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標(biāo)題: Titlebook: Energy Metabolism and Obesity; Research and Clinica Patricia A. Donohoue Book 2008 Humana Press 2008 Balance.Diabetes.Energy.Insulin.Insuli [打印本頁]

作者: 哥哥大傻瓜    時(shí)間: 2025-3-21 20:08
書目名稱Energy Metabolism and Obesity影響因子(影響力)




書目名稱Energy Metabolism and Obesity影響因子(影響力)學(xué)科排名




書目名稱Energy Metabolism and Obesity網(wǎng)絡(luò)公開度




書目名稱Energy Metabolism and Obesity網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Energy Metabolism and Obesity被引頻次




書目名稱Energy Metabolism and Obesity被引頻次學(xué)科排名




書目名稱Energy Metabolism and Obesity年度引用




書目名稱Energy Metabolism and Obesity年度引用學(xué)科排名




書目名稱Energy Metabolism and Obesity讀者反饋




書目名稱Energy Metabolism and Obesity讀者反饋學(xué)科排名





作者: hegemony    時(shí)間: 2025-3-21 21:00

作者: 東西    時(shí)間: 2025-3-22 03:00

作者: 浪蕩子    時(shí)間: 2025-3-22 06:46
Huizhong Tian,Tao Li,Zhibo SongThe authors review the pathogenesis and diagnostic evaluation of insulin resistance in obesity and the polycystic ovary syndrome. They discuss the benefits and risks of lifestyle intervention and pharmacotherapy and present a tailored approach to pharmacotherapy in high risk adolescents.
作者: HARD    時(shí)間: 2025-3-22 12:30

作者: 影響帶來    時(shí)間: 2025-3-22 14:38

作者: 影響帶來    時(shí)間: 2025-3-22 18:36

作者: harmony    時(shí)間: 2025-3-22 22:10
J. D. Blankensteijn,William M. Abbott M.D.gulated via an ongoing process called energy homeostasis, or the long-term matching of food intake to energy expenditure. Reductions from an individual’s “normal” weight due to lack of sufficient food lowers levels of adiposity signals (leptin and insulin) reaching the brain from the blood, activate
作者: 浪蕩子    時(shí)間: 2025-3-23 01:31

作者: BOOR    時(shí)間: 2025-3-23 08:31

作者: 壓迫    時(shí)間: 2025-3-23 11:51
Elizabeth W. Hubbard,Daniel J. Sucatorough afferent neural and hormonal signals reaching the hypothalamus, with resultant efferent projections of the autonomic nervous system innervating the muscles, viscera, and adipose tissue. The efferent system is of particular interest, as it has its own pathologies, which can be modulated to achi
作者: 手榴彈    時(shí)間: 2025-3-23 15:04

作者: inhumane    時(shí)間: 2025-3-23 19:40
Numbers of Vertebrae in Hominoid Evolution,essed carbohydrates is likely to have increased and physical activity levels declined. However, the rates at which obesity and diabetes have increased differ across people of varying ethnicities living in the same environment, suggesting the presence of interaction between ethnic-specific factors, s
作者: Corporeal    時(shí)間: 2025-3-23 22:44
Fusion Techniques for Traumatic Injuriesrticoid excess. Although morning plasma cortisol concentrations are low or normal in obesity, it is possible that tissue cortisol levels are increased. The enzyme 11α-hydroxysteroid dehydrogenase type 1 (11α-HSD1) catalyzes the conversion of inactive cortisone to active cortisol and is present in ma
作者: 鄙視讀作    時(shí)間: 2025-3-24 05:09

作者: 試驗(yàn)    時(shí)間: 2025-3-24 09:18

作者: Aesthete    時(shí)間: 2025-3-24 13:43

作者: 生氣地    時(shí)間: 2025-3-24 15:39
Leptin Signaling In the Brain,gulated via an ongoing process called energy homeostasis, or the long-term matching of food intake to energy expenditure. Reductions from an individual’s “normal” weight due to lack of sufficient food lowers levels of adiposity signals (leptin and insulin) reaching the brain from the blood, activate
作者: 專心    時(shí)間: 2025-3-24 21:20
Inactivating Melanocortin 4 Receptor Mutations and Human Obesity,MC4R) as a regulator of energy homeostasis in rodents. Human genetic studies showed that mutations in the MC4R gene are the most common monogenic form of obesity. About 80 different mutations have been identified from cohorts from various ethnic origins. Functional analyses of the mutant receptors r
作者: Mitigate    時(shí)間: 2025-3-25 02:31

作者: staging    時(shí)間: 2025-3-25 04:47
The Efferent Arm of the Energy Balance Regulatory Pathway: Neuroendocrinology and Pathology,rough afferent neural and hormonal signals reaching the hypothalamus, with resultant efferent projections of the autonomic nervous system innervating the muscles, viscera, and adipose tissue. The efferent system is of particular interest, as it has its own pathologies, which can be modulated to achi
作者: 流逝    時(shí)間: 2025-3-25 10:49

作者: handle    時(shí)間: 2025-3-25 14:05
Interaction Between Physical Activity and Genetic Factors in Complex Metabolic Disease,essed carbohydrates is likely to have increased and physical activity levels declined. However, the rates at which obesity and diabetes have increased differ across people of varying ethnicities living in the same environment, suggesting the presence of interaction between ethnic-specific factors, s
作者: 宇宙你    時(shí)間: 2025-3-25 19:43

作者: Jogging    時(shí)間: 2025-3-25 23:06
,Antipsychotic Medication–Induced Weight Gain and Risk for Diabetes and Cardiovascular Disease, and cardiovascular disease (CVD), with related increases in mortality. Increased adiposity is associated with decreases in insulin sensitivity, leading to increased risk of hyperglycemia and hyperlipidemia. Current evidence supports the hypothesis that treatment with antipsychotic medications is as
作者: irradicable    時(shí)間: 2025-3-26 04:01
The Surgical Approach to Morbid Obesity,inning in 1966, which restricts food intake and produces less malabsorption. Both operations exposed the distal ileum to glucose and fat, which stimulates secretion of glucagon-like peptide-1 (GLP-1). This is a brake hormone for gastric emptying and intestinal peristalsis. GLP-1 is also an incretin
作者: maudtin    時(shí)間: 2025-3-26 06:27

作者: hereditary    時(shí)間: 2025-3-26 08:43

作者: discord    時(shí)間: 2025-3-26 13:24

作者: 誓言    時(shí)間: 2025-3-26 19:26
Zulejha Merhemic,Majda M. Thurnhertaining normal body weight in humans is highlighted by the finding that disordered melanocortin signaling results in early-onset morbid obesity and dramatic increases in lean body mass in humans. Emerging evidence suggests that blocking this system via pharmacologic antagonists to the type 4 melanoc
作者: Exaggerate    時(shí)間: 2025-3-26 23:35

作者: 大酒杯    時(shí)間: 2025-3-27 04:56
https://doi.org/10.1007/978-981-15-7265-4and thus the primary dual action of ghrelin/GHSs on both GH secretion and food intake teleologically seem most reasonable and sound. The fundamental action of these dual effects is underscored in body growth of children and the impaired action of GH in the undernourished, and, thus, this is consider
作者: 陳腐的人    時(shí)間: 2025-3-27 08:18
Numbers of Vertebrae in Hominoid Evolution,given locus by comparison with those who do not. Because exercising skeletal muscle is a major site for glucose and lipid metabolism, variants in the genes that are located within muscle and that are up-regulated in response to physical activity present interesting candidates for testing in studies
作者: OPINE    時(shí)間: 2025-3-27 11:30
Fusion Techniques for Traumatic Injuriestabolic disease. Genetic studies have shown associations between polymorphisms in the . gene and components of the metabolic syndrome. Nonspecific inhibition of this enzyme leads to improved hepatic insulin sensitivity, while more selective compounds are being developed to potentially treat central
作者: sinoatrial-node    時(shí)間: 2025-3-27 14:14
Neurological Deficit After Spinal Osteotomy, aripiprazole, to larger increases during treatment with agents such as olanzapine and clozapine (e.g., 4–10 kg). Substantial evidence indicates that increases in adiposity are associated with decreases in insulin sensitivity in persons with and without psychiatric disease. The effects of increasing
作者: 考博    時(shí)間: 2025-3-27 19:14
Leptin Signaling In the Brain,s nutrients and generates signals and activation of neuronal systems and circuits that regulate energy homeostasis. This chapter focuses on how these signals are received and integrated by the central nervous system.
作者: 松軟    時(shí)間: 2025-3-28 00:32
The Role of Central Melanocortins in Cachexia,taining normal body weight in humans is highlighted by the finding that disordered melanocortin signaling results in early-onset morbid obesity and dramatic increases in lean body mass in humans. Emerging evidence suggests that blocking this system via pharmacologic antagonists to the type 4 melanoc
作者: GLADE    時(shí)間: 2025-3-28 04:43
The Efferent Arm of the Energy Balance Regulatory Pathway: Neuroendocrinology and Pathology,ctivity increases to store energy. Hypothalamic obesity, an example of “organic” leptin resistance, manifests similarly. Finally, anorexia nervosa is an example of dissociation between weight loss, but with increased sympathetic modulation. Understanding the relationship between these two efferent a
作者: Basal-Ganglia    時(shí)間: 2025-3-28 07:59
The Role of Growth Hormone Secretagogues and Ghrelin in Feeding and Body Composition,and thus the primary dual action of ghrelin/GHSs on both GH secretion and food intake teleologically seem most reasonable and sound. The fundamental action of these dual effects is underscored in body growth of children and the impaired action of GH in the undernourished, and, thus, this is consider
作者: 勤勞    時(shí)間: 2025-3-28 12:57

作者: 休閑    時(shí)間: 2025-3-28 14:47
,11β-Hydroxysteroid Dehydrogenase Type 1 and Obesity,tabolic disease. Genetic studies have shown associations between polymorphisms in the . gene and components of the metabolic syndrome. Nonspecific inhibition of this enzyme leads to improved hepatic insulin sensitivity, while more selective compounds are being developed to potentially treat central
作者: Cardioversion    時(shí)間: 2025-3-28 19:36
,Antipsychotic Medication–Induced Weight Gain and Risk for Diabetes and Cardiovascular Disease, aripiprazole, to larger increases during treatment with agents such as olanzapine and clozapine (e.g., 4–10 kg). Substantial evidence indicates that increases in adiposity are associated with decreases in insulin sensitivity in persons with and without psychiatric disease. The effects of increasing
作者: Affiliation    時(shí)間: 2025-3-29 02:51
2523-3785 treatment.? The excellent overviews in this book will undoubtedly provide readers with a better understanding of the multifaceted entity of obesity.978-1-61737-710-5978-1-60327-139-4Series ISSN 2523-3785 Series E-ISSN 2523-3793
作者: adhesive    時(shí)間: 2025-3-29 04:13

作者: adumbrate    時(shí)間: 2025-3-29 07:15

作者: Synthesize    時(shí)間: 2025-3-29 14:25
Book 2008enables researchers and clinicians to face the challenge of understanding this complex topic and applying the information to the practice of obesity prevention and treatment.? The excellent overviews in this book will undoubtedly provide readers with a better understanding of the multifaceted entity of obesity.
作者: 陰郁    時(shí)間: 2025-3-29 17:36
Anton Wernig,Andras Nanassy,Sabina Müllersis. Many of the genes identified relate to the development and function of the hypothalamus and central control of food intake and energy homeostasis. Allelic variations in these genes could contribute to nonsyndromic forms of obesity.
作者: 纖細(xì)    時(shí)間: 2025-3-29 21:32
https://doi.org/10.1007/978-3-642-72527-2d-type receptor. A classification scheme was proposed to catalogue the ever-increasing array of MC4R mutations. Based on studies from other G protein–coupled receptors, potential therapeutic approaches to correct the dysfunctional MC4Rs are highlighted.
作者: 借喻    時(shí)間: 2025-3-30 01:11

作者: 強(qiáng)壯    時(shí)間: 2025-3-30 05:43

作者: INTER    時(shí)間: 2025-3-30 10:39
Inactivating Melanocortin 4 Receptor Mutations and Human Obesity,d-type receptor. A classification scheme was proposed to catalogue the ever-increasing array of MC4R mutations. Based on studies from other G protein–coupled receptors, potential therapeutic approaches to correct the dysfunctional MC4Rs are highlighted.
作者: neuron    時(shí)間: 2025-3-30 15:40





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