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標(biāo)題: Titlebook: Clostridium difficile; Methods and Protocol Peter Mullany,Adam P. Roberts Book 2010 Springer Science+Business Media, LLC 2010 DNA.Microarra [打印本頁]

作者: CYNIC    時間: 2025-3-21 19:14
書目名稱Clostridium difficile影響因子(影響力)




書目名稱Clostridium difficile影響因子(影響力)學(xué)科排名




書目名稱Clostridium difficile網(wǎng)絡(luò)公開度




書目名稱Clostridium difficile網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Clostridium difficile被引頻次




書目名稱Clostridium difficile被引頻次學(xué)科排名




書目名稱Clostridium difficile年度引用




書目名稱Clostridium difficile年度引用學(xué)科排名




書目名稱Clostridium difficile讀者反饋




書目名稱Clostridium difficile讀者反饋學(xué)科排名





作者: TOXIN    時間: 2025-3-21 22:38

作者: 膽小懦夫    時間: 2025-3-22 03:57

作者: 壟斷    時間: 2025-3-22 05:25
,Synopsis: Schizophrenie — was ist das?,ell as its own. TcdE has high homologies with bacteriophage holin proteins. TcdC negatively regulates toxin synthesis by interfering with the RNA polymerase formed with TcdR. Therefore, TcdR and TcdC constitute specific regulators of toxin gene transcription thereby tightly regulating toxin synthesi
作者: overture    時間: 2025-3-22 10:54
Daniel Hell,Magret Fischer-Gestefeld Nevertheless, whole-scale genome sequencing remains an expensive endeavour and such comparisons are limited to only a handful of strains. DNA microarrays represent an alternative technology for whole genome comparisons enabling a “birds eye view” of all the genes absent or present in a given genome
作者: BLA    時間: 2025-3-22 15:50
Was verursacht Schizophrenien?, chromosome via a single crossover event. As an alternative, recombination-independent strategies have been devised that are reliant upon a re-targeted group II intron. One element in particular, the ClosTron, provides the facility for the positive selection of insertional mutants. The generation of
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作者: 癡呆    時間: 2025-3-23 02:41
Isolation and Culture Techniques. This combination of tests should include culture (with toxin testing of the isolate), demonstration of toxin direct from the faeces and the detection of . antigen. This chapter outlines the methods used to establish the laboratory diagnosis of CDI and also includes the investigation of environment
作者: 友好關(guān)系    時間: 2025-3-23 09:31
Multilocus Sequence Typing for ,eveals a clonal population structure. Mutational evolution of . is characterized, with point mutation generating new alleles at a frequency eightfold higher than recombinational exchange. Phylogenetic analysis shows that human and animal isolates do not cluster in distinct lineages, and that no hype
作者: Glossy    時間: 2025-3-23 11:06

作者: 金哥占卜者    時間: 2025-3-23 16:04
Comparative Genome Analysis of , Using DNA Microarrays Nevertheless, whole-scale genome sequencing remains an expensive endeavour and such comparisons are limited to only a handful of strains. DNA microarrays represent an alternative technology for whole genome comparisons enabling a “birds eye view” of all the genes absent or present in a given genome
作者: Dorsal-Kyphosis    時間: 2025-3-23 18:06
ClosTron-Targeted Mutagenesis chromosome via a single crossover event. As an alternative, recombination-independent strategies have been devised that are reliant upon a re-targeted group II intron. One element in particular, the ClosTron, provides the facility for the positive selection of insertional mutants. The generation of
作者: nocturnal    時間: 2025-3-23 22:42
Clostridium difficile978-1-60327-365-7Series ISSN 1064-3745 Series E-ISSN 1940-6029
作者: Fester    時間: 2025-3-24 03:37
Peter Mullany,Adam P. RobertsServes as a comprehensive guide to most of the current techniques for manipulating and analyzing C. difficile.Written by scientists who have pioneered modern C. difficile research.Brings together in o
作者: 有角    時間: 2025-3-24 07:23

作者: 傳染    時間: 2025-3-24 12:53
E. Straube,G. Polzer,G. Buchkremerged from an obscure, and often under-researched, bacterium to one of major clinical importance, at least in industrialized nations. Coupled with the increased interest in this organism researchers now have a greater understanding of its genetic content and molecular epidemiology; a direct consequenc
作者: 預(yù)兆好    時間: 2025-3-24 18:13

作者: 較早    時間: 2025-3-24 21:15
Ch. Beal Anderson,L. Ereshefskylitis or toxic megacolon. The disease arises as a major complication of antibiotic therapy and is most commonly acquired in hospital. The laboratory investigation of faecal samples is supportive of a clinical suspicion that a patient has the disease. Currently the mainstay of diagnosis is the demons
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https://doi.org/10.1007/978-3-642-83960-3cell wall and its components also have to mediate interactions with the host cells and their products. In this chapter we discuss the various methods used for dissecting the cell surface and the biochemical and immunological procedures that are commonly used to analyse the properties of the proteins
作者: 軟弱    時間: 2025-3-25 22:43
https://doi.org/10.1007/978-3-642-83960-3itis, particularly in hospitalized, elderly patients. Studies to date suggest that . toxins A and B play a major role in the observed colonic inflammation and associated disease pathogenesis; however, the role of other potential bacterial factors at present remains unknown. Early effects of . on hos
作者: HUMID    時間: 2025-3-26 00:50

作者: Fermentation    時間: 2025-3-26 07:22
Was verursacht Schizophrenien?,ological processes they undertake. This has led to increasing efforts directed at deriving greater information on their basic biology, most notably through genome sequence. Accordingly, annotated sequences of all of the most important species are now available. However, full exploitation of the data
作者: medieval    時間: 2025-3-26 12:26
,Das heutige Wissen um ?Schizophrenien“,ulent” strains associated with increased rates of mortality and severity of disease in humans has highlighted the need to study this organism at the molecular level. These studies will increase our knowledge of the mechanisms by which . causes disease and facilitate the rational design of new and im
作者: 匍匐    時間: 2025-3-26 15:25
https://doi.org/10.1007/978-3-662-09811-0at is to use the conjugative transposon Tn.. Tn. enters the genome of most strains of . with no obvious target site preference. In order to use the genome strain . 630 for transposon mutagenesis a erythromycin-sensitive derivative . 630Δ. was constructed and the Tn. derivative, Tