標(biāo)題: Titlebook: Experimental Hypertension and Therapeutic Progress: Vasodilation and Beyond; Bernward Garthoff,Andreas M. Knorr,Friedel Seuter Conference [打印本頁(yè)] 作者: hearing-aid 時(shí)間: 2025-3-21 16:52
書(shū)目名稱Experimental Hypertension and Therapeutic Progress: Vasodilation and Beyond影響因子(影響力)
書(shū)目名稱Experimental Hypertension and Therapeutic Progress: Vasodilation and Beyond影響因子(影響力)學(xué)科排名
書(shū)目名稱Experimental Hypertension and Therapeutic Progress: Vasodilation and Beyond網(wǎng)絡(luò)公開(kāi)度
書(shū)目名稱Experimental Hypertension and Therapeutic Progress: Vasodilation and Beyond網(wǎng)絡(luò)公開(kāi)度學(xué)科排名
書(shū)目名稱Experimental Hypertension and Therapeutic Progress: Vasodilation and Beyond被引頻次
書(shū)目名稱Experimental Hypertension and Therapeutic Progress: Vasodilation and Beyond被引頻次學(xué)科排名
書(shū)目名稱Experimental Hypertension and Therapeutic Progress: Vasodilation and Beyond年度引用
書(shū)目名稱Experimental Hypertension and Therapeutic Progress: Vasodilation and Beyond年度引用學(xué)科排名
書(shū)目名稱Experimental Hypertension and Therapeutic Progress: Vasodilation and Beyond讀者反饋
書(shū)目名稱Experimental Hypertension and Therapeutic Progress: Vasodilation and Beyond讀者反饋學(xué)科排名
作者: Finasteride 時(shí)間: 2025-3-21 20:33 作者: 火光在搖曳 時(shí)間: 2025-3-22 02:18
Erich Grie?ler,Harald Rohracher first argument for the implication of a change in Ca. Channels in hypertension is the blood pressure-lowering effect of dihydropyridine Ca. antagonists (MacGregor et al. 1982; Kazda and Knorr 1990). These antagonists have also been shown to suppress the myogenic active tone displayed by blood vesse作者: 和平主義者 時(shí)間: 2025-3-22 07:59
Pharmacogenetic Study in Asthma,histicated preparation far from the physiological State. Therefore, we decided to study this question in anesthetized dogs in which the CA were infused directly into the left renal artery (LRA) at a dose not influencing systemic blood pressure [8]. Nevertheless, the . effects of CA are clinically no作者: Compatriot 時(shí)間: 2025-3-22 12:34
Functional Modifications in Blood Vessels of Hypertensive Rats, first argument for the implication of a change in Ca. Channels in hypertension is the blood pressure-lowering effect of dihydropyridine Ca. antagonists (MacGregor et al. 1982; Kazda and Knorr 1990). These antagonists have also been shown to suppress the myogenic active tone displayed by blood vesse作者: 盡忠 時(shí)間: 2025-3-22 13:22
Influence of Calcium Antagonists on Kidney Function of Hypertensive Rats,histicated preparation far from the physiological State. Therefore, we decided to study this question in anesthetized dogs in which the CA were infused directly into the left renal artery (LRA) at a dose not influencing systemic blood pressure [8]. Nevertheless, the . effects of CA are clinically no作者: 盡忠 時(shí)間: 2025-3-22 20:43 作者: 致敬 時(shí)間: 2025-3-22 21:56
Experimental Hypertension and Therapeutic Progress: Vasodilation and Beyond978-3-642-79338-7作者: 一條卷發(fā) 時(shí)間: 2025-3-23 01:46 作者: lipids 時(shí)間: 2025-3-23 06:55
Protection Against Hypertensive Tissue Damage,anda of the pharmaceutical industry. In my experience, this sceptical attitude is rather common among critical clinicians. And frankly speaking, from their point of view, such an attitude is fairly well substantiated.作者: 歡笑 時(shí)間: 2025-3-23 11:37
Could Kinins Contribute to the Vasculoprotective Effect of Potassium Supplementation?,ssible mechanisms involved in vasculoprotection and have promoted their study in the laboratory animal. In humans they have led to the INTACT and MIDAS trials for calcium Channel blockers and to the PROTECT trial for angiotensin-converting enzyme (ACE) inhibitors.作者: BILK 時(shí)間: 2025-3-23 16:38 作者: AVERT 時(shí)間: 2025-3-23 21:11
Clinical Relevance of Atrial Natriuretic Peptide,apan and in North America had independently sequenced and synthesized both a prohormone and major active constituent of atrial myocyte dense granules [2–6] called atrial natriuretic peptide, polypeptide (ANP) or factor (ANF).作者: animated 時(shí)間: 2025-3-24 00:05 作者: 信條 時(shí)間: 2025-3-24 04:53 作者: 繼承人 時(shí)間: 2025-3-24 08:03
