells as revealed by studies showing important roles in the cardiovascular system, in cell signalling processes, post-translational modifications and in the immune system. Regarding the latter, using pharmacological and genetic approaches scientists have shown this molecule

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https://doi.org/10.1007/978-3-030-79446-0n inflammation has emerged. This chapter will discuss the involvement of H.S in various inflammatory diseases. Furthermore, the contribution of reactive oxygen species (ROS), adhesion molecules, and leukocyte recruitment in H.S-mediated inflammation will be discussed. The interrelationship of H.S wi

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Conclusion: Unsettling the Museum,oprotective activities. Increasing evidence also indicates that H.S can suppress the production of inflammatory mediators by immune cells, for example, T cells and macrophages. Inflammation is closely related to an immune response in several diseases such as rheumatoid arthritis (RA), multiple scler

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Theatrical Apparatuses of Disjunction,found to be members of mammalian gasotransmitters family. Both H.S and SO. are endogenously produced in sulfur-containing amino acid metabolic pathway in vivo. The enzymes catalyzing the formation of H.S are mainly CBS, CSE, and 3-MST, and the key enzymes for SO. production are AAT1 and AAT2. Endoge

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