標(biāo)題: Titlebook: DNA Polymorphisms as Disease Markers; D. J. Galton,G. Assmann (Director) Book 1991 Springer Science+Business Media New York 1991 DNA.Lipid [打印本頁] 作者: 浮淺 時(shí)間: 2025-3-21 17:04
書目名稱DNA Polymorphisms as Disease Markers影響因子(影響力)
書目名稱DNA Polymorphisms as Disease Markers影響因子(影響力)學(xué)科排名
書目名稱DNA Polymorphisms as Disease Markers網(wǎng)絡(luò)公開度
書目名稱DNA Polymorphisms as Disease Markers網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱DNA Polymorphisms as Disease Markers被引頻次
書目名稱DNA Polymorphisms as Disease Markers被引頻次學(xué)科排名
書目名稱DNA Polymorphisms as Disease Markers年度引用
書目名稱DNA Polymorphisms as Disease Markers年度引用學(xué)科排名
書目名稱DNA Polymorphisms as Disease Markers讀者反饋
書目名稱DNA Polymorphisms as Disease Markers讀者反饋學(xué)科排名
作者: PARA 時(shí)間: 2025-3-21 22:25 作者: sterilization 時(shí)間: 2025-3-22 04:18
A Candidate Gene Approach to the Genetics of Normal Lipid Variation: DNA Association Studies and Genors combined. In addition, the contributing genes do not act alone; rather they are interacting with other genes and environmental factors in determining disease risk. Therefore, the effect of a gene measured in one environment or population may be very different from its effect in another.作者: 躲債 時(shí)間: 2025-3-22 06:47 作者: 挖掘 時(shí)間: 2025-3-22 10:48
https://doi.org/10.1007/978-1-4020-9787-4 large aberrations in the DNA primary structure. A much more complicated situation is present in common disease where important roles for disease expression have been attributed to endogenous as well as exogenous factors (1).作者: penance 時(shí)間: 2025-3-22 13:07 作者: penance 時(shí)間: 2025-3-22 18:44
S. Z. Husain,V. H. Heywood,K. R. Markhamecretes VLDL particles and, similar to chylomicrons in the bloodstream, VLDLs are partly lipolyzed by LPL and the VLDLremnants or IDL particles formed are taken up with high affinity by the hepatic LDL receptors.作者: 不要不誠實(shí) 時(shí)間: 2025-3-22 21:53
Two Concepts of Opposition, Multiple Squareswo apo B abnormalities that cause hypercholesterolemia were investigated. The case of a family affected with defective apo B-100 and of a woman with severe hypercholesterolemia and hyperapobetalipoproteinemia are presented.作者: PACK 時(shí)間: 2025-3-23 02:24 作者: 服從 時(shí)間: 2025-3-23 06:23
Molecular Genetics Approach to Polygenic Disease—Initial Results from Atherosclerosis Research large aberrations in the DNA primary structure. A much more complicated situation is present in common disease where important roles for disease expression have been attributed to endogenous as well as exogenous factors (1).作者: 凹處 時(shí)間: 2025-3-23 09:54 作者: Chipmunk 時(shí)間: 2025-3-23 14:03 作者: FRONT 時(shí)間: 2025-3-23 21:59 作者: 蚊帳 時(shí)間: 2025-3-23 22:19 作者: 預(yù)定 時(shí)間: 2025-3-24 04:49 作者: 細(xì)頸瓶 時(shí)間: 2025-3-24 09:28 作者: 黃瓜 時(shí)間: 2025-3-24 14:44
978-1-4613-6640-9Springer Science+Business Media New York 1991作者: RECUR 時(shí)間: 2025-3-24 16:36 作者: 疲憊的老馬 時(shí)間: 2025-3-24 19:47
“To Implement Meaningful Change”Coronary atherosclerosis is a major cause of death in Western society and its pathology is as complex as it is important. There is no one agent responsible for all such atherogenesis, it is mostly a multifactorial disease and it can be the end product of many influences, both environmental and genetic.作者: 千篇一律 時(shí)間: 2025-3-25 01:36
Atherosclerosis: The Genetic Analysis of a Multi-Factorial DiseaseCoronary atherosclerosis is a major cause of death in Western society and its pathology is as complex as it is important. There is no one agent responsible for all such atherogenesis, it is mostly a multifactorial disease and it can be the end product of many influences, both environmental and genetic.作者: Cleave 時(shí)間: 2025-3-25 06:57
Compounds Derived from Aminoacetic Acidsoccurs. Differences between monogenic and common diseases are outlined. Utilization of intermediate phenotypes rather than of the common disease . as the diagnostic trait may be helpful for genetic analysis. Various genetic approaches with particular emphasis on linkage and association studies are d作者: Fibrillation 時(shí)間: 2025-3-25 09:05
Compounds Derived from Acetic Acidseases are the result of the interaction of numerous etiological factors, both genetic and environmental, each making a relatively small contribution to overall liability. Therefore, the contribution to disease risk of variation at any single gene is likely small when compared to all other risk fact作者: Chauvinistic 時(shí)間: 2025-3-25 13:09
https://doi.org/10.1007/978-1-4020-9787-4sease. This success in understanding inborn errors of metabolism, however, has been largely restricted to monogenic disease or to defects that involve large aberrations in the DNA primary structure. A much more complicated situation is present in common disease where important roles for disease expr作者: 搜集 時(shí)間: 2025-3-25 16:02
F. Chialva,G. Gabri,P. A. P. Liddle,F. Ulianen identified by cDNA cloning, at least two of which (GLUT 1 and GLUT 4) are expressed in insulin-sensitive tissues. The GLUT 1 CHepG2-type) transporter is the more widely distributed of these two proteins. It provides many cells with their basal glucose requirement for ATP production and the biosyn作者: 退潮 時(shí)間: 2025-3-25 23:00
13C-NMR Analysis of Essential Oilscy varies in degree, being complete in insulin dependent diabetes (IDDM), and relative in non-insulin dependent diabetes (NIDDM). The strong association of certain HLA immune-response genes on chromosome 6 with IDDM provides a prominent genetic marker to distinguish these two types of diabetes (Truc作者: impale 時(shí)間: 2025-3-26 01:32 作者: Ondines-curse 時(shí)間: 2025-3-26 04:48 作者: Arboreal 時(shí)間: 2025-3-26 10:05
https://doi.org/10.1007/978-3-0348-0379-3ns of fasting subjects resulting in a pronounced increase in plasma triglycerides .. Familial deficiencies of lipoprotein lipase (LPL) . and of apolipoprotein C-II ., a polypeptide cofactor of LPL, are well-defined causes of this syndrome. LPL, the enzyme most frequently reduced or absent in chylomi作者: perimenopause 時(shí)間: 2025-3-26 16:26
https://doi.org/10.1007/978-1-137-05175-2f first degree relatives of 121 men and 96 women with CAD showed the increased risk of death from the disease was five and seven fold greater than in matched controls for males and females respectively.. Many subsequent studies have confirmed this trend.. Further evidence for the implication of a ge作者: caldron 時(shí)間: 2025-3-26 19:56 作者: 平躺 時(shí)間: 2025-3-26 22:49
“Disharmony and Religious Feuds”d in intergenic and intronic DNA. As many as 1:300 bases may be variable (1). This creates a wealth of new genetic markers that can be used to track almost any locus in pedigree or population studies. Clinical genetics has been transformed from a study of pedigrees in which rare mutant proteins co-s作者: Priapism 時(shí)間: 2025-3-27 05:09
Compounds Derived from Aminoacetic Acidsoccurs. Differences between monogenic and common diseases are outlined. Utilization of intermediate phenotypes rather than of the common disease . as the diagnostic trait may be helpful for genetic analysis. Various genetic approaches with particular emphasis on linkage and association studies are discussed.作者: 新義 時(shí)間: 2025-3-27 07:58 作者: laparoscopy 時(shí)間: 2025-3-27 11:08 作者: terazosin 時(shí)間: 2025-3-27 17:29
