標(biāo)題: Titlebook: Early Life Origins of Health and Disease; E. Marelyn Wintour,Julie A. Owens Book 2006 The Editor(s) (if applicable) and The Author(s), und [打印本頁] 作者: LEVEE 時(shí)間: 2025-3-21 16:38
書目名稱Early Life Origins of Health and Disease影響因子(影響力)
書目名稱Early Life Origins of Health and Disease影響因子(影響力)學(xué)科排名
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書目名稱Early Life Origins of Health and Disease網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Early Life Origins of Health and Disease被引頻次
書目名稱Early Life Origins of Health and Disease被引頻次學(xué)科排名
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書目名稱Early Life Origins of Health and Disease讀者反饋
書目名稱Early Life Origins of Health and Disease讀者反饋學(xué)科排名
作者: frenzy 時(shí)間: 2025-3-21 21:03 作者: COLON 時(shí)間: 2025-3-22 03:19 作者: 軟弱 時(shí)間: 2025-3-22 07:03 作者: WITH 時(shí)間: 2025-3-22 09:57 作者: wangle 時(shí)間: 2025-3-22 14:53 作者: wangle 時(shí)間: 2025-3-22 19:12
Developmental Programming of Cardiovascular Dysfunctionsimilarity with the human data and may imply an important role in the aetiology of developmentally induced cardiovascular risk. Studies of inflammatory bio-markers, lipid profiles and compliance in animal models are too few to allow comparison. Increasing evidence for altered sympathetic activity in作者: 犬儒主義者 時(shí)間: 2025-3-22 22:58 作者: 制造 時(shí)間: 2025-3-23 04:08
Programming of Obesity—Experimental Evidenceetal programming recent experimental studies suggest that leptin and insulin resistance are critical endocrine defects in the pathogenesis of programming-induced obesity and metabolic disorders. Recent work has also shown that offspring of undernourished mothers display reduced locomotor activity in作者: 吸引人的花招 時(shí)間: 2025-3-23 06:05 作者: 聚集 時(shí)間: 2025-3-23 11:47
Programming Effects of Moderate and Binge Alcohol Consumptionanimal studies of fetal exposure to socially relevant amounts of alcohol are likely to grow in importance. Alcohol exposure is an important contributing factor to small birth weight in human populations and may also contribute to the production of the small birth weight phenotype. In addition, becau作者: 突襲 時(shí)間: 2025-3-23 15:23 作者: 獸皮 時(shí)間: 2025-3-23 21:34 作者: Halfhearted 時(shí)間: 2025-3-24 01:53
Hypoxia, Fetal Growth and Developmental Origins of Health and Diseaseassociation between low birth weight and increased risk of high blood pressure in adulthood, as first described by Barker, has literally exploded a new field of research investigating the effects of maternofetal nutrition on fetal growth, birth weight and subsequent cardiovascular disease. However, 作者: jocular 時(shí)間: 2025-3-24 05:41
Book 2006me. The book is a ‘must’ for all scientists interested in researching these areas, as there is a critical evaluation of the methodology used and suggestions for the ‘optimal’ way in which to investigate these phenomena. .作者: Enervate 時(shí)間: 2025-3-24 10:00 作者: flaggy 時(shí)間: 2025-3-24 12:16
https://doi.org/10.1007/978-3-662-13473-3logical rather than neurodevelopmental paradigms, if these programmed changes in neuromotor function and the mechanisms responsible are to be fully understood. We also introduce the idea that the impact on the individual of maladaptive neuromotor programming might be reduced by the development of ea作者: CRP743 時(shí)間: 2025-3-24 15:16 作者: 迷住 時(shí)間: 2025-3-24 21:23
https://doi.org/10.1007/978-3-662-24655-9that either balanced undernutrition or restriction of specific nutrients promotes metabolic and physiological disturbance and also relative adiposity in adult life. Intrauterine influences upon feeding, metabolism and the deposition of adipose tissue may well be mediated at the level of the hypothal作者: 易于 時(shí)間: 2025-3-25 00:00 作者: 套索 時(shí)間: 2025-3-25 04:01 作者: 毀壞 時(shí)間: 2025-3-25 08:04 作者: indubitable 時(shí)間: 2025-3-25 14:03
https://doi.org/10.1007/978-3-658-31992-2etal programming recent experimental studies suggest that leptin and insulin resistance are critical endocrine defects in the pathogenesis of programming-induced obesity and metabolic disorders. Recent work has also shown that offspring of undernourished mothers display reduced locomotor activity in作者: frenzy 時(shí)間: 2025-3-25 15:58
https://doi.org/10.1007/978-3-662-29437-6tions between small size at birth, neonatal catch-up growth and later metabolic disease. In this chapter we will review what has been learnt from human and experimental studies about the mechanistic basis for poor metabolic homeostasis following restricted fetal growth and neonatal catch-up growth, 作者: debris 時(shí)間: 2025-3-26 00:01 作者: 間接 時(shí)間: 2025-3-26 01:28
