標(biāo)題: Titlebook: Erythropoietin and the Nervous System; Ahmet H?ke (Associate Professor of Neurology and N Book 2006 Springer-Verlag US 2006 Nervous System [打印本頁] 作者: opioid 時(shí)間: 2025-3-21 16:39
書目名稱Erythropoietin and the Nervous System影響因子(影響力)
書目名稱Erythropoietin and the Nervous System影響因子(影響力)學(xué)科排名
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書目名稱Erythropoietin and the Nervous System網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Erythropoietin and the Nervous System被引頻次
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書目名稱Erythropoietin and the Nervous System讀者反饋
書目名稱Erythropoietin and the Nervous System讀者反饋學(xué)科排名
作者: Vertical 時(shí)間: 2025-3-21 22:27
,A Review of China’s Economy in 2013, way to the bone marrow where it regulates red cell production by preventing apoptosis of erythroid progenitor cells. Recently, EPO has emerged as a multifunctional growth factor that plays a significant role in the nervous system. Both EPO and its receptor are expressed throughout the brain in glia作者: dissolution 時(shí)間: 2025-3-22 00:49
Current Chinese Economic Report Seriesen identified by the meticulous work of many excellent research groups. The aim of this chapter is to identify and discuss the key signaling molecules and events published in numerous reports that are involved in EPO mediated neuroprotection. In order to provide a better overview, the signaling mole作者: 辮子帶來幫助 時(shí)間: 2025-3-22 07:44
https://doi.org/10.1007/978-981-19-8536-2) is a physiological response to hypoxia in oxygen-deprived tissues. Hypoxia-induced epo gene expression is regulated by the transcriptional activator hypoxia inducible factor (HIF). The epo gene contains a HIF binding site in its 3′ untranslated region, and its expression is upregulated concomitant作者: 生存環(huán)境 時(shí)間: 2025-3-22 09:28
Current Chinese Economic Report Seriesin maturation and contribute to variable patterns of brain injury in preterm and term neonates after HI. Following HI, a multifactorial cascade is initiated that injures the developing brain and can lead to significant morbidity and mortality. Despite intense investigation, no effective intervention作者: 騙子 時(shí)間: 2025-3-22 16:54 作者: 騙子 時(shí)間: 2025-3-22 17:37
,Forecast of China’s Economy for 2014–2015,ursors in the bone marrow. EPO and EPO receptor protein are also expressed by CNS neurons and glial cells. EPO exerts neuroprotective effects in various experimental in vitro and in vivo models of neural injury, such as mechanical trauma, neuroinflammation, and cerebral and retinal ischemia. In this作者: 儲備 時(shí)間: 2025-3-22 22:03 作者: indoctrinate 時(shí)間: 2025-3-23 03:45 作者: 大氣層 時(shí)間: 2025-3-23 07:07 作者: 闖入 時(shí)間: 2025-3-23 09:55 作者: 摘要 時(shí)間: 2025-3-23 15:07
International Management Knowledgepanied by an inflammatory reaction including production of inflammatory cytokines, leukocyte infiltration, and astroglial activation/ proliferation. In models of middle cerebral artery occlusion, EPO decreases production of tumor necrosis factor (TNF), interleukin (IL)-6, and of monocyte chemoattrac作者: 免費(fèi) 時(shí)間: 2025-3-23 22:01 作者: Verify 時(shí)間: 2025-3-23 23:16
978-1-4899-7383-2Springer-Verlag US 2006作者: neologism 時(shí)間: 2025-3-24 03:32
https://doi.org/10.1007/978-0-387-30011-5Nervous System; brain; central nervous system; death; hypoxia; inflammation; neuroprotection; neuroscience; 作者: Opponent 時(shí)間: 2025-3-24 06:52 作者: 小卒 時(shí)間: 2025-3-24 12:15 作者: Kidnap 時(shí)間: 2025-3-24 18:44
