標(biāo)題: Titlebook: Emerging Strategies in Neuroprotection; Paul J. Marangos,Harbans Lal Book 1992 Birkh?user Boston 1992 Evolution.biology.brain.neuroscience [打印本頁] 作者: angiotensin-I 時(shí)間: 2025-3-21 17:21
書目名稱Emerging Strategies in Neuroprotection影響因子(影響力)
書目名稱Emerging Strategies in Neuroprotection影響因子(影響力)學(xué)科排名
書目名稱Emerging Strategies in Neuroprotection網(wǎng)絡(luò)公開度
書目名稱Emerging Strategies in Neuroprotection網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Emerging Strategies in Neuroprotection被引頻次
書目名稱Emerging Strategies in Neuroprotection被引頻次學(xué)科排名
書目名稱Emerging Strategies in Neuroprotection年度引用
書目名稱Emerging Strategies in Neuroprotection年度引用學(xué)科排名
書目名稱Emerging Strategies in Neuroprotection讀者反饋
書目名稱Emerging Strategies in Neuroprotection讀者反饋學(xué)科排名
作者: OCTO 時(shí)間: 2025-3-21 22:31
Modeling Neurodegeneration and Neuroprotection in Hippocampal Slicestechnique became a useful tool in the hands of electrophysiologists and pharmacologists. Yamamoto and McIlwain (1966) were the first to show that mammalian brain (prepyriform cortex) slices maintained in vitro exhibited electrical activity comparable to that obtained in vivo. Along with cell culture作者: gnarled 時(shí)間: 2025-3-22 04:12
Stroke Models for Preclinical Trials of Neuroprotective Agentsain during or following ischemic insult. Despite the numerous claims of therapeutic success in animal models in the past two decades, no therapeutic agents have been approved by the Food and Drug Administration (FDA) for patients with acute ischemic stroke in the United States (Pulsinelli and Buchan作者: 敘述 時(shí)間: 2025-3-22 08:29
Animal Models of Ischemiaficulty determining whether a proposed therapy is effective. Appropriate studies in patients are complex, and few animal models are adequate for realistic and efficient tests of pharmacologic methods to reduce damage or restore neurologic function. Most cerebral stroke models have not been designed 作者: Muffle 時(shí)間: 2025-3-22 09:06 作者: MEN 時(shí)間: 2025-3-22 13:28 作者: MEN 時(shí)間: 2025-3-22 21:07 作者: ingenue 時(shí)間: 2025-3-22 21:41
Calcium Channel Blockers and Neuroprotectionolved within a short period of time (minutes), will lead to a core of severely ischemic tissue that may not be salvaged. However, the ultimate size of the brain infarct also depends on the “penumbra,” a zone of tissue around the core of the infarct where blood flow is still maintained above a neuron作者: 縮短 時(shí)間: 2025-3-23 01:46
Self-Defense of the Brain: Adenosinergic Strategies in Neurodegenerationedicine. The breadth and scope of the pathologic factors are such that, even with the improved understanding of the involved events, it is extremely difficult to decide which of them are the primary instigators of the ensuing damage and which are but secondary effects of an ongoing process of the ne作者: nonsensical 時(shí)間: 2025-3-23 09:09 作者: 鍍金 時(shí)間: 2025-3-23 12:33
Lazaroids: Novel Cerebroprotective Antioxidantstion in the acute pathophysiology of central nervous system (CNS) injury and ischemia (i.e., stroke) (Braughler and Hall, 1989; Hall and Braughler, 1989). The 21-aminosteroids, or “l(fā)azaroids,” are a novel series of compounds being developed for the acute treatment of traumatic or ischemic CNS injury作者: Lipoprotein 時(shí)間: 2025-3-23 15:02
Membrane-Derived Lipid Second Messengers as Targets for Neuroprotection: Platelet-Activating Factor in several human pathologies including asthma and shock, cardiac and systemic anaphylaxis, immune and renal disorders, and a variety of inflammatory conditions (Braquet et al., 1987; Prescott et al., 1990). In addition, several lines of evidence suggest the implication of PAF in various pathophysio作者: 錢財(cái) 時(shí)間: 2025-3-23 20:59 作者: Osteons 時(shí)間: 2025-3-24 01:00 作者: fetter 時(shí)間: 2025-3-24 02:30
Temperature Modulation of Neuronal Injurymental and clinical literature, on therapeutic hypo- and hyperthermia. The protective effect of profound hypothermia (30°C or less) has been well established during cardiac and intracranial surgical procedures necessitating interruption of blood flow to the brain. Hypothermia has also been and conti作者: Gerontology 時(shí)間: 2025-3-24 09:35
Clinical Trial Issues in Stroke Therapy(1990) stated that during this period we will see “The prevention of 80% of all strokes and protection of the brain during the acute stroke.” To accomplish this task new therapies must be developed. The safety and efficacy of any new agent must be demonstrated in adequate and well-controlled trials.作者: 截?cái)?nbsp; 時(shí)間: 2025-3-24 10:39
Book 1992 as the decade of the brain, based largely on the rapid escalation of advances in the molecular neuro- sciences and the likelihood that these will bear therapeutic fruit before the turn of the century. There is little doubt that the 1970s and 1980s have witnessed more remarkable advances in the mole作者: CODE 時(shí)間: 2025-3-24 18:06 作者: 膽大 時(shí)間: 2025-3-24 22:36 作者: Emg827 時(shí)間: 2025-3-25 01:37 作者: BLA 時(shí)間: 2025-3-25 04:14 作者: 招待 時(shí)間: 2025-3-25 11:23 作者: 頭腦冷靜 時(shí)間: 2025-3-25 15:27 作者: 饒舌的人 時(shí)間: 2025-3-25 16:13
,Einführung in die Welt des Neuromarketing,nues to be used in the head-injured patient for the control of increased intracranial pressure that is refractory to hyperventilation, ventricular drainage, osmotherapy, and barbiturates. Conversely, the adjunctive chemotherapeutic effect of selective brain tumor hyperthermia remains under intense investigation.作者: 下垂 時(shí)間: 2025-3-25 20:56 作者: SCORE 時(shí)間: 2025-3-26 02:05 作者: 憤怒事實(shí) 時(shí)間: 2025-3-26 08:16 作者: EXALT 時(shí)間: 2025-3-26 11:27
Primary Cultures for Testing Neuroprotective Drugsure systems and their characteristics. Then we shall discuss various possible ways to experimentally induce and measure neuronal damage in these cultures in analogy to ischemic damage in vivo. Several examples of neuroprotective drug effects demonstrated in vitro will be reviewed.作者: Felicitous 時(shí)間: 2025-3-26 13:55
Stroke Models for Preclinical Trials of Neuroprotective Agentsmal stroke models (Graham, 1988; Molinari, 1988; Wiebers et al., 1990; Zivin and Grotta, 1990). An important issue, i.e., the quality of animal trials, however, has not been adequately addressed previously and will be the main focus of this review.作者: 字形刻痕 時(shí)間: 2025-3-26 19:53
Membrane-Derived Lipid Second Messengers as Targets for Neuroprotection: Platelet-Activating Factorrg et al., 1990), and experimental allergic encephalomyelitis (Howat et al., 1989). Recent evidence shows a role for PAF in the release of excitatory amino acid neurotransmitters (Clark et al., 1991) and in long-term potentiation (Del Cerro et al., 1990).作者: Evocative 時(shí)間: 2025-3-27 00:55
the 1990s as the decade of the brain, based largely on the rapid escalation of advances in the molecular neuro- sciences and the likelihood that these will bear therapeutic fruit before the turn of the century. There is little doubt that the 1970s and 1980s have witnessed more remarkable advances i作者: 尖牙 時(shí)間: 2025-3-27 03:19
Cerebrovascular Disorders in Adultsifficult to decide which of them are the primary instigators of the ensuing damage and which are but secondary effects of an ongoing process of the neuronal destruction. The most striking example of the magnitude of the problems that must be faced is provided by the diseases that affect the supply of blood to the brain.作者: lavish 時(shí)間: 2025-3-27 05:47
Neurology and Psychiatry of Women89). The 21-aminosteroids, or “l(fā)azaroids,” are a novel series of compounds being developed for the acute treatment of traumatic or ischemic CNS injury that have been specifically designed to localize within cell membranes and inhibit lipid peroxidation reactions.作者: 流動(dòng)才波動(dòng) 時(shí)間: 2025-3-27 10:10 作者: Mawkish 時(shí)間: 2025-3-27 15:45
Visual Loss and Other Visual Disturbances,ldrum, 1990, and in Chapters 3 (Hsu et al.), 4 (Zivin), and 5 (Hayes et al.) of this volume]. Here we shall consider the various pharmacologic options for reducing excitatory effects, reviewing the drugs available for modifying the different stages of the excitotoxic process and the evidence for their cerebroprotective efficacy in animal models.作者: Systemic 時(shí)間: 2025-3-27 21:42
