Titlebook: α–Gal and Anti–Gal; α1,3–Galactosyltrans Uri Galili,José Luis Avila Book 1999 Springer Science+Business Media New York 1999 Antigen.Transpl

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,Graves’ Disease as a Model for Anti-Gal Involvement in Autoimmune Diseases,bungen der Gutachterkommissionen und Schlichtungsstellen für die Statistikjahre 2007 und 2008, die auf den Pressekonferenzen der Bundes?rztekammer in Berlin den Jahren 2008 und 2009 ver?ffentlicht wurden, zeigen, dass sich die vorgebrachten Patientenvorwürfe am h?ufigsten auf die Durchführung einer

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https://doi.org/10.1007/978-3-211-74420-8ane signaling. Their glycans (sugar chains) protrude from the surface, and display considerable structural complexity. The glycan structures assembled in a given cell type are dictated in part by the subset of glycosyltransferases that are translationally expressed. As the relative levels of the var

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https://doi.org/10.1007/978-3-211-74420-8ty in their overall structure. The α1,3GT, which was simultaneously cloned from the cow (Joziasse et al., 1989) and the mouse (Larsen et al., 1989), was one of the earliest glycosyltransferases for which complete sequence information was available. Since then, over 40 distinct glycosyltransferase ge

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https://doi.org/10.1007/978-3-211-74420-8h first discovered in plant seeds, they also have been found in other vegetative tissues of the plant world (leaves, stems, bark, roots, tubers, etc.). In recent years, lectins have been isolated from microorganisms, insects, Crustacea, rodents, and most recently from various tissues of primates whe

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