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標(biāo)題: Titlebook: Extracellular ATP and adenosine as regulators of endothelial cell function; Implications for hea Evgenia Gerasimovskaya,Elzbieta Kaczmarek [打印本頁]

作者: Forestall    時間: 2025-3-21 16:41
書目名稱Extracellular ATP and adenosine as regulators of endothelial cell function影響因子(影響力)




書目名稱Extracellular ATP and adenosine as regulators of endothelial cell function影響因子(影響力)學(xué)科排名




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書目名稱Extracellular ATP and adenosine as regulators of endothelial cell function網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Extracellular ATP and adenosine as regulators of endothelial cell function被引頻次




書目名稱Extracellular ATP and adenosine as regulators of endothelial cell function被引頻次學(xué)科排名




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書目名稱Extracellular ATP and adenosine as regulators of endothelial cell function年度引用學(xué)科排名




書目名稱Extracellular ATP and adenosine as regulators of endothelial cell function讀者反饋




書目名稱Extracellular ATP and adenosine as regulators of endothelial cell function讀者反饋學(xué)科排名





作者: penance    時間: 2025-3-21 21:09
Nucleotides and Novel Signaling Pathways in Endothelial Cells: Possible Roles in Angiogenesis, Endowing nucleotide-mediated activation of focal adhesion kinase (FAK), AMP-activated protein kinase (AMPK) and endothelial nitric oxide synthase (eNOS), and document the role of Ca. in these pathways. Activation by extracellular nucleotides of mitogen-activated protein kinases (MAPK), such as extracell
作者: 令人作嘔    時間: 2025-3-22 02:57

作者: Licentious    時間: 2025-3-22 05:53
The P2Y2 Nucleotide Receptor in Vascular Inflammation and Angiogenesis,gioplasty, and venous bypass graft failure. We have shown that activation of the P2Y. nucleotide receptor (P2Y.R) in vascular endothelial cells in vitro induces VCAM-1 (vascular cell adhesion molecule-1) expression and promotes the adherence of monocytes. VEGF, a pleiotropic factor that regulates en
作者: 中和    時間: 2025-3-22 12:38
Role of Purine-Converting Ecto-Enzymes in Angiogenic Phenotype of Pulmonary Artery Adventitial Vasally hypoxic calves. Because extracellular ATP and ADP are important regulators of vascular cell function, we hypothesized that these nucleotides may contribute to vasa vasorum neovascularization induced by chronic hypoxia. Treatment of pulmonary artery adventitial vasa vasorum endothelial cells (VVE
作者: 聲音刺耳    時間: 2025-3-22 15:44
Stimulation of Wound Revascularization by Adenosine Receptor Activation,ved that topical application of a selective adenosine A. receptor agonist promotes wound healing in experimental animals, both healthy and with impaired healing. Histological analysis revealed that adenosine promoted granulation tissue formation, with increased cellularity, matrix deposition, and ve
作者: 聲音刺耳    時間: 2025-3-22 17:15

作者: 和諧    時間: 2025-3-22 22:08
Regulated Extracellular Nucleotide Metabolism and Function at the Mucosa,denosine (Ado) can regulate a number of processes at the mucosa, e.g. dampening of the inflammatory response and resultant tissue protection. Ado receptors are widely expressed on a variety of organs and cell types and have been demonstrated to be central to the modulation of inflammation. In many i
作者: BUST    時間: 2025-3-23 01:29

作者: 擦試不掉    時間: 2025-3-23 07:09

作者: 自戀    時間: 2025-3-23 10:28

作者: Flat-Feet    時間: 2025-3-23 14:11

作者: esoteric    時間: 2025-3-23 21:56
https://doi.org/10.1007/978-3-319-94310-7 hyperpolarising factors, and prostacyclins (to a lesser extent). They are crucial for shear stress-mediated increases in blood flow and for the increase of tissue perfusion during exercise and hypoxia. The endothelium can also release potent contractile tetraphosphate nucleotides (AP.A and UP.A). T
作者: CLAIM    時間: 2025-3-23 22:49
Data Analytics in Manufacturingwing nucleotide-mediated activation of focal adhesion kinase (FAK), AMP-activated protein kinase (AMPK) and endothelial nitric oxide synthase (eNOS), and document the role of Ca. in these pathways. Activation by extracellular nucleotides of mitogen-activated protein kinases (MAPK), such as extracell
作者: bleach    時間: 2025-3-24 05:23
J. I. Igartua,J. Retegi,J. A. Egurencules and fluid between the blood and interstitial fluid. It is well known that loss of this barrier (permeability increase) results in tissue inflammation, the hall mark of inflammatory diseases such as acute lung injury (ALI) and a severe form of it, acute respiratory distress syndrome (ARDS). Apa
作者: seruting    時間: 2025-3-24 09:06

作者: Campaign    時間: 2025-3-24 13:22
https://doi.org/10.1007/978-3-031-26232-6lly hypoxic calves. Because extracellular ATP and ADP are important regulators of vascular cell function, we hypothesized that these nucleotides may contribute to vasa vasorum neovascularization induced by chronic hypoxia. Treatment of pulmonary artery adventitial vasa vasorum endothelial cells (VVE
作者: 健談的人    時間: 2025-3-24 17:13
https://doi.org/10.1007/978-3-030-19139-9ved that topical application of a selective adenosine A. receptor agonist promotes wound healing in experimental animals, both healthy and with impaired healing. Histological analysis revealed that adenosine promoted granulation tissue formation, with increased cellularity, matrix deposition, and ve
作者: SKIFF    時間: 2025-3-24 20:58
Production of the 70:20:10 Webinarease and development. Hypoxia stabilizes the hypoxia-inducible transcription factors -1α and -2α and also increases adenosine levels thereby activating the adenosine receptor signaling pathways. While these pathways have been described independently to a greater extent, increasing evidence suggests
作者: theta-waves    時間: 2025-3-25 02:09
Prices for Inputs Sold to Competitors,denosine (Ado) can regulate a number of processes at the mucosa, e.g. dampening of the inflammatory response and resultant tissue protection. Ado receptors are widely expressed on a variety of organs and cell types and have been demonstrated to be central to the modulation of inflammation. In many i
作者: BILK    時間: 2025-3-25 07:15

作者: 蔓藤圖飾    時間: 2025-3-25 09:59
Anthony J. Mahler,John Friedlandnd pathological processes. Direct IC between adjacent cells is mainly mediated by gap junctions, whereas paracrine signaling provides an indirect pathway for IC between neighboring cells. Purinergic mediators are important messengers in this paracrine signaling, and recent evidence shows that connex
作者: 偽善    時間: 2025-3-25 12:42
Syed Arslan Haider,Sonia Tabassum Aneesittently released ATP, related to exocytosis of insulin secretory granules, affects the secretory activity of the capillary endothelium, we have demonstrated that isolated endothelial cells respond to activation of P2Y. receptors with pronounced and extended rises of [Ca.].. The presence of such ATP
作者: 乳汁    時間: 2025-3-25 18:37

作者: STELL    時間: 2025-3-25 23:46
Evgenia Gerasimovskaya,Elzbieta KaczmarekThe first book with a unique focus on the effects of extracellular nucleotides and adenosine in endothelial cells..Describes regulatory role of extracellular nucleotides and adenosine in angiogenesis,
作者: 壯麗的去    時間: 2025-3-26 00:12

作者: 永久    時間: 2025-3-26 06:07

作者: Deject    時間: 2025-3-26 09:54
Purinergic and Pyriminergic Activation of the Endothelium in Regulation of Tissue Perfusion, circulatory shock and regulation of blood flow in most organs of the body. Purinergic and pyriminergic endothelium-mediated effects are important for physiological and patophysiological regulation of tissue perfusion.
作者: GIBE    時間: 2025-3-26 13:57
of extracellular nucleotides and adenosine in angiogenesis,.Extracellular purines and pyrimidines (ATP, ADP, UTP and adenosine) are released into the extracellular milieu in response to a variety of stress conditions and act as important regulators of vascular homeostasis. ..This new book is unique
作者: 機密    時間: 2025-3-26 17:49
Production of the 70:20:10 Webinarg the adenosine receptor signaling pathways. While these pathways have been described independently to a greater extent, increasing evidence suggests that there is significant crosstalk. Here we will summarize the contributions and interdependence of hypoxia-inducible transcription factors and the adenosine receptor pathways in vascular growth.
作者: analogous    時間: 2025-3-27 00:44
https://doi.org/10.1007/978-3-030-19139-9ssel density. Adenosine may exert effects on many different cell types that finally result in promotion of angiogenesis and, furthermore, homing of endothelial precursor cells into sites of injury. We will review the effect of adenosine receptor activation on microvascular endothelial cell function and on wound revascularization.
作者: Inveterate    時間: 2025-3-27 03:47

