標(biāo)題: Titlebook: Cytokine Frontiers; Regulation of Immune Takayuki Yoshimoto,Tomohiro Yoshimoto Book 2014 Springer Japan 2014 Anti-inflammatory cytokines.Pr [打印本頁(yè)] 作者: 適婚女孩 時(shí)間: 2025-3-21 17:44
書目名稱Cytokine Frontiers影響因子(影響力)
書目名稱Cytokine Frontiers影響因子(影響力)學(xué)科排名
書目名稱Cytokine Frontiers網(wǎng)絡(luò)公開度
書目名稱Cytokine Frontiers網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Cytokine Frontiers被引頻次
書目名稱Cytokine Frontiers被引頻次學(xué)科排名
書目名稱Cytokine Frontiers年度引用
書目名稱Cytokine Frontiers年度引用學(xué)科排名
書目名稱Cytokine Frontiers讀者反饋
書目名稱Cytokine Frontiers讀者反饋學(xué)科排名
作者: 煞費(fèi)苦心 時(shí)間: 2025-3-21 21:32 作者: 矛盾 時(shí)間: 2025-3-22 04:18
The Roles of IL-17A and IL-17F in Infection and Inflammatory Disorders17A among the IL-17 family and binds to the same IL-17 receptor A (IL-17RA) and IL-17RC receptor complex as IL-17A. Recent studies have demonstrated that IL-17A and IL-17F are also produced by multiple lineages of immune cells, including γδ T cells, NKT cells, and innate lymphoid cells. These cytoki作者: adipose-tissue 時(shí)間: 2025-3-22 05:33 作者: 獨(dú)輪車 時(shí)間: 2025-3-22 09:48 作者: 嚙齒動(dòng)物 時(shí)間: 2025-3-22 14:41
Interleukin-22: A Bridge Between Epithelial Innate Host Defense and Immune Cellsipheral tissue epithelial cells. IL-22 functions as a unique messenger from immune system to tissue epithelial cells and to regulate homeostasis of epithelia. IL-22 is able to directly enhance antimicrobial defense mechanisms in epithelial cells and to facilitate epithelial barrier repair and wound 作者: 嚙齒動(dòng)物 時(shí)間: 2025-3-22 20:04 作者: 事情 時(shí)間: 2025-3-23 00:47
IL-36: An Epithelial Cytokine Important in Psoriasisby epithelial tissues. Similar to other IL-1 family cytokines, they promote inflammatory responses by activating kinases such as JNK, p38, and the IKKs, and activating transcription factors such as NFκB. They only do this, however, after N-terminal processing, which increases their specific activity作者: 紅潤(rùn) 時(shí)間: 2025-3-23 04:35
Anti-TNF Therapy: 20 Years from Our First Therapeutic Adventuredisease tissue in rheumatoid arthritis and on exploring the dysregulation of cytokine expression within that tissue..From those first successful clinical trials we conducted in 1992 with infliximab, which were widely publicized more than a year before publication, a competition emerged between a set作者: Rct393 時(shí)間: 2025-3-23 09:29 作者: BARGE 時(shí)間: 2025-3-23 11:40 作者: Neolithic 時(shí)間: 2025-3-23 16:28
Thymic Stromal Lymphopoietin (TSLP)h hematopoietic and nonhematopoietic cell lineages, including dendritic cells (DCs), basophils, eosinophils, mast cells, CD4., CD8., and natural killer (NK) T cells, B cells, and epithelial cells. Although the role of TSLP in the promotion of TH2 responses has been extensively studied in the context作者: INCUR 時(shí)間: 2025-3-23 18:26 作者: 倫理學(xué) 時(shí)間: 2025-3-24 02:16
Interleukin-27: Regulation of Immune Responses and Disease Development by a Pleiotropic Cytokine witti-inflammatory functions depending on the context. IL-27 promotes early helper T (Th)1 differentiation and generation of cytotoxic T cells and IL-10-producing regulatory T cells. In addition, IL-27 inhibits differentiation of CD4. T cells into Th1, Th2, Th9, and Th17 cells; suppresses their respons作者: ventilate 時(shí)間: 2025-3-24 02:23