Erythropoietin in Spinal Cord Injury,ic, traumatic and inflammatory SCI. The recent development of non-erythropoietic derivatives of EPO with outstanding preclinical characteristics encourages evaluation of tissue-protective cytokines in clinical trials of spinal cord injury.作者: endocardium 時(shí)間: 2025-3-24 22:43 作者: ligature 時(shí)間: 2025-3-25 01:59
as to generate carbamylated EPO (CEPO), which has no affinity for the EPO receptor, but the same neuroprotective potency as EPO. Our data suggest that the cytoprotective signal transduction of EPO is distinct from the hematopoietic signaling machinery.作者: Mediocre 時(shí)間: 2025-3-25 04:28 作者: 騙子 時(shí)間: 2025-3-25 10:47
Development of Non-Erythropoietic Erythropoietin Variants for Neuroprotection,as to generate carbamylated EPO (CEPO), which has no affinity for the EPO receptor, but the same neuroprotective potency as EPO. Our data suggest that the cytoprotective signal transduction of EPO is distinct from the hematopoietic signaling machinery.作者: 思想上升 時(shí)間: 2025-3-25 12:22
https://doi.org/10.1057/978-1-137-59605-5rent from the hematopoietic system. Recent evidence of EPO as a protective factor in various injury models in the nervous system and heart has raised the possibility that EPO can exert protective effects in many organs in the body. However, whether the mechanism of protective action involves inhibition of apoptosis remains to be seen.作者: FAZE 時(shí)間: 2025-3-25 16:46 作者: 高腳酒杯 時(shí)間: 2025-3-25 23:17
China Economic Performance in 2016,. In addition, we show that this endogenous pathway can be therapeutically exploited by administering exogenous EPO in vivo and in vitro. Our data suggest that EPO prevents axonal degeneration, and may therefore be therapeutically useful in a wide variety of human neurological diseases characterized by axonopathy.作者: CREST 時(shí)間: 2025-3-26 02:20 作者: Forage飼料 時(shí)間: 2025-3-26 04:50 作者: 妨礙 時(shí)間: 2025-3-26 09:41
,An Endogenous Pathway Preventing Axonal Degeneration Mediated by Schwann Cell — Derived Erythropoie. In addition, we show that this endogenous pathway can be therapeutically exploited by administering exogenous EPO in vivo and in vitro. Our data suggest that EPO prevents axonal degeneration, and may therefore be therapeutically useful in a wide variety of human neurological diseases characterized by axonopathy.作者: Exposition 時(shí)間: 2025-3-26 15:14 作者: 懦夫 時(shí)間: 2025-3-26 20:16 作者: CHAFE 時(shí)間: 2025-3-26 22:30
Book 2006mulates red blood cell production, in recent years many laboratories have shown that EPO can act as a neuroprotective compound in a variety of injury paradigms in the nervous system. Past experience with relatively safety profile of this FDA-approved drug makes it an ideal candidate to take it into 作者: 惰性氣體 時(shí)間: 2025-3-27 01:15 作者: REP 時(shí)間: 2025-3-27 08:10 作者: 庇護(hù) 時(shí)間: 2025-3-27 12:59 作者: FID 時(shí)間: 2025-3-27 15:25
Expression of Erythropoietin and Its Receptor in the Central Nervous System, way to the bone marrow where it regulates red cell production by preventing apoptosis of erythroid progenitor cells. Recently, EPO has emerged as a multifunctional growth factor that plays a significant role in the nervous system. Both EPO and its receptor are expressed throughout the brain in glia作者: CRAFT 時(shí)間: 2025-3-27 18:43 作者: aggrieve 時(shí)間: 2025-3-27 21:57
Regulation of Erythropoietin Expression in the Nervous System: The Hypoxia Inducible Factor,) is a physiological response to hypoxia in oxygen-deprived tissues. Hypoxia-induced epo gene expression is regulated by the transcriptional activator hypoxia inducible factor (HIF). The epo gene contains a HIF binding site in its 3′ untranslated region, and its expression is upregulated concomitant作者: 人類 時(shí)間: 2025-3-28 04:59 作者: 憤怒事實(shí) 時(shí)間: 2025-3-28 06:27 作者: Gum-Disease 時(shí)間: 2025-3-28 14:22
Erythropoietin Neuroprotection in the Retina,ursors in the bone marrow. EPO and EPO receptor protein are also expressed by CNS neurons and glial cells. EPO exerts neuroprotective effects in various experimental in vitro and in vivo models of neural injury, such as mechanical trauma, neuroinflammation, and cerebral and retinal ischemia. In this作者: Gentry 時(shí)間: 2025-3-28 18:39 作者: 方便 時(shí)間: 2025-3-28 21:16 作者: minion 時(shí)間: 2025-3-29 02:01
Erythropoietin and Neuroprotection in the Peripheral Nervous System: , Studies,can cross the blood brain barrier and inhibit neuronal death and inflammatory infiltrates induced by ischemia in the brain. Recent evidence also suggests that rhEPO is a potent neuroprotective agent in primary sensory neurons and peripheral glia involved in pain transmission. When L5 spinal nerve is作者: vector 時(shí)間: 2025-3-29 05:38
,An Endogenous Pathway Preventing Axonal Degeneration Mediated by Schwann Cell — Derived Erythropoieneration and neuronal death are likely to be diverse. In this chapter, we describe a novel, endogenous pathway that prevents axonal degeneration. We show that in response to axonal injury, peri-axonal Schwann cells release erythropoietin (EPO), which via binding to EPO receptors on neurons prevents 作者: meritorious 時(shí)間: 2025-3-29 10:40
Role of Erythropoietin in Inflammatory Pathologies of the CNS,panied by an inflammatory reaction including production of inflammatory cytokines, leukocyte infiltration, and astroglial activation/ proliferation. In models of middle cerebral artery occlusion, EPO decreases production of tumor necrosis factor (TNF), interleukin (IL)-6, and of monocyte chemoattrac作者: insincerity 時(shí)間: 2025-3-29 13:46 作者: 材料等 時(shí)間: 2025-3-29 17:54
Book 2006ifferent disciplines of neuroscience to review the current state-of-the-art in EPO and the nervous system. This book will benefit scientists and clinicians interested in neuroprotection in the broadest sense..作者: CURL 時(shí)間: 2025-3-29 22:51
,A Review of China’s Economy in 2013, responses. Thus, hypoxically upregulated EPO is a naturally self-regulated physiological protective mechanism in the mammalian brain, especially during ischemia. As EPO is also a clinically extremely well studied and tolerated compound, its use in stroke patients is tempting.作者: Defense 時(shí)間: 2025-3-30 00:51
https://doi.org/10.1007/978-981-19-8536-2sensing mechanism that regulates the activation of HIF under hypoxic conditions. An important challenge for the future is to determine how modulating the activation of HIF with the subsequent expression of EPO can be beneficial for neural survival under stress conditions that involve hypoxia.作者: ensemble 時(shí)間: 2025-3-30 07:14 作者: Anticoagulant 時(shí)間: 2025-3-30 11:45 作者: Commodious 時(shí)間: 2025-3-30 15:48
https://doi.org/10.1007/978-3-642-40044-5f rhEPO are not limited to direct effects on neurons. System administration of rhEPO in animals given a chronic constriction injury to the sciatic nerve, a model known to produce the pro-inflammatory cytokine, TNF-α, at the injury site results in significant reductions in Schwann cell produced TNF-α作者: TEM 時(shí)間: 2025-3-30 19:29
International Management Knowledgedicating that one of the mechanisms by which inflammation promotes and extend damage could be through inhibition of EPO production. Thus, EPO should be viewed as part of the inflammation/anti-inflammation network in the CNS.
