標題: Titlebook: Inflammation-Associated Depression: Evidence, Mechanisms and Implications; Robert Dantzer,Lucile Capuron Book 2017 Springer International [打印本頁] 作者: Negate 時間: 2025-3-21 19:43
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書目名稱Inflammation-Associated Depression: Evidence, Mechanisms and Implications被引頻次
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書目名稱Inflammation-Associated Depression: Evidence, Mechanisms and Implications讀者反饋學科排名
作者: omnibus 時間: 2025-3-21 23:00
er the credit line balance has been paid off. Because the agent receives enough in dividends, he has no incentives to misreport. When having a larger credit line is optimal, in order to be allowed such credit, it may happen that the debt is negative, meaning that the firm needs to maintain a margin 作者: 解決 時間: 2025-3-22 02:28 作者: IDEAS 時間: 2025-3-22 08:15
Lucile Capuron,Nathalie Castanoninary differential equation for the principal’s expected utility process as a function of the agent’s expected utility process. That equation can then be solved numerically for various cases, including the case in which the agent can quit, or be replaced by another agent, or be trained and promoted.作者: 牌帶來 時間: 2025-3-22 10:10
Angelos Halarisons of his project selection. We discuss numerical examples which suggest that if the manager privately makes project selection decisions for the firm which currently operates under a high-risk environment, then strikingly, the PPS can appear to increase with the overall volatility of the firm, if c作者: 雜色 時間: 2025-3-22 16:45 作者: 河潭 時間: 2025-3-22 20:24 作者: 雇傭兵 時間: 2025-3-22 22:38
Robert Dantzerons of his project selection. We discuss numerical examples which suggest that if the manager privately makes project selection decisions for the firm which currently operates under a high-risk environment, then strikingly, the PPS can appear to increase with the overall volatility of the firm, if c作者: Triglyceride 時間: 2025-3-23 03:00 作者: 教育學 時間: 2025-3-23 05:59
Karol Ramirez,Jaime Fornaguera-Trías,John F. Sheridann, especially from the French and German legal systems. Cash-on-delivery is regarded as an ancillary obligation of the carrier and, accordingly, the limits and the nature of the liability assumed by the transporter are illustrated. The chapter concludes with an analysis of cases in which goods are u作者: carbohydrate 時間: 2025-3-23 09:41 作者: Alopecia-Areata 時間: 2025-3-23 16:05
Neil A. Harrisonuction. Out of this presumption grew the cross-bridge model. in which filament sliding is presumed to be driven by the cyclic interaction of cross-bridges with complementary actin sites located along the thin filaments.978-1-4684-4705-7978-1-4684-4703-3Series ISSN 0065-2598 Series E-ISSN 2214-8019 作者: Perigee 時間: 2025-3-23 22:01 作者: 路標 時間: 2025-3-24 01:04 作者: Agronomy 時間: 2025-3-24 04:51 作者: Limerick 時間: 2025-3-24 08:31 作者: 軟弱 時間: 2025-3-24 11:25 作者: 厚顏 時間: 2025-3-24 18:24
Role of Kynurenine Metabolism Pathway Activation in Major Depressive Disorders,es have found activation of the kynurenine pathway and/or preferential activation of the neurotoxic (QA) pathway at the expense of the production of the NMDA antagonist, kynurenic acid. Initially, activation of the kynurenine pathway was believed to precipitate depressive symptoms by depleting brain作者: 使苦惱 時間: 2025-3-24 22:14
Suicidality and Activation of the Kynurenine Pathway of Tryptophan Metabolism,roinflammation. As the body of research in these areas grows, targeting the kynurenine pathway enzymes and metabolites may provide novel therapeutic opportunities for detection, treatment, and ultimately prevention of suicidal behavior.作者: integral 時間: 2025-3-24 23:51 作者: Mitigate 時間: 2025-3-25 05:26 作者: entice 時間: 2025-3-25 10:33
1866-3370 book “Inflammation-Associated Depression” is to present this field of research and its implications in a didactic and comprehensive manner to basic and clinical scientists, psychiatrists, physicians, and students at the graduate level.978-3-319-84581-4978-3-319-51152-8Series ISSN 1866-3370 Series E-ISSN 1866-3389 作者: 玩忽職守 時間: 2025-3-25 14:56
