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標(biāo)題: Titlebook: Diabetes; An Old Disease, a Ne Shamim I. Ahmad Book 2013 The Editor(s) (if applicable) and The Author(s), under exclusive license to Spring [打印本頁(yè)]

作者: 強(qiáng)烈興趣    時(shí)間: 2025-3-21 19:34
書目名稱Diabetes影響因子(影響力)




書目名稱Diabetes影響因子(影響力)學(xué)科排名




書目名稱Diabetes網(wǎng)絡(luò)公開度




書目名稱Diabetes網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Diabetes被引頻次




書目名稱Diabetes被引頻次學(xué)科排名




書目名稱Diabetes年度引用




書目名稱Diabetes年度引用學(xué)科排名




書目名稱Diabetes讀者反饋




書目名稱Diabetes讀者反饋學(xué)科排名





作者: 挑剔小責(zé)    時(shí)間: 2025-3-21 21:08
Peiwei Li,Daniel Rodriguez Ramireza committee of the japan Diabetes Society in 2004. Intensive insulin therapy is a standard therapy. We should pay special attention to early development of microvascular complications in fulminant type 1 diabetes.
作者: 吃掉    時(shí)間: 2025-3-22 01:54

作者: 悠然    時(shí)間: 2025-3-22 04:35
https://doi.org/10.1007/978-3-031-25910-4tion in muscle. If elevated plasma fatty acid concentrations reduce endothelial nitric oxide synthase in the artery wall then this also could contribute to the increased atherosclerosis in diabetes. Also treatment of diabetes is discussed briefly in relation to cardiovascular disease.
作者: Galactogogue    時(shí)間: 2025-3-22 12:12
Cardiovascular Disease and Diabetes,tion in muscle. If elevated plasma fatty acid concentrations reduce endothelial nitric oxide synthase in the artery wall then this also could contribute to the increased atherosclerosis in diabetes. Also treatment of diabetes is discussed briefly in relation to cardiovascular disease.
作者: 矛盾    時(shí)間: 2025-3-22 14:35
Mindfulness in Behavioral Health system, periodontal disease, retinopathy, nephropathy, somatic and autonomic neuropathy, cardiovascular diseases and diabetic foot. Also included are the current management and treatments, and emerging therapies.
作者: 矛盾    時(shí)間: 2025-3-22 17:03

作者: STIT    時(shí)間: 2025-3-22 22:22
The Evolution of Online Booking Systemsn the adaptive and innate immune systems, with numerous cell types thought to contribute to pathogenesis. Like any complex disease, the variation in severity and incidence of T1DM can be attributed to a combination of genetic and environmental factors.
作者: BADGE    時(shí)間: 2025-3-23 04:31
Natural radionuclides in human haireve blood glucose, blood pressure and proteunuric goals, dietary protein and salt restriction, weight reduction, aggressive lipid lowering, smoking cessation and exercise. Multiple intensive interventions reduce cardiovascular events as well as nephropathy by about half when compared with conventional multifactorial treatment.
作者: 有限    時(shí)間: 2025-3-23 07:00

作者: 拖債    時(shí)間: 2025-3-23 12:42
J. McNeil,é. Doucet,J. -P. Chaputerance to the vicious circle of destructive insulitis. However, the events triggering islet auto immunity as well as the transition to overt diabetes are still largely unknown, making prevention and treatment strategies still a challenge.
作者: gorgeous    時(shí)間: 2025-3-23 17:07
https://doi.org/10.1007/978-3-031-25910-4 deals with the core elements promoting insulin resistance, associated with impaired insulin signalling pathway and adipocyte dysfunction. A detailed understanding of these basic pathophy siological mechanisms is critical for the development of novel therapeutic strategies to treat diabetes.
作者: 強(qiáng)行引入    時(shí)間: 2025-3-23 21:23
Introduction to Diabetes Mellitus, system, periodontal disease, retinopathy, nephropathy, somatic and autonomic neuropathy, cardiovascular diseases and diabetic foot. Also included are the current management and treatments, and emerging therapies.
作者: 暗語(yǔ)    時(shí)間: 2025-3-23 22:26

作者: DENT    時(shí)間: 2025-3-24 05:11
Impairment of Immune Systems in Diabetes,n the adaptive and innate immune systems, with numerous cell types thought to contribute to pathogenesis. Like any complex disease, the variation in severity and incidence of T1DM can be attributed to a combination of genetic and environmental factors.
作者: Euphonious    時(shí)間: 2025-3-24 08:01

作者: aptitude    時(shí)間: 2025-3-24 11:01
Glycemic Variability in Type 2 Diabetes Mellitus,red with focus on the mechanisms of tissue damage from this variability along with the oxidative stress. Additionally, treatment strategies to optimize HbA1. and glycemic variability with the goal of reducing risk of complications in persons with diabetes are reviewed.
作者: 粘    時(shí)間: 2025-3-24 15:47
,Immunobiology of β-Cell Destruction,erance to the vicious circle of destructive insulitis. However, the events triggering islet auto immunity as well as the transition to overt diabetes are still largely unknown, making prevention and treatment strategies still a challenge.
作者: falsehood    時(shí)間: 2025-3-24 21:22

作者: 暗語(yǔ)    時(shí)間: 2025-3-24 23:37

作者: 漫步    時(shí)間: 2025-3-25 04:54
Fulminant Type 1 Diabetes Mellitus,a committee of the japan Diabetes Society in 2004. Intensive insulin therapy is a standard therapy. We should pay special attention to early development of microvascular complications in fulminant type 1 diabetes.
作者: blackout    時(shí)間: 2025-3-25 10:35
Emerging Type 2 Diabetes in Young Adults,tcome, raising the possibility of a serious public health challenge in the next few decades. This chapter reviews the current understanding of the phenomenon of T2DM in the young adults and discusses the clinical challenges in managing this high risk group.
作者: 解脫    時(shí)間: 2025-3-25 14:25
0065-2598 ease and is also one of the most common.? It is very difficult to reach an accurate estimate for the global prevalence of diabetes since the standards and methods of data collection vary widely in different parts of the world. In addition, many potential sufferers are not included in the count becau
作者: anarchist    時(shí)間: 2025-3-25 16:54

作者: 孵卵器    時(shí)間: 2025-3-25 22:19

作者: arsenal    時(shí)間: 2025-3-26 00:13

作者: frozen-shoulder    時(shí)間: 2025-3-26 06:10
Fulminant Type 1 Diabetes Mellitus,ting in nearly no residual insulin secretion even just after the onset. The number of patients presumably amounts to 5,000~7,000 in Japan. The involvement of both, genetic background and viral infection, has been suggested in the pathogenesis of this disease. Diagnostic criteria were established by
作者: 斷斷續(xù)續(xù)    時(shí)間: 2025-3-26 09:27

作者: Cerumen    時(shí)間: 2025-3-26 14:47
Global Prevalence and Future of Diabetes Mellitus,hed by the International Diabetes Federation in 2011.. These include countries and territories in Europe, Africa, Middle East, North Africa, North America and Caribbean islands, South and Central America, South-East Asia and the Western Pacific Islands. Actual prevalence of DM (cases per 100 populat
作者: LIMN    時(shí)間: 2025-3-26 20:50
Type 2 Diabetes Mellitus, Pandemic in 21st Century,luent countries, is gradually afflicting also the developing world. This chapter shows the threat that the T2DM epidemic represents to mankind, with the astonishing recent discoveries on the role of obesity and of body fat in this metabolic disorder. Presently, the highest prevalence of T2DM is in S
作者: Ige326    時(shí)間: 2025-3-26 22:35
Emerging Type 2 Diabetes in Young Adults,demic driven by the increasing burden of obesity. Evidence is accumulating to suggest that this young diabetic cohort is an aggressive phenotype which leads to the premature development of complications that not only have impact on the quality of life but also unfavourably influence the long term ou
作者: UTTER    時(shí)間: 2025-3-27 03:19

作者: indicate    時(shí)間: 2025-3-27 07:49
Periodontal Disease and Diabetes,ciency with alteration in the metabolism of lipids, carbohydrates and proteins culminating with different manifestations in different organisms. In humans hyperglycemia is the main consequence of defects in the secretion and/or action of insulin, and its deregulation can produce secondary lesions in
作者: 闖入    時(shí)間: 2025-3-27 13:07

作者: BARK    時(shí)間: 2025-3-27 16:23
Nephropathy in Diabetes, the risk factors of Diabetic Nephropathy (DN). Hyperglycemia-induced metabolic and hemodynamic pathways are recognized to be mediators of kidney injury. Multiple biochemical pathways have been postulated that explain how hyperglycemia causes tissue damage: Non-enzymatic glycation that generates adv
作者: 表被動(dòng)    時(shí)間: 2025-3-27 18:07

作者: B-cell    時(shí)間: 2025-3-27 22:16

作者: accessory    時(shí)間: 2025-3-28 06:01

作者: Crohns-disease    時(shí)間: 2025-3-28 06:55
Diabetic Autonomic Neuropathy,rom endocrine factors, this neurological disease may cause symptoms, whose differential diagnosis needs a good knowledge of the whole internal medicine. DAN is strongly involved in the development of diabetic foot, ulceration and amputation. The life threatening consequences of cardiac and patient-f
作者: Infinitesimal    時(shí)間: 2025-3-28 13:42
,Immunobiology of β-Cell Destruction,of Langerhans. A susceptible genetic background is necessary, but not sufficient, for the development of the disease. Epidemiological and clinical observationsunderscore the importance of environmental factors as triggers of type 1 diabetes, currently under investigation. Islet-specific autoantibodi
作者: Interregnum    時(shí)間: 2025-3-28 15:35
Cardiovascular Disease and Diabetes,ans. Absence of insulin or insensitivity of tissues to insulin leads to hyperglycaemia and elevated plasma fatty acid concentration. Hyperglycaemia can lead to modification of protein functions which can contribute to accelerated atherosclerosis. The latter is the pathological condition which underl
作者: 全面    時(shí)間: 2025-3-28 22:14

作者: 肌肉    時(shí)間: 2025-3-29 02:30

作者: Critical    時(shí)間: 2025-3-29 03:41

作者: Hyperplasia    時(shí)間: 2025-3-29 11:12
978-1-4939-4672-3The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Science+Busines
作者: 剝皮    時(shí)間: 2025-3-29 14:29

作者: MONY    時(shí)間: 2025-3-29 17:39

作者: Ophthalmologist    時(shí)間: 2025-3-29 20:05
Peiwei Li,Daniel Rodriguez Ramirezting in nearly no residual insulin secretion even just after the onset. The number of patients presumably amounts to 5,000~7,000 in Japan. The involvement of both, genetic background and viral infection, has been suggested in the pathogenesis of this disease. Diagnostic criteria were established by
作者: 知道    時(shí)間: 2025-3-30 01:55

作者: CIS    時(shí)間: 2025-3-30 05:53
S. De Spirt,W. Stahl,U. Heinrichhed by the International Diabetes Federation in 2011.. These include countries and territories in Europe, Africa, Middle East, North Africa, North America and Caribbean islands, South and Central America, South-East Asia and the Western Pacific Islands. Actual prevalence of DM (cases per 100 populat
作者: 小步舞    時(shí)間: 2025-3-30 08:29

作者: 開花期女    時(shí)間: 2025-3-30 16:22
Dietary red ginseng and skin protectiondemic driven by the increasing burden of obesity. Evidence is accumulating to suggest that this young diabetic cohort is an aggressive phenotype which leads to the premature development of complications that not only have impact on the quality of life but also unfavourably influence the long term ou
作者: AGOG    時(shí)間: 2025-3-30 17:44
The Evolution of Online Booking Systemsangerhans. It is a complex process that results from the loss of tolerance to insulin and other β-cell-specific antigens. Various genetic and environmental factors have been studied so far, but precise causation has yet to be established. Numerous studies in rodents and human subjects have been perf
作者: 怕失去錢    時(shí)間: 2025-3-30 23:04

作者: 外向者    時(shí)間: 2025-3-31 02:18
https://doi.org/10.3920/978-90-8686-728-8of this chapter is to introduce diabetic retinopathy, a leading cause of blindness in people of the working age. The clinical course of retinopathy, anatomical changes, its pathogenesis and current treatment are described, followed by an overview of the emerging drug therapies for the potential trea
作者: arrogant    時(shí)間: 2025-3-31 06:38
Natural radionuclides in human hair the risk factors of Diabetic Nephropathy (DN). Hyperglycemia-induced metabolic and hemodynamic pathways are recognized to be mediators of kidney injury. Multiple biochemical pathways have been postulated that explain how hyperglycemia causes tissue damage: Non-enzymatic glycation that generates adv
作者: 反抗者    時(shí)間: 2025-3-31 11:12
Elizabeth M. Brooks,James Robertson) is a major complication of long standing diabetes, accounting for nearly 35% of all hospital admissions in diabetic climcs. It also accounts for nearly 80% of all nonlraumatic amputations of the lower limb. Due to its significant morbidity and mortality, DF has a staggering economic impact not onl
作者: interior    時(shí)間: 2025-3-31 15:23
Postprandial drowsiness in dyspepsialications. The relationship of hemoglobin HbA1. and other glycosylation end products (AGEs) to these complications, particularly microvascular disease, is well understood. More recent evidence suggests that glycemic variability may be associated with diabetes macrovascular complications. As HbA1. is
作者: Addictive    時(shí)間: 2025-3-31 18:49
J. McNeil,é. Doucet,J. -P. Chaput diverse clinical pictures. The most important outcome of somatic and autonomic DN are the development of diabetic foot followed by diabetic ulceration and possible amputation. In this chapter the definition, epidemiology, pathophysiology and classification of somatic DN will be discussed. Attention
作者: 減少    時(shí)間: 2025-4-1 00:46

作者: HAVOC    時(shí)間: 2025-4-1 02:04
J. McNeil,é. Doucet,J. -P. Chaputof Langerhans. A susceptible genetic background is necessary, but not sufficient, for the development of the disease. Epidemiological and clinical observationsunderscore the importance of environmental factors as triggers of type 1 diabetes, currently under investigation. Islet-specific autoantibodi
作者: 調(diào)整校對(duì)    時(shí)間: 2025-4-1 09:13
https://doi.org/10.1007/978-3-031-25910-4ans. Absence of insulin or insensitivity of tissues to insulin leads to hyperglycaemia and elevated plasma fatty acid concentration. Hyperglycaemia can lead to modification of protein functions which can contribute to accelerated atherosclerosis. The latter is the pathological condition which underl
作者: 抒情短詩(shī)    時(shí)間: 2025-4-1 14:03
Federica Buongiorno,Xenia Chiaramontetations, such as activation of the KRAS2 oncogene, inactivation of the tumor-suppressor gene CDKN2A, inactivation of the tumor-suppressor gene TP53 and deleted in pancreatic cancer 4 (.) gene defects are seen in those with pancreatic cancer. Approximately 80%of those patients, diagnosed with pancrea




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