Titlebook: Advances in Down Syndrome Research; G. Lubec Book 2003 Springer-Verlag/Wien 2003 Alzheimer‘s disease.Chromosom.Down syndrome.Emotion.Micro

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Alexandra Jonas,Nicolai von Ondarzaus gene pairs. Functional annotation of these genes, based on literature reports and computationally-derived predictions, shows that a broad range of cellular processes are represented. A goal of Down syndrome research is to determine which of these processes are perturbed by overexpression of chrom

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Schriften zur Geldtheorie und Geldpolitik-like neuropathological changes. The molecular defects underlying the DS — phenotype may be due to overexpression of genes encoded on chromosome 21. This so-called gene dosage hypothesis is still controversial and demands systematic work on protein expression. A series of transcription factors (TF)

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Manfred Schartl,Julia Maria Erber-Schropports have been linking folate metabolism to DS and indeed, chromosome 21 even encodes for a specific folate carrier. The availability of brain tissue along with the advent of proteomics enabled us to identify and quantify C1-tetrahydrofolate synthase (THF-S), a key element in folate metabolism in br

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