Titlebook: Advances in Eicosanoid Research; C. N. Serhan,H. D. Perez Conference proceedings 2000 Springer-Verlag Berlin Heidelberg 2000 Anti-Inflamma

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https://doi.org/10.1007/978-3-642-68304-6) as the initial products of polyunsaturated fatty-acid oxygenation (Fig. 1). The enzyme that catalyzes this reaction is cyclo-oxygenase (COX)., and it also catalyzes the reduction of the hydroperoxide to an alcohol, thereby forming PGH. (Hamberg and Samuelsson 1973: Nugteren and Hazelhof 1973).

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https://doi.org/10.1007/3-540-56253-2Samuelsson 1983). Membrane-bound arachidonic acid is released by cytosolic phospholipase A. and is further metabolized into LTA. by 5-lipoxygenase. This highly unstable epoxide may undergo enzymatic hydrolysis into the dihydroxy acid LTB. or may be conjugated with glutathione to form LTC.. These two

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Multiple Stocks, Multiple Locations,mice was examined after treatment with interleukin-1β (IL-1), tumor necrosis factor α (TNF), acidic fibroblast growth factor (FGF) and phorbol ester (PMA). Compared with their wild-type (COX1./COX-2.) counterparts, COX-1. or COX-2. cells exhibited substantially enhanced expression of the remaining f

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Dimitrios S. Dendrinos,Michael Sonisrted to various types of leukotrienes (LTs; Samuelsson et al. 1987; Shimizu and Wolfe 1990; Serhan et al. 1996). Recent fiindings from our laboratories and others have shown that cytosolic phospholipase A. (cPLA.) plays a dominant role in arachidonate release in various cells (Bonventre et al. 1997;

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Chaos and Stability in Planetary Systems shigellosis) and chronic inflammatory diseases of the intestine (i.e., Crohn’s disease and ulcerative colitis) collectively referred to as inflammatory bowel diseases (IBDs; Yamada et al. 1995). The symptoms (diarrhea, cramping) associated with active intestinal inflammation can be quite debilitati

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Collapse of Tori in Dissipative Mappingsstimuli, such as growth factors, cytokines, chemokines and circulating hormones. Released AA is metabolized via the cyclo-oxygenase (COX 1 and COX 2), lipoxygenase (LOX) or P450-dependent epoxygenase pathways to generate eicosanoids. In addition to their normal biological activities, such as stimula

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https://doi.org/10.1007/978-3-662-04047-8Anti-Inflammatory Drugs; Asthma; Eicosanoide; Termination; cyclooxygenase; enzymes; metabolism; signal tran

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Advances in Eicosanoid Research978-3-662-04047-8Series ISSN 0947-6075

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