Ebrahim Haroon,Andrew H. Millernds of most countries belonging to the European Union. It covers contracts and different negotiating models as well as compensation, liability of the carrier, and damages..978-3-031-08489-8978-3-031-08487-4Series ISSN 2662-141X Series E-ISSN 2662-1428 作者: 打谷工具 時間: 2025-3-25 18:51
Anindya Bhattacharya,Wayne C. Drevets be of interest to researchers in industrial safety, as well as safety professionals and project managers within engineering industries..978-3-030-89791-8978-3-030-89792-5Series ISSN 2191-530X Series E-ISSN 2191-5318 作者: Prologue 時間: 2025-3-26 00:04
Caitlín N. M. Hastings,Hannah Sheridan,Carmine M. Pariante,Valeria Mondelli作者: 無意 時間: 2025-3-26 01:31 作者: Firefly 時間: 2025-3-26 08:04
Evidence for Inflammation-Associated Depression,on and depression wherein one process can drive the other. A wealth of animal and clinical studies have demonstrated an association between concentrations of pro-inflammatory cytokines – specifically interleukin (IL)-1β, IL-6, and tumor necrosis factor-α – and depressive symptoms. There is also evid作者: 職業(yè)拳擊手 時間: 2025-3-26 10:27
Role of Inflammation in the Development of Neuropsychiatric Symptom Domains: Evidence and Mechanismiteria driven by inflammation. Based on the model of inflammation-induced depression it has been possible to identify distinct pathophysiological pathways leading to alterations in neurotransmitter metabolism with specific relevance for the development of symptom constellations that are common to va作者: 諄諄教誨 時間: 2025-3-26 14:40 作者: 拖網 時間: 2025-3-26 19:54 作者: 享樂主義者 時間: 2025-3-26 23:09
Mechanisms of Inflammation-Associated Depression: Immune Influences on Tryptophan and Phenylalanine, most importantly of tryptophan and phenylalanine, plays a central role. In addition, neuropsychiatric conditions that go along with disorders that are characterized by acute or chronic inflammation, such as the development of depression, decreased quality of life or cognitive impairments, are conn作者: 消音器 時間: 2025-3-27 04:55 作者: 做方舟 時間: 2025-3-27 05:36
Depression in Autoimmune Diseases,ystem not only leads to inflammation in affected organs, but also mediates behavior abnormalities including fatigue and depression-like symptoms. This review focuses on the different pathways involved in the communication of the immune system with the neuronal network and the body’s timing system. T作者: EWE 時間: 2025-3-27 11:57 作者: Pander 時間: 2025-3-27 14:51 作者: 喚醒 時間: 2025-3-27 19:19
The Role of Dopamine in Inflammation-Associated Depression: Mechanisms and Therapeutic Implicationscytokines affect the basal ganglia and dopamine to mediate depressive symptoms related to motivation and motor activity. Findings have included inflammation-associated reductions in ventral striatal responses to hedonic reward, decreased dopamine and dopamine metabolites in cerebrospinal fluid, and 作者: Intersect 時間: 2025-3-27 23:43 作者: 悲觀 時間: 2025-3-28 04:48
Role of Kynurenine Metabolism Pathway Activation in Major Depressive Disorders,ory processes may play a causal role in the developmental of depressive illness. While there may be multiple causal pathways through which inflammatory processes affect mood, activation of the kynurenine pathway is essential for the development of depression-like behavior in rodents. Studies of hepa作者: dura-mater 時間: 2025-3-28 08:20
Suicidality and Activation of the Kynurenine Pathway of Tryptophan Metabolism,s calculated to be the 14th leading cause of death around the world. While the biological mechanisms causing suicidal ideation and behavior are not fully understood, increased levels of inflammation, arising from various sources, have been detected in the central nervous system and the peripheral bl作者: 阻止 時間: 2025-3-28 10:54 作者: 盡責 時間: 2025-3-28 16:37
Are Non-steroidal Anti-Inflammatory Drugs Clinically Suitable for the Treatment of Symptoms in Deprtivity in the periphery and in the brain has brought support to a concept of depression-associated inflammation. However, these immunological changes – and inflammation in particular – in depression have only been recently targeted for treatment. Non-steroidal anti-inflammatory drugs (NSAIDs) have b作者: 障礙物 時間: 2025-3-28 21:50 作者: Compassionate 時間: 2025-3-28 23:40 作者: tattle 時間: 2025-3-29 05:08 作者: 蹣跚 時間: 2025-3-29 10:10
