Titlebook: Advances in Nitrate Therapy; Adam Schneeweiss,Marija Weiss Book 19881st edition Springer-Verlag Berlin Heidelberg 1988 Bluthochdruck.Herzf

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https://doi.org/10.1007/978-3-642-79592-3 treatment of chronic stable angina pectoris, but the indications for them have markedly expanded. They now include all forms of angina pectoris and myocardial ischemia, congestive heart failure and hypertensive emergencies. The beneficial effects of nitrates in all these conditions result from thei

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https://doi.org/10.1007/978-3-662-06303-3nin-angio- tensin system or other vasodilatory mechanisms. Although it has been suggested that some of the dilatory effect of nitrates is exerted by the release of vasodilating prostaglandins, most investigators believe that this is not the main mechanism of action. Thus it is generally accepted tha

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Archaeology in a Middle Countryechanism of their effect is unknown. On the contrary, several mechanisms, each of which contributes to the effect, have been described. However, the relative importance of these mechanisms is not known, and it is therefore difficult to specify the predominant mechanism.

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https://doi.org/10.1007/978-3-642-83130-0y improved the conservative therapy of unstable angina. These formulations, originally used only in coronary care units, are now used in general medical wards and emergencies rooms, without hemodynamic monitoring, and in some countries also in mobile coronary care units.

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Gravity Sensing by Higher Plantsc modality. Even at low doses nitrates produce venodilation, resulting in decreases in left and right ventricular filling pressures. At higher doses nitrates cause, in a majority of patients, a decrease in afterload, resulting in an increase in cardiac output. Nitrates are effective bothin acute and

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Mechanosensitive Ion Channels in Bacteriaesults from valvular diseases (particularly aortic and mitral stenosis) has been questioned by some investigators. Recent studies have revealed, however, that nitrates are useful in some of these patients.

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