“Dissatisfaction Evidenced by Some”interaction. The results of these studies suggest that genetic liability to CHD is influenced significantly by a locus or loci in the apo AI/CIII/AIV gene region. Since the association of this region with familial CHD involves different alleles or haplotypes in different sections of the population (作者: 山間窄路 時(shí)間: 2025-3-27 20:38 作者: Mendicant 時(shí)間: 2025-3-27 23:46
HDL and Reverse Transport of Cholesterol: Insights from Mutants作者: Budget 時(shí)間: 2025-3-28 05:11
Genetic Variation at the APOA-I, C-III, A-IV Gene Complex: A Critical Review of the Associations Bet作者: 連鎖 時(shí)間: 2025-3-28 09:29 作者: 王得到 時(shí)間: 2025-3-28 10:24
Lipoprotein Lipase Gene Variants in Subjects with Hypertriglyceridaemia and Coronary Atherosclerosisobesity, it has also been demonstrated to have a strong genetic component. Segregational analysis in certain pedigrees reveals a bimodal triglyceride distribution consistent with autosomal dominant inheritance..作者: 運(yùn)動(dòng)的我 時(shí)間: 2025-3-28 14:52 作者: OREX 時(shí)間: 2025-3-28 20:55 作者: BARGE 時(shí)間: 2025-3-29 00:08 作者: athlete’s-foot 時(shí)間: 2025-3-29 06:20
olved in the mapping of these DNA mutations to meet together to discuss the implications of their studies. The main issues are whether such DNA polymorphisms will lead to an identification of major aetiological loci and which are the best techniques to achieve this? What is the cause of the differences in fre978-1-4613-6640-9978-1-4615-3690-1作者: CANDY 時(shí)間: 2025-3-29 08:48 作者: neuron 時(shí)間: 2025-3-29 13:34 作者: 不規(guī)則 時(shí)間: 2025-3-29 16:01
Molecular Genetics Approach to Polygenic Disease—Initial Results from Atherosclerosis Researchsease. This success in understanding inborn errors of metabolism, however, has been largely restricted to monogenic disease or to defects that involve large aberrations in the DNA primary structure. A much more complicated situation is present in common disease where important roles for disease expr作者: 亂砍 時(shí)間: 2025-3-29 21:47 作者: Incorruptible 時(shí)間: 2025-3-30 02:44
Molecular Genetic Approach to Polygenic Disease: Non-Insulin Dependent Diabetes Mellitus (NIDDM)cy varies in degree, being complete in insulin dependent diabetes (IDDM), and relative in non-insulin dependent diabetes (NIDDM). The strong association of certain HLA immune-response genes on chromosome 6 with IDDM provides a prominent genetic marker to distinguish these two types of diabetes (Truc作者: 大吃大喝 時(shí)間: 2025-3-30 04:36 作者: 嘮叨 時(shí)間: 2025-3-30 09:02
Abnormalities of Apolipoprotein B Metabolism in the Lipid Clinicbolism. Elevated LDL cholesterol concentrations may derive either from a defective clearance of LDL or from a reduced removal. In the present report two apo B abnormalities that cause hypercholesterolemia were investigated. The case of a family affected with defective apo B-100 and of a woman with s作者: 糾纏,纏繞 時(shí)間: 2025-3-30 16:12 作者: 怪物 時(shí)間: 2025-3-30 16:32 作者: NOMAD 時(shí)間: 2025-3-30 21:21
RFLP Markers of Familial Coronary Heart Disease in close relatives before age 60 was strongly associated with the allele status of four RFLP in the apo AI/CIII/AIV gene region. In a simultaneous multiple logistic regression analysis of CHD on a number of covariates, a family history emerged with significant odds ratio of 2.553 (p = 0.034) in sub作者: 套索 時(shí)間: 2025-3-31 04:46 作者: Redundant 時(shí)間: 2025-3-31 08:27 作者: 條街道往前推 時(shí)間: 2025-3-31 10:33 作者: Ingredient 時(shí)間: 2025-3-31 15:46
