標(biāo)題: Titlebook: Cytokines and Joint Injury; Wim B. Berg,Pierre Miossec Book 2004 Springer Basel AG 2004 Arthritis.bone.cytokine.cytokines.diseases.inflamm [打印本頁] 作者: Nixon 時(shí)間: 2025-3-21 17:16
書目名稱Cytokines and Joint Injury影響因子(影響力)
書目名稱Cytokines and Joint Injury影響因子(影響力)學(xué)科排名
書目名稱Cytokines and Joint Injury網(wǎng)絡(luò)公開度
書目名稱Cytokines and Joint Injury網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Cytokines and Joint Injury被引頻次
書目名稱Cytokines and Joint Injury被引頻次學(xué)科排名
書目名稱Cytokines and Joint Injury年度引用
書目名稱Cytokines and Joint Injury年度引用學(xué)科排名
書目名稱Cytokines and Joint Injury讀者反饋
書目名稱Cytokines and Joint Injury讀者反饋學(xué)科排名
作者: Toxoid-Vaccines 時(shí)間: 2025-3-21 21:32 作者: SUE 時(shí)間: 2025-3-22 02:15 作者: Foolproof 時(shí)間: 2025-3-22 04:53 作者: 摻和 時(shí)間: 2025-3-22 11:40 作者: eucalyptus 時(shí)間: 2025-3-22 13:17 作者: eucalyptus 時(shí)間: 2025-3-22 17:11
Robert J. Klaassen MD,Nancy L. Young PhD historical approach to the definition of TNFα as a therapeutic target that has heralded three blockbuster drugs and consolidated the future of ‘biodrugs’ as an important sector of future pharmaceutical products.作者: 規(guī)范就好 時(shí)間: 2025-3-22 22:25
Jochen H. O. Hoffmann,Alexander H. Enkd immunocompetent cells into the extravascular space. IL-1 is also an angiogenic factor and plays a role in tumor metastasis and blood vessel supply [.]. Mice lacking IL-1 receptors fail to develop proliferative lesions of vascular smooth muscle cells in mechanically injured arteries. In humans with作者: crescendo 時(shí)間: 2025-3-23 02:13
Current IgG Products and Future Perspectiveshe human. An important distinction between these two molecules is that pro-IL-1β is biologically inactive, whereas both pro-IL-lα and mature IL-lα exhibit full receptor binding activity. In addition, to its biologic effects following receptor binding, pro-IL-lα exerts other functions inside cells [.作者: 不利 時(shí)間: 2025-3-23 08:48 作者: 上下連貫 時(shí)間: 2025-3-23 11:40
Jeffrey Wang,Wei-Chiang Shen,Jennica L. Zaror, non-or partial-responder patients are not infrequent and inflammatory disease usually flares upon discontinuation of treatment [., .]. The fact that TNFα independent pathways operate in inflammatory synovitis carries significant pathogenetic implications for existing disease models and exemplifie作者: DAFT 時(shí)間: 2025-3-23 16:00
Junyan A. Ji,Jun Liu,Y. John Wangnd direct cellular interactions [.-.]. In pathologic conditions, the production of cytokines and MMP by infiltrating and resident tissue cells escapes regulatory mechanisms. The activity of proinflammatory cytokines is counterbalanced by numerous mechanisms of which cytokine inhibitors — IL-1 recept作者: paleolithic 時(shí)間: 2025-3-23 18:54 作者: 歡笑 時(shí)間: 2025-3-23 22:20 作者: 小教堂 時(shí)間: 2025-3-24 04:41
Book 2004es IL-15, IL-17 and IL-18 and their positioning in the complex cytokine interplay. Cytokine regulation of destructive enzymes, RANKL, the endogenous inhibitor OPG and their crucial roles as central players in joint erosion are highlighted. Together, the chapters provide a complete and balanced view 作者: Fortuitous 時(shí)間: 2025-3-24 08:53 作者: 商業(yè)上 時(shí)間: 2025-3-24 13:27
The IL-1 family: The role of IL-1 and IL-18 in inflammation,d immunocompetent cells into the extravascular space. IL-1 is also an angiogenic factor and plays a role in tumor metastasis and blood vessel supply [.]. Mice lacking IL-1 receptors fail to develop proliferative lesions of vascular smooth muscle cells in mechanically injured arteries. In humans with作者: HERTZ 時(shí)間: 2025-3-24 17:15
Targeted IL-1 inhibition,he human. An important distinction between these two molecules is that pro-IL-1β is biologically inactive, whereas both pro-IL-lα and mature IL-lα exhibit full receptor binding activity. In addition, to its biologic effects following receptor binding, pro-IL-lα exerts other functions inside cells [.作者: 宿醉 時(shí)間: 2025-3-24 21:22 作者: Redundant 時(shí)間: 2025-3-25 00:34 作者: 頭盔 時(shí)間: 2025-3-25 04:29
Cell-cell contact in chronic inflammation: the importance to cytokine regulation in tissue destructnd direct cellular interactions [.-.]. In pathologic conditions, the production of cytokines and MMP by infiltrating and resident tissue cells escapes regulatory mechanisms. The activity of proinflammatory cytokines is counterbalanced by numerous mechanisms of which cytokine inhibitors — IL-1 recept作者: 騎師 時(shí)間: 2025-3-25 11:31
The role of proteinases in joint tissue destruction,ng growth and development, synthesis of matrix components exceeds the rate of degradation; reduction in the rate of matrix synthesis and an increase in the rate of degradation occur during matrix resorption [.] .作者: 責(zé)怪 時(shí)間: 2025-3-25 11:38 作者: Postmenopause 時(shí)間: 2025-3-25 17:33 作者: defuse 時(shí)間: 2025-3-25 22:52 作者: 拋射物 時(shí)間: 2025-3-26 02:34
The IL-1 family: The role of IL-1 and IL-18 in inflammation, inflammation and autoimmune diseases. The most salient and relevant properties of IL-1 in inflammation are the initiation of cyclooxygenase type 2 (COX-2), type 2 phospholipase A and inducible nitric oxide syntha se (iNOS). This accounts for the large amount of prostaglandin-E. (PGE.), platelet act作者: Bother 時(shí)間: 2025-3-26 06:13 作者: 憤憤不平 時(shí)間: 2025-3-26 12:14
Is IL-6 a therapeutic target?, the multiple joints [.]. The pathological features of the affected joints consist of hyperplasia of inflamed synovium and erosion of cartilage and bone where the pannus tissue invades. There are numerous immunocompetent cells such as T cells, macrophages, dendritic cells and plasma cells infiltrati作者: etidronate 時(shí)間: 2025-3-26 13:23
The clinical use of anti-cytokine therapy in rheumatic diseases,. Although the precise aetiopathogenesis of these inflammatory arthropathies have not been elucidated, inflammation and damage are common features. Active joint inflammation results in pain, stiffness and dysfunction. Chronic synovitis leads to cartilage and bone damage through mechanisms that have 作者: 看法等 時(shí)間: 2025-3-26 17:36 作者: Fretful 時(shí)間: 2025-3-26 21:26 作者: 廣大 時(shí)間: 2025-3-27 02:54
Cell-cell contact in chronic inflammation: the importance to cytokine regulation in tissue destruct cells, neutrophils and mononuclear phagocytes. This influx of inflammatory cells in the target tissue is associated with the proliferation of invading and resident cells and frequently with destruction and remodeling of the extracellular matrix. In the joint structures, the destruction of the organ作者: 公式 時(shí)間: 2025-3-27 09:05
The role of proteinases in joint tissue destruction,ne can be destroyed. Cartilage contains different types of collagen [.], rod-shaped molecules that aggregate in a staggered array to form cross-linked fibers giving connective tissues strength and rigidity. Trapped between these collagen fibers are the aggrecan molecules [.] that consist of three gl作者: Urea508 時(shí)間: 2025-3-27 11:16 作者: 發(fā)怨言 時(shí)間: 2025-3-27 14:51
Cytokine regulation of bone destruction,s in humans is being remodeled [.]. The growth, development and maintenance of bone is a highly regulated process [.]. Morphogenesis and remodeling of bone involve coordinate regulation of bone-forming cells (osteoblasts) and bone-resorbing cells (osteoclasts) [.,.]. Osteoblasts and osteoclasts aris作者: 放逐 時(shí)間: 2025-3-27 18:07 作者: semiskilled 時(shí)間: 2025-3-28 01:32
Jochen H. O. Hoffmann,Alexander H. Enk inflammation and autoimmune diseases. The most salient and relevant properties of IL-1 in inflammation are the initiation of cyclooxygenase type 2 (COX-2), type 2 phospholipase A and inducible nitric oxide syntha se (iNOS). This accounts for the large amount of prostaglandin-E. (PGE.), platelet act作者: 保存 時(shí)間: 2025-3-28 03:05 作者: 增強(qiáng) 時(shí)間: 2025-3-28 07:13 作者: 我要威脅 時(shí)間: 2025-3-28 13:12
https://doi.org/10.1007/978-3-319-13081-1. Although the precise aetiopathogenesis of these inflammatory arthropathies have not been elucidated, inflammation and damage are common features. Active joint inflammation results in pain, stiffness and dysfunction. Chronic synovitis leads to cartilage and bone damage through mechanisms that have 作者: 用樹皮 時(shí)間: 2025-3-28 15:52 作者: 系列 時(shí)間: 2025-3-28 21:05
Linker Design for Antibody–Drug Conjugatesmmatory patterns associated with rheumatoid arthritis (RA). At the same time, the list of additional cytokines with similar biological effects has been growing extensively. Among these, IL-17 is of interest since it shares properties and interacts with TNF? and IL-1. In addition, being produced by T作者: chandel 時(shí)間: 2025-3-29 01:50 作者: mechanical 時(shí)間: 2025-3-29 07:01 作者: 重疊 時(shí)間: 2025-3-29 10:30
https://doi.org/10.1007/978-1-4614-5456-4e complementary forces of dark (yin) and light (yang) govern all aspects of life. Yin and yang convey the interdependence of opposites. Such reciprocity is a common feature of many biological processes - including skeletal maintenance.作者: 痛得哭了 時(shí)間: 2025-3-29 13:22
Cancer Drug Discovery and Developments in humans is being remodeled [.]. The growth, development and maintenance of bone is a highly regulated process [.]. Morphogenesis and remodeling of bone involve coordinate regulation of bone-forming cells (osteoblasts) and bone-resorbing cells (osteoclasts) [.,.]. Osteoblasts and osteoclasts aris作者: LINE 時(shí)間: 2025-3-29 19:23 作者: ULCER 時(shí)間: 2025-3-29 19:43 作者: 剝削 時(shí)間: 2025-3-30 02:08
,Receptor activator of nuclear factor кB ligand (RANKL) , osteoprotegerin (OPG): the yin and yang ofe complementary forces of dark (yin) and light (yang) govern all aspects of life. Yin and yang convey the interdependence of opposites. Such reciprocity is a common feature of many biological processes - including skeletal maintenance.作者: Fatten 時(shí)間: 2025-3-30 06:43
Wim B. Berg,Pierre MiossecGives a balanced view on central cytokines and joint pathology.International authorship作者: 現(xiàn)代 時(shí)間: 2025-3-30 12:11 作者: 半導(dǎo)體 時(shí)間: 2025-3-30 12:40
https://doi.org/10.1007/978-3-0348-7883-8Arthritis; bone; cytokine; cytokines; diseases; inflammation; joint; necrosis; osteoarthritis; pathology; prot作者: ostrish 時(shí)間: 2025-3-30 19:40
978-3-0348-9609-2Springer Basel AG 2004
