派博傳思國際中心

標題: Titlebook: Retroviral Insertion and Oncogene Activation; Hsing-Jien Kung,Peter K. Vogt Book 1991 The Editor(s) (if applicable) and The Author(s), und [打印本頁]

作者: 巡洋    時間: 2025-3-21 18:45
書目名稱Retroviral Insertion and Oncogene Activation影響因子(影響力)




書目名稱Retroviral Insertion and Oncogene Activation影響因子(影響力)學科排名




書目名稱Retroviral Insertion and Oncogene Activation網(wǎng)絡公開度




書目名稱Retroviral Insertion and Oncogene Activation網(wǎng)絡公開度學科排名




書目名稱Retroviral Insertion and Oncogene Activation被引頻次




書目名稱Retroviral Insertion and Oncogene Activation被引頻次學科排名




書目名稱Retroviral Insertion and Oncogene Activation年度引用




書目名稱Retroviral Insertion and Oncogene Activation年度引用學科排名




書目名稱Retroviral Insertion and Oncogene Activation讀者反饋




書目名稱Retroviral Insertion and Oncogene Activation讀者反饋學科排名





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L. F. Lock,N. A. Jenkins,N. G. Copelando case studies shows the complexity of factors influencing migrants’ investment strategies for remittances, and critically appraise the “triple win” circular migration model. The case studies focus on Tunisian and Senegalese migrants who come to France on short-stay visas (students, seasonal workers
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P. N. Tsichlis,P. A. Lazoment on Tariffs and Trade (GATT) negotiations. At that time, most developing countries (the “South”) were pursuing protectionist import-substitution policies and calling for a “New International Economic Order” to redress the alleged unfairness of the international economic system, while remaining o
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ment on Tariffs and Trade (GATT) negotiations. At that time, most developing countries (the “South”) were pursuing protectionist import-substitution policies and calling for a “New International Economic Order” to redress the alleged unfairness of the international economic system, while remaining o
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Retroviral Insertion and Oncogene Activation978-3-642-76524-7Series ISSN 0070-217X Series E-ISSN 2196-9965
作者: FACET    時間: 2025-3-23 15:05
0070-217X ontrol over its own expression through nontranslated sequences in the long terminal repeat (L TR). Some retroviruses also code for nonstructural proteins that further regulate proviral expression. Integration changes the cell genome; it adds viral genes, and in the case of transducing retroviruses a
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Mutagenesis of the Mouse Germline Using Retroviruses,ertional mutagens. The presence of a transposable element within or near the mutant gene acts as a molecular tag which aids in cloning the mutated gene of interest. This approach has been used with great success in several organisms, most notably . (R. 1988).
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Mutagenesis of the Mouse Germline Using Retroviruses,utations in radiation-or chemical-treated mice (GREEN 1981). Although much has been learned from the study of these mutations, molecular analysis of the mutated genes has often been hampered by the lack of molecular access to them. To circumvent this problem, transposable elements can be used as ins
作者: SHOCK    時間: 2025-3-24 16:03
Insertional Mutagenesis in Mouse Mammary Tumorigenesis, genes provide a particularly good example of the theme of this volume: host cell oncogenes activated by insertion of proviral DNA. They belong especially to this group because most of them have been discovered by virtue of insertional activation, through the application of provirus tagging methods.
作者: 變色龍    時間: 2025-3-24 20:52
Feline Leukaemia Virus: Generation of Pathogenic and Oncogenic Variants,us-host interaction and generally occurs after a long latent period (H. et al. 1976; J. 1984). For these reasons, it has been customary to classify FeLV as a chronic leukaemogenic retrovirus and to place the acute transforming C-type retroviruses in a separate subclass. In our view, this classificat
作者: detach    時間: 2025-3-25 00:32
Virus-Host Interactions and the Pathogenesis of Murine and Human Oncogenic Retroviruses, the retroviruses interact with the host in a variety of ways. Thus, viral gene products interact with cellular proteins and modulate or subvert their function. Furthermore, the proviral DNA integrates into the cellular genome introducing mutations which alter the biology of the infected cell. These
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Virus-Host Interactions and the Pathogenesis of Murine and Human Oncogenic Retroviruses, function. Furthermore, the proviral DNA integrates into the cellular genome introducing mutations which alter the biology of the infected cell. These interactions between the virus and the host and the phenomena associated with them determine the pathogenicity of retroviruses (L. and T. 1990 and this volume).
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Book 1991r its own expression through nontranslated sequences in the long terminal repeat (L TR). Some retroviruses also code for nonstructural proteins that further regulate proviral expression. Integration changes the cell genome; it adds viral genes, and in the case of transducing retroviruses also adds c
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ly sustained by Canadian and US think tanks, visionaries and policymakers. As well as being noted in many academic articles (for example, Swanson 1994; Courchène 1995; Edgington 1995; Alper 1996; Blatter 1996) and attracting the attention of mainstream policy-oriented think tanks like the Carnegie E
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H. J. Kung,C. Boerkoel,T. H. Carterf the neighbouring country. Thus we have to distinguish . of cross-border economic interaction, and we should be aware of the emerging . of cross-border modes of development in Central Europe, In this context, the outcome of current attempts to reposition the German-Polish border region in the new E
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P. N. Tsichlis,P. A. Lazoe of communism have persuaded much of the developing world to liberalize trade and participate actively in the new World Trade Organization (WTO), the successor to the GATT. Even China now eagerly seeks membership in the WTO as it emerges as a major exporter.
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e of communism have persuaded much of the developing world to liberalize trade and participate actively in the new World Trade Organization (WTO), the successor to the GATT. Even China now eagerly seeks membership in the WTO as it emerges as a major exporter.
作者: deface    時間: 2025-3-27 02:14
0070-217X , transcription of the cellular proto-oncogene myc was upregulated through the integration of a complete or partial provirus in its vicinity. This landmark disc978-3-642-76526-1978-3-642-76524-7Series ISSN 0070-217X Series E-ISSN 2196-9965
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