作者: 向下    時間: 2025-3-27 06:01

作者: acrobat    時間: 2025-3-27 12:07

作者: CLOWN    時間: 2025-3-27 16:02
Purinergic Signalling in Pancreatic Islet Endothelial Cells,pposed to entrain these cells into a common rhythm. Since the islet endothelial cells respond to activation of P2Y. receptors with extended elevation of [Ca.]., it is likely that the accompanying release of ATP is prolonged. Accordingly, the endothelial cells may have a tonic inhibitory action on the coordination of islet release pulses.
作者: dearth    時間: 2025-3-27 20:06

作者: 細(xì)胞學(xué)    時間: 2025-3-27 22:18

作者: 使害怕    時間: 2025-3-28 04:42

作者: 大方不好    時間: 2025-3-28 07:49
Miroslav Ma?covsky,Michal Balog,Jozef Jurkoe up-regulation of VCAM-1. The P2Y.R also contains an arginine-glycine-aspartate domain that mediates interactions with α.β./β. integrins that enable nucleotides to increase cell migration, an important event in inflammation and tumor angiogenesis. Taken together, these findings indicate that P2Y.Rs
作者: Duodenitis    時間: 2025-3-28 10:43

作者: 拉開這車床    時間: 2025-3-28 16:54

作者: 蚊帳    時間: 2025-3-28 19:07

作者: nugatory    時間: 2025-3-28 23:34
Anthony J. Mahler,John Friedlandmore, our results demonstrate that connexin43 hemichannels provide the pathway for ATP release. Our experiments show that the paracrine IC is strongly reduced in the presence of inflammatory agents, such as histamine and thrombin, that enhance actomyosin contraction. The effect of these agents is du
作者: Osmosis    時間: 2025-3-29 06:38
Nucleotides and Novel Signaling Pathways in Endothelial Cells: Possible Roles in Angiogenesis, Endoing through P2 receptors may exert anti-inflammatory, anti-oxidative, pro-proliferative, and anti-apoptotic effects in the endothelium. Sustaining of eNOS activation in ECs exposed to high glucose concentrations supports our hypothesis that extracellular nucleotides play a protective role against en
作者: Kindle    時間: 2025-3-29 08:02
Extracellular Purines in Endothelial Cell Barrier Regulation,upled receptors (GPCR). Selective activation of endothelial purinoreceptors responsible for barrier protection might form a basis for the treatment of various disorders. The therapeutic potential of purinoreceptors is rapidly expanding field in pharmacology and some selective agonists became recentl
作者: Heresy    時間: 2025-3-29 12:55
The P2Y2 Nucleotide Receptor in Vascular Inflammation and Angiogenesis,e up-regulation of VCAM-1. The P2Y.R also contains an arginine-glycine-aspartate domain that mediates interactions with α.β./β. integrins that enable nucleotides to increase cell migration, an important event in inflammation and tumor angiogenesis. Taken together, these findings indicate that P2Y.Rs
作者: 輪流    時間: 2025-3-29 17:27

作者: Orchiectomy    時間: 2025-3-29 21:39

作者: 腫塊    時間: 2025-3-30 02:33

作者: dictator    時間: 2025-3-30 07:43
ATP Release Via Connexin Hemichannels Controls Intercellular Propagation of Ca2+ Waves in Corneal Emore, our results demonstrate that connexin43 hemichannels provide the pathway for ATP release. Our experiments show that the paracrine IC is strongly reduced in the presence of inflammatory agents, such as histamine and thrombin, that enhance actomyosin contraction. The effect of these agents is du
作者: 可能性    時間: 2025-3-30 09:21
Book 2010othelial barrier function, and angiogenesis are discussed. The mechanisms of ATP release and its role in intercellular communication are also presented. .In addition, the book provides the most up to date mechanisms of extracellular nucleotide metabolism by purine-converting ecto-enzymes and their c
作者: FUSC    時間: 2025-3-30 15:12

作者: BLOT    時間: 2025-3-30 17:21
Extracellular ATP and adenosine as regulators of endothelial cell functionImplications for hea




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