scientific contributions and his involvement in drug development in our company, Bayer AG. In a recent article by Eugene Garfield (Current ContentslLife Sciences 36:3-15, 1993), what most of us participating in this workshop have supposed for years was confirmed: that Kazda is one of the leading sci作者: 大量殺死 時(shí)間: 2025-3-24 11:22 作者: 甜得發(fā)膩 時(shí)間: 2025-3-24 14:58 作者: 軍火 時(shí)間: 2025-3-24 19:46
https://doi.org/10.1007/978-3-031-09875-8ssible mechanisms involved in vasculoprotection and have promoted their study in the laboratory animal. In humans they have led to the INTACT and MIDAS trials for calcium Channel blockers and to the PROTECT trial for angiotensin-converting enzyme (ACE) inhibitors.作者: Esophagitis 時(shí)間: 2025-3-25 01:03 作者: 宇宙你 時(shí)間: 2025-3-25 04:29
https://doi.org/10.1007/978-3-642-79338-7Calcium antagonists; Calciumantagonisten; Hochdruck; Pharmakologie; Stroke; hypertension; pathophysiology; 作者: hurricane 時(shí)間: 2025-3-25 08:43 作者: hypertension 時(shí)間: 2025-3-25 13:11
https://doi.org/10.1007/978-3-030-75875-2Inhibition of atrial natriuretic peptide (ANP) degradation by enkephalinase is known to increase endogenous ANP levels and to induce similar functional effects as administration of exogenous ANP. The influence of this principle on cardiac function in rats with hypertension-induced heart failure was investigated.作者: Psychogenic 時(shí)間: 2025-3-25 18:28
Sinorphan Improves Cardiac Structure and Function in Aged Stroke-Prone Spontaneously Hypertensive RInhibition of atrial natriuretic peptide (ANP) degradation by enkephalinase is known to increase endogenous ANP levels and to induce similar functional effects as administration of exogenous ANP. The influence of this principle on cardiac function in rats with hypertension-induced heart failure was investigated.作者: 消散 時(shí)間: 2025-3-25 22:06 作者: 凌辱 時(shí)間: 2025-3-26 00:15
,Die Genomgr??e bei Pflanzen ist dynamisch,oth muscle cells (SMC) in carotid arteries of rabbits which were exposed to a local atherogenous stimulus. These substances have been recommended as antiatherogenic drugs. However, when using animal models for studying drug effects on the development of intimal proliferation of vascular SMC in the c作者: LITHE 時(shí)間: 2025-3-26 04:17
Erich Grie?ler,Harald Rohracheron et al. 1987; Boonen and De Mey 1990; Bodin et al. 1993). This hyperre-activity has been suggested to arise either from a change in receptor affinity (Nyborg and Bevan 1988), in receptor number, or in the transduction mechanisms of the receptor (Asano et al. 1988; Johnson et al. 1991). In addition作者: Mirage 時(shí)間: 2025-3-26 11:31 作者: MULTI 時(shí)間: 2025-3-26 15:05 作者: AXIOM 時(shí)間: 2025-3-26 20:00 作者: Pelvic-Floor 時(shí)間: 2025-3-27 00:39 作者: 痛苦一下 時(shí)間: 2025-3-27 01:09 作者: Culmination 時(shí)間: 2025-3-27 05:44
http://image.papertrans.cn/e/image/318845.jpg作者: 評(píng)論性 時(shí)間: 2025-3-27 09:35 作者: amputation 時(shí)間: 2025-3-27 16:37
Effect of Nitrendipine on Coronary Arteries in Spontaneously Hypertensive Rats: An Electron Microsc984), hemodynamic effects (Fleckenstein 1983), pharmacokinetics (Raemsch and Sommer 1984), and clinical effects (Hulthen et al. 1984; Tammen et al. 1984) of nitrendipine. This study deals with the effect of the drug in terms of electron microscopic and morphometric investigation.作者: myalgia 時(shí)間: 2025-3-27 21:46 作者: sperse 時(shí)間: 2025-3-28 00:02 作者: 迎合 時(shí)間: 2025-3-28 04:34 作者: expansive 時(shí)間: 2025-3-28 08:21 作者: 縱欲 時(shí)間: 2025-3-28 12:53 作者: mettlesome 時(shí)間: 2025-3-28 16:29 作者: 立即 時(shí)間: 2025-3-28 22:03
Could Kinins Contribute to the Vasculoprotective Effect of Potassium Supplementation?, the regression, of vascular hypertensive and age–related damage. Since the early 1980s, they have provided a stimulus for the investigation of the possible mechanisms involved in vasculoprotection and have promoted their study in the laboratory animal. In humans they have led to the INTACT and MIDA作者: CLEAR 時(shí)間: 2025-3-29 01:34