Interleukin-35: A Novel Mediator of Peripheral Toleranceing cells (APCs), IL-35 is produced by regulatory T (Treg) cells and suppresses cell proliferation. It has been shown to play an important role in many disease models and has been recently shown to have additional functions aside from inhibition of proliferation, including inducing its own expressio作者: 青春期 時(shí)間: 2025-3-24 09:34 作者: cloture 時(shí)間: 2025-3-24 11:33 作者: 不適 時(shí)間: 2025-3-24 16:22 作者: paleolithic 時(shí)間: 2025-3-24 22:41 作者: 易于交談 時(shí)間: 2025-3-25 02:46 作者: 傾聽 時(shí)間: 2025-3-25 06:07
The Global and Regional Climate System molecular structures, receptor structures, and signal transduction pathways. Each cytokine, however, acts on distinct types of cells that express their specific receptor. The functional IL-18 receptor is specifically induced by cytokines such as IL-2 and IL-12. During innate immune responses, IL-1β作者: cornucopia 時(shí)間: 2025-3-25 10:39 作者: 我怕被刺穿 時(shí)間: 2025-3-25 15:06 作者: 頂點(diǎn) 時(shí)間: 2025-3-25 16:49
https://doi.org/10.1007/978-3-540-38819-7ral to the pathogenesis of many immune-mediated inflammatory disorders and was shown to act via a novel IL-17-producing T helper subset, the Th17 cell. Many innate cells also respond to IL-23 and are important in both resistance to infection and in mediating autoimmune pathology. These cells are cha作者: Mettle 時(shí)間: 2025-3-25 21:16
G. Sundstr?m,? Larsson,M. Tarkpeaby epithelial tissues. Similar to other IL-1 family cytokines, they promote inflammatory responses by activating kinases such as JNK, p38, and the IKKs, and activating transcription factors such as NFκB. They only do this, however, after N-terminal processing, which increases their specific activity作者: Keshan-disease 時(shí)間: 2025-3-26 03:59
R. F. Addison,A. B. McKague,C. C. Waldendisease tissue in rheumatoid arthritis and on exploring the dysregulation of cytokine expression within that tissue..From those first successful clinical trials we conducted in 1992 with infliximab, which were widely publicized more than a year before publication, a competition emerged between a set作者: heirloom 時(shí)間: 2025-3-26 07:06
https://doi.org/10.1007/978-3-540-38522-6it has been demonstrated that IL-25 (IL-17E) is involved in the initiation and amplification of type 2 immune responses. Recent studies have shown that a number of cell types including activated Th2 cells, epithelial cells, mast cells, eosinophils, and macrophages produce IL-25 in response to a vari作者: ORBIT 時(shí)間: 2025-3-26 10:23
R. Anliker,G. C. Butler,V. Zitkoere it acts as an alarmin by binding to the receptor ST2. The receptor ST2 is widely expressed in the human body, and IL-33 exerts a wide range of biological activities depending on the target cells and cellular context. IL-33 is essential for host protection from helminth infections by promoting ty作者: organic-matrix 時(shí)間: 2025-3-26 15:56
Lóránt Dávid,Zoltán Ilyés,Zoltán Barosh hematopoietic and nonhematopoietic cell lineages, including dendritic cells (DCs), basophils, eosinophils, mast cells, CD4., CD8., and natural killer (NK) T cells, B cells, and epithelial cells. Although the role of TSLP in the promotion of TH2 responses has been extensively studied in the context作者: 秘密會(huì)議 時(shí)間: 2025-3-26 20:42
Lóránt Dávid,Zoltán Ilyés,Zoltán Barosic cells, B cells, and T cells. A major role of IL-10 is to act as a feedback regulator of the immune response by inhibiting the production of inflammatory cytokines by innate cells such as macrophages and dendritic cells. Thus, the production of IL-10 in response to microbial flora is required to i作者: 思考才皺眉 時(shí)間: 2025-3-26 22:22 作者: 多嘴 時(shí)間: 2025-3-27 02:15 作者: 珊瑚 時(shí)間: 2025-3-27 07:03 作者: Inoperable 時(shí)間: 2025-3-27 11:51 作者: 即席 時(shí)間: 2025-3-27 17:34 作者: 不要嚴(yán)酷 時(shí)間: 2025-3-27 18:34 作者: laceration 時(shí)間: 2025-3-28 00:55