Excitatory Amino Acids and Neuroprotectionldrum, 1990, and in Chapters 3 (Hsu et al.), 4 (Zivin), and 5 (Hayes et al.) of this volume]. Here we shall consider the various pharmacologic options for reducing excitatory effects, reviewing the drugs available for modifying the different stages of the excitotoxic process and the evidence for their cerebroprotective efficacy in animal models.作者: GULP 時(shí)間: 2025-3-27 22:04
978-1-4684-6798-7Birkh?user Boston 1992作者: CAND 時(shí)間: 2025-3-28 05:01
Advances in Neuroprotectionhttp://image.papertrans.cn/e/image/308382.jpg作者: PANG 時(shí)間: 2025-3-28 10:09
https://doi.org/10.1007/978-1-4684-6796-3Evolution; biology; brain; neuroscience; receptor作者: duplicate 時(shí)間: 2025-3-28 11:15 作者: galley 時(shí)間: 2025-3-28 15:25
https://doi.org/10.1007/978-3-7091-8200-0technique became a useful tool in the hands of electrophysiologists and pharmacologists. Yamamoto and McIlwain (1966) were the first to show that mammalian brain (prepyriform cortex) slices maintained in vitro exhibited electrical activity comparable to that obtained in vivo. Along with cell culture作者: Expostulate 時(shí)間: 2025-3-28 21:46
Neurologische Untersuchungs-Schemataain during or following ischemic insult. Despite the numerous claims of therapeutic success in animal models in the past two decades, no therapeutic agents have been approved by the Food and Drug Administration (FDA) for patients with acute ischemic stroke in the United States (Pulsinelli and Buchan作者: GRATE 時(shí)間: 2025-3-29 01:15 作者: 持久 時(shí)間: 2025-3-29 06:14
https://doi.org/10.1007/978-3-642-70007-1rm in a clinical context dates from the sixteenth century, during which concussion or . was used to designate the effects of injuries to the brain without fracture to the skull (Johnson, 1634). Quite appropriately, concussion has subsequently been applied collectively to the wide range of biomechani作者: PHIL 時(shí)間: 2025-3-29 10:46
https://doi.org/10.1007/978-0-387-88555-1stologic studies of Olney and his colleagues during the early 1970s. These investigators found that excitatory amino acids such as glutamate, when administered to immature rodents, induced degenerative changes in neurons in the retina and in the hypothalamus (Olney, 1971; Olney et al., 1971). Subseq作者: Glower 時(shí)間: 2025-3-29 13:31
Visual Loss and Other Visual Disturbances, a role of excitotoxicity in the brain damage that accompanies cerebral ischemia and trauma has been presented elsewhere (Choi and Rothman, 1990; Meldrum, 1990; Meldrum and Garthwaite, 1990; Olney, 1990) and will not be further reviewed here. Nor shall we provide any review or analysis of the differ作者: DUST 時(shí)間: 2025-3-29 19:19
https://doi.org/10.1007/978-1-59259-984-4olved within a short period of time (minutes), will lead to a core of severely ischemic tissue that may not be salvaged. However, the ultimate size of the brain infarct also depends on the “penumbra,” a zone of tissue around the core of the infarct where blood flow is still maintained above a neuron作者: Frenetic 時(shí)間: 2025-3-29 23:26
Cerebrovascular Disorders in Adultsedicine. The breadth and scope of the pathologic factors are such that, even with the improved understanding of the involved events, it is extremely difficult to decide which of them are the primary instigators of the ensuing damage and which are but secondary effects of an ongoing process of the ne作者: facetious 時(shí)間: 2025-3-30 03:51 作者: Moderate 時(shí)間: 2025-3-30 04:29
Neurology and Psychiatry of Womention in the acute pathophysiology of central nervous system (CNS) injury and ischemia (i.e., stroke) (Braughler and Hall, 1989; Hall and Braughler, 1989). The 21-aminosteroids, or “l(fā)azaroids,” are a novel series of compounds being developed for the acute treatment of traumatic or ischemic CNS injury作者: Cytokines 時(shí)間: 2025-3-30 11:37 作者: Delectable 時(shí)間: 2025-3-30 16:09 作者: 尊敬 時(shí)間: 2025-3-30 18:30
Daniel Kondziella,Gunhild Waldemarstudies (Auer et al., 1984a)]. EEG silence is the harbinger of a series of biochemical events leading to hypoglycemic brain damage (Siesj?, 1988), structurally manifest as neuronal death (Auer et al., 1984b). Since hypoglycemia is one of the major effects of insulin administration, the very suggesti作者: mortuary 時(shí)間: 2025-3-30 21:05 作者: 對(duì)手 時(shí)間: 2025-3-31 01:55