Celina S. Liu,Alexander Adibfar,Nathan Herrmann,Damien Gallagher,Krista L. Lanct?ta 55:303–328,1987) is the first to use a continuous-time model, showing that doing that can, in fact, lead to simple, while realistic optimal contracts. In particular, if the principal and the agent maximize expected utility from terminal output value, and have non-separable cost of effort and expon作者: 過濾 時間: 2025-3-29 11:41 作者: 沖擊力 時間: 2025-3-29 16:56 作者: fibula 時間: 2025-3-29 21:25
Charlotte D’Mello,Mark G. Swainsion utilizes the representation of each admissible contract, in transforming the agent’s problem in a weak formulation with the mean-volatility controlled outcome into an equivalent problem in a strong formulation with a volatility controlled outcome. We present a general Hamiltonian for the princi作者: artless 時間: 2025-3-30 01:38 作者: impaction 時間: 2025-3-30 07:07
Robert Dantzersion utilizes the representation of each admissible contract, in transforming the agent’s problem in a weak formulation with the mean-volatility controlled outcome into an equivalent problem in a strong formulation with a volatility controlled outcome. We present a general Hamiltonian for the princi作者: 牲畜欄 時間: 2025-3-30 09:25
Christopher R. Pryce,Adriano Fontanasion utilizes the representation of each admissible contract, in transforming the agent’s problem in a weak formulation with the mean-volatility controlled outcome into an equivalent problem in a strong formulation with a volatility controlled outcome. We present a general Hamiltonian for the princi作者: 玷污 時間: 2025-3-30 15:09 作者: Polydipsia 時間: 2025-3-30 18:29 作者: Lineage 時間: 2025-3-30 20:42
Jennifer C. Felgerand biochemical information available today, it has been a simple matter to speculate about the possible ways in which tension generation and shortening might occur. The advent of the sliding filament model had an immediate impact on these theories; within several years they fell by the wayside, and作者: 證明無罪 時間: 2025-3-31 03:38
Neil A. Harrisonand biochemical information available today, it has been a simple matter to speculate about the possible ways in which tension generation and shortening might occur. The advent of the sliding filament model had an immediate impact on these theories; within several years they fell by the wayside, and作者: 一起 時間: 2025-3-31 07:18
Anindya Bhattacharya,Wayne C. Drevetsusing contractors within engineering industries.Provides adv.This open access book examines the increase in outsourcing, contracting and subcontracting as ways of organising work. It explores the impact of these employment arrangements on public safety, particularly when they are linked to complex s作者: 提煉 時間: 2025-3-31 10:43 作者: 宣傳 時間: 2025-3-31 14:35
Immune-to-Brain Communication Pathways in Inflammation-Associated Sickness and Depression,a-to-brain routes have received increasing attention for their ability to modulate brain function. In this chapter we discuss periphery-to-brain communication pathways and their potential role in mediating inflammation-associated sickness behaviors and depression.作者: STEER 時間: 2025-3-31 19:48 作者: aggrieve 時間: 2025-4-1 01:14 作者: 失望未來 時間: 2025-4-1 04:20
Does Diet Matter? The Use of Polyunsaturated Fatty Acids (PUFAs) and Other Dietary Supplements in Ile there is a definite need for more research in all these supplements to determine true efficacy, dosage and target populations, they can be used as mono- or adjunctive therapies to good effect, and show superior safety profiles when compared with more traditional alternatives.作者: 慢慢啃 時間: 2025-4-1 07:10 作者: 僵硬 時間: 2025-4-1 13:16
Depression in Autoimmune Diseases,he latter is built up by a hierarchically organized expression of clock genes. As discussed here, the activation of the immune system interferes with high amplitude expression of clock genes, an effect which may play a pivotal role in depression-like behavior in autoimmune diseases.作者: agonist 時間: 2025-4-1 15:22
Brain Structures Implicated in Inflammation-Associated Depression,nflammation-induced-sickness behavior, and in so doing revealed often-remarkable commonalities with circuit abnormalities observed in MDD. This review aims to provide the first synthesis of this work illustrating areas of convergence and divergence with the MDD literature as well as highlighting areas for future study.
