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標(biāo)題: Titlebook: Glucose-sensing Receptor in Pancreatic Beta-cells; Itaru Kojima Book 2018 Springer Nature Singapore Pte Ltd. 2018 Insulin secretion.Diabet [打印本頁]

作者: 管玄樂團(tuán)    時間: 2025-3-21 16:59
書目名稱Glucose-sensing Receptor in Pancreatic Beta-cells影響因子(影響力)




書目名稱Glucose-sensing Receptor in Pancreatic Beta-cells影響因子(影響力)學(xué)科排名




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書目名稱Glucose-sensing Receptor in Pancreatic Beta-cells網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Glucose-sensing Receptor in Pancreatic Beta-cells被引頻次




書目名稱Glucose-sensing Receptor in Pancreatic Beta-cells被引頻次學(xué)科排名




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書目名稱Glucose-sensing Receptor in Pancreatic Beta-cells年度引用學(xué)科排名




書目名稱Glucose-sensing Receptor in Pancreatic Beta-cells讀者反饋




書目名稱Glucose-sensing Receptor in Pancreatic Beta-cells讀者反饋學(xué)科排名





作者: judicial    時間: 2025-3-21 20:15

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作者: 生命層    時間: 2025-3-22 05:14
,The Role of the Glucose-Sensing Receptor in Glucose-Induced Insulin Secretion in Pancreatic β-Cellscked by an inhibitor of T1R3 or deletion of the T1R3 gene, glucose-induced insulin secretion (GIIS) is significantly reduced. In perifusion system, both first and second phases of GIIS are attenuated by the inhibition of the glucose-sensing receptor. Collectively, the glucose-sensing receptor is inv
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https://doi.org/10.1007/978-1-4471-3089-5cked by an inhibitor of T1R3 or deletion of the T1R3 gene, glucose-induced insulin secretion (GIIS) is significantly reduced. In perifusion system, both first and second phases of GIIS are attenuated by the inhibition of the glucose-sensing receptor. Collectively, the glucose-sensing receptor is inv
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https://doi.org/10.1007/978-981-13-0002-8Insulin secretion; Diabetes; Sweet Taste Receptor; Glucoreceptor; Metabolism
作者: 陰郁    時間: 2025-3-23 14:54
KATP Channel-Independent Pathway and the Glucoreceptor,. Therefore, the first phase of glucose-induced secretion, i.e., fusion of the beta granule and the plasma membrane, likely arises from both glucose-receptor binding and closure of KATP channels. The molecular nature of the so-called KATP-independent glucose action appears to be diverse and still re
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作者: gruelling    時間: 2025-3-24 04:02
as identification, channel pathway, receptor signal and physiological role. Readers will gain a thorough understanding of the glucose-sensing receptor?and glucose metabolism in pancreatic beta-cells, new insights into the?pathophysiology of diabetes, and learn about new targets for the treatment of diabetes.?.978-981-13-0002-8
作者: 慢慢啃    時間: 2025-3-24 09:24
Book 2018iological role. Readers will gain a thorough understanding of the glucose-sensing receptor?and glucose metabolism in pancreatic beta-cells, new insights into the?pathophysiology of diabetes, and learn about new targets for the treatment of diabetes.?.
作者: 斥責(zé)    時間: 2025-3-24 11:47
Learning Strategies and Learning Styles. Therefore, the first phase of glucose-induced secretion, i.e., fusion of the beta granule and the plasma membrane, likely arises from both glucose-receptor binding and closure of KATP channels. The molecular nature of the so-called KATP-independent glucose action appears to be diverse and still re
作者: ETCH    時間: 2025-3-24 15:46
https://doi.org/10.1007/0-387-28310-2and PKC activity. Importantly, these rapid signals were independent of glucose metabolism and were reproduced by addition of nonmetabolizable glucose analogs. Since these signals were inhibited by inhibition of Gq or Gs, it is quite likely that glucose activates a cell-surface receptor and generates
作者: Mercantile    時間: 2025-3-24 22:47
https://doi.org/10.1007/978-1-4471-3089-5irst acts on the cell-surface glucose-sensing receptor and primes the metabolic pathway of glucose. Glucose then enters β-cells and is metabolized through already activated metabolic pathway. The receptor pathway and the metabolic pathway act coordinately to stimulate insulin secretion.
作者: Nmda-Receptor    時間: 2025-3-25 01:06
Book 2018 findings on the glucose-sensing receptor have now shed new light on the “glucoreceptor theory.”.This volume reviews recent advances concerning the glucose-sensing receptor in pancreatic beta-cells..The history of research into pancreatic beta-cells is long and complex; accordingly, the first chapte
作者: 昏迷狀態(tài)    時間: 2025-3-25 05:50
ews the complex history of the glucose-sensing machinery in .Since the 1970s, there has been much discussion about the “glucoreceptor” and?“substrate site” and which of these two is the dominant theory, but new findings on the glucose-sensing receptor have now shed new light on the “glucoreceptor th
作者: 預(yù)測    時間: 2025-3-25 10:46
Jean-Claude Falmagne,Jean-Paul Doignonbuds of the tongue. Interestingly, inhibitors of the sweet taste receptor attenuate insulin secretion induced by glucose in pancreatic islets. These results raise an interesting possibility that glucose activates a cell-surface “glucoreceptor,” which resembles the sweet taste receptor in the tongue, and exerts its action in pancreatic β-cells.
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作者: insidious    時間: 2025-3-25 16:05
Technological Progress and Pious Modernity: Secular Liberals Fall Behind the Timeson of improved human prospects, however, is often laced with ambivalence: we have acquired greater power to destroy one another and the natural environment, and to produce more inventive forms of oppression. Therefore, secular liberals have often been accused of being too optimistic about the techni
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Timbre Perception with Cochlear Implantsept. This chapter addresses how people with severe hearing loss who have been fitted with a cochlear implant perceive timbre. A cochlear implant is a medical device that allows a deaf person to perceive sounds by stimulating their auditory nerve directly. Unlike a pair of glasses that perfectly rest
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作者: 舉止粗野的人    時間: 2025-3-26 15:40
Software for Analyzing Security for Healthcare Organizationsing the information infrastructure of a medical institution can cause colossal financial damage to medical institutions and companies, and, more importantly, pose a real threat to the health and life of patients. This work describes the process of assessing the security of the information infrastructure of healthcare institutions.
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Rocsildes Canoy,Tias Gunsllogeneic settings, the graft-vs-leukemia (GVL) activity should be responsible for very substantial disease control . and seems to cure a subset of poor-risk patients but at the price of an excessively high treatment-related mortality (TRM) .. The recent development of a nonmyeloablative conditionin
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