Titlebook: Actin; Biophysics, Biochemi James E. Estes,Paul J. Higgins Book 1994 The Editor(s) (if applicable) and The Author(s), under exclusive licen

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Klen ?opi? Pucihar,Matja? Kljunts show significant re-organization. While “normal” cells with well-spread morphologies have numerous bundles of microfilaments, transformed cells with rounded morphologies show dispersed microfilament patterns.. These changes in the structure of actin cables, as well as in substrate adhesion, are c

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Gregory L. Ulmer,John Craig Freemanpment, neurite outgrowth, wound healing, inflammation, and cancer metastasis. The actin cytoskeleton in animal cells must also drastically rearrange in a cell-cycle dependent manner in order to perform cytokinesis. Actin microfilament dynamics are intimately involved in all of these common types of

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Salvatore Iaconesi,Oriana Persico 1991; Hynes, 1992). These complex, adhesion-related cellular processes are mediated through transmembrane contact receptors of the cadherin and integrin families of receptors (Takeichi, 1991; Hynes, 1992). In the cytoplasmic domain, these receptors interact with cytoskeletal plaque proteins such as

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https://doi.org/10.1007/978-3-319-06203-7i.e. basal or suprabasal) within the tissue .. Under non-perturbed conditions, the turnover rate within the epidermis is relatively slow and the keratinocytes exist primarily in a non-migratory mode with cells exhibiting attachment to the basement membrane and to other surrounding cells via families

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Advances in Experimental Medicine and Biologyhttp://image.papertrans.cn/a/image/144092.jpg