The Roles of IL-17A and IL-17F in Infection and Inflammatory Disordersal and fungal infections. However, it is now clear that IL-17A and IL-17F have overlapping—yet distinct—roles in innate and adoptive immune responses. Here, we describe the recent data on the roles of IL-17A and IL-17F in inflammatory disorders and host defense and discuss the potential of these molecules as therapeutic targets.作者: Prognosis 時(shí)間: 2025-3-28 03:12 作者: Myofibrils 時(shí)間: 2025-3-28 07:18
The Global and Regional Climate System and its receptor were only identified in 2000, a tremendous amount has been learned already, leading to the development of potential therapeutic approaches to cancer as well as potentially allowing for the specific manipulation of IL-21 effects in a spectrum of autoimmune diseases, allergic responses, and viral infections.作者: hypnogram 時(shí)間: 2025-3-28 11:04 作者: 真實(shí)的人 時(shí)間: 2025-3-28 18:27
S. Lustig,P. Schramel,Ph. Quevauvillery disease models and has been recently shown to have additional functions aside from inhibition of proliferation, including inducing its own expression in non-Treg cells. In this chapter, we discuss the history and current status of IL-35 biology, as well as suggest where the field might move in the future.作者: NEX 時(shí)間: 2025-3-28 19:53 作者: 燈絲 時(shí)間: 2025-3-28 22:58
Lóránt Dávid,Zoltán Ilyés,Zoltán Barosmune response to a pathogen which could result in chronic infection. Regulation of the immune response by IL-10 has been shown to play a role in control of allergic, inflammatory, and autoimmune diseases, and further knowledge may advance our ability to use antagonists or agonists of IL-10 for immune therapies.作者: 使激動(dòng) 時(shí)間: 2025-3-29 06:59
C. Dietl,D. Laschka,M. W?ber,L. Peichlity against a variety of mouse and human tumors and suppresses the development of inflammatory, allergic, and autoimmune diseases. In this chapter, we review the molecular characterization of IL-27 and its receptor, its pro- and anti-inflammatory properties, and the therapeutic implications.作者: 清晰 時(shí)間: 2025-3-29 10:05 作者: Ossification 時(shí)間: 2025-3-29 13:57
Interleukin-10: Cytokines in Anti-inflammation and Tolerancemune response to a pathogen which could result in chronic infection. Regulation of the immune response by IL-10 has been shown to play a role in control of allergic, inflammatory, and autoimmune diseases, and further knowledge may advance our ability to use antagonists or agonists of IL-10 for immune therapies.作者: conservative 時(shí)間: 2025-3-29 17:27 作者: medieval 時(shí)間: 2025-3-29 19:48 作者: 樹木心 時(shí)間: 2025-3-30 03:17
The Interleukin-1 Familyne is present in most cells and readily released upon cell death. Although treatment for autoimmune diseases often includes immunosuppressive drugs, blocking the IL-1 receptor is effective as an anti-inflammatory therapy for either IL-1α or IL-1β.作者: Fretful 時(shí)間: 2025-3-30 07:16 作者: 錯(cuò)誤 時(shí)間: 2025-3-30 09:12 作者: 賭博 時(shí)間: 2025-3-30 15:31
Interleukin-22: A Bridge Between Epithelial Innate Host Defense and Immune Cellsd, as a cytokine, IL-22 is capable of induction of proinflammatory responses, especially in synergy with other cytokines. Consequently, IL-22 contributes to pathogenesis of certain inflammatory diseases for example psoriasis.作者: Arthropathy 時(shí)間: 2025-3-30 20:25 作者: 反饋 時(shí)間: 2025-3-30 23:46