https://doi.org/10.1007/978-3-663-05478-8vels amongst women of reproductive age, and indeed, in pregnant women, places in sharp focus our scant understanding of the ramifications of this on offspring health, not only in the immediate neonatal period but, as a result of the recent spotlight on the early origins of adult disease and syndrome作者: Creatinine-Test 時(shí)間: 2025-3-26 06:00 作者: 鑲嵌細(xì)工 時(shí)間: 2025-3-26 08:27 作者: 防止 時(shí)間: 2025-3-26 13:56 作者: Duodenitis 時(shí)間: 2025-3-26 18:14 作者: Torrid 時(shí)間: 2025-3-26 21:28 作者: ligature 時(shí)間: 2025-3-27 04:11
https://doi.org/10.1007/978-3-663-05605-8eling complexes. Epigenetic marks are established early in development, are mitotically, and in some cases meiotically heritable, and can have a profound impact on an organism’s phenotype. Thus, epigenetic modifications provide a mechanism by which the permanent changes associated with fetal programming can occur.作者: arthroplasty 時(shí)間: 2025-3-27 06:00 作者: 輕彈 時(shí)間: 2025-3-27 13:06 作者: 上釉彩 時(shí)間: 2025-3-27 15:44 作者: 廢止 時(shí)間: 2025-3-27 19:05 作者: 消耗 時(shí)間: 2025-3-28 00:08
E. Marelyn Wintour,Julie A. OwensPresents a very up-to date and readable summary of our current understanding of the importance of life before birth, from conception to delivery, and in infancy and childhood, on the subsequent health作者: 開頭 時(shí)間: 2025-3-28 02:33
Advances in Experimental Medicine and Biologyhttp://image.papertrans.cn/e/image/300754.jpg作者: 北極人 時(shí)間: 2025-3-28 08:13
Early Life Origins of Health and Disease978-0-387-32632-0Series ISSN 0065-2598 Series E-ISSN 2214-8019 作者: extinct 時(shí)間: 2025-3-28 11:02 作者: Altitude 時(shí)間: 2025-3-28 16:25 作者: Extort 時(shí)間: 2025-3-28 21:55 作者: Frequency 時(shí)間: 2025-3-29 02:59
978-1-4419-3950-0The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Science+Busines作者: 輕而薄 時(shí)間: 2025-3-29 03:14
https://doi.org/10.1007/978-3-322-84357-9 Metabolic Syndrome. The fetal origins of adult disease hypothesis proposes that these associations reflect permanent changes in metabolism, body composition and tissue structure caused by undernutrition during critical periods of early development. An alternative hypothesis is that both small size 作者: 松果 時(shí)間: 2025-3-29 08:21 作者: incarcerate 時(shí)間: 2025-3-29 14:45
https://doi.org/10.1007/978-3-662-13473-3an motor system. Even less is known about how an adverse intrauterine event or environment might program motor learning, memory and function throughout the lifespan. We are only beginning to investigate how events during development of the brain and central nervous system might predispose some indiv作者: LAST 時(shí)間: 2025-3-29 17:48 作者: 情感 時(shí)間: 2025-3-29 22:07 作者: 谷類 時(shí)間: 2025-3-30 01:21
https://doi.org/10.1007/978-3-7091-5363-5ulthood. Epigenetic mechanisms, which regulate the broad diversity of tissue-specific gene expression, are established during development and largely maintained throughout adulthood. Hence, to the extent that nutrition during development affects the establishment of epigenetic gene regulatory mechan作者: Mitigate 時(shí)間: 2025-3-30 04:15 作者: 軍械庫 時(shí)間: 2025-3-30 11:25 作者: carotid-bruit 時(shí)間: 2025-3-30 16:25
https://doi.org/10.1007/978-3-663-15733-5ction and a range of adulthood indices of cardiovascular dysfunction and risk. Whilst the emphasis has been on the programming of hypertension, there is also evidence for an impact of the early life environment on later development of vascular endothelial dilator dysfunction and associated risk fact作者: 鋼盔 時(shí)間: 2025-3-30 19:41
https://doi.org/10.1007/978-3-662-33773-8ing epidemic proportions in some sectors of the community and is often found in association with the two most characterised adult onset diseases, hypertension and noninsulin dependent diabetes mellitus. Epidemiological studies in humans have identified various maternal states such as anemia, diabete作者: Efflorescent 時(shí)間: 2025-3-31 00:19 作者: Licentious 時(shí)間: 2025-3-31 04:56 作者: 富饒 時(shí)間: 2025-3-31 08:31
https://doi.org/10.1007/978-3-662-35040-9 exposure of the fetus to elevated levels of glucocorticoids, or exposure at inappropriate times, subtly disturbs normal fetal development. Experimental studies have demonstrated that late gestational exposure to excess glucocorticoids causes programming of a number of organ systems.作者: Genome 時(shí)間: 2025-3-31 10:44
Zur Physiologie und Hygiene der Luftfahrtes. These studies have generally utilized excessive alcohol exposure models, and examined resultant outcomes that are gross and can be measured in the offspring as a fetus or at birth. Very few studies have addressed the possible long term, or programming, effects of prenatal alcohol exposure. Even 作者: 你正派 時(shí)間: 2025-3-31 14:35 作者: Abjure 時(shí)間: 2025-3-31 20:04
Zur Regelung von Automobilmaschinenew, derive from adverse events in utero, such as maternal nutrition deficiency, infection and hypoxia. The hypothesis was originally derived from neuropathological changes in patients with established schizophrenia that are highly suggestive of impaired neural development occurring around mid-gestat作者: NAUT 時(shí)間: 2025-3-31 22:46 作者: 頌揚(yáng)國(guó)家 時(shí)間: 2025-4-1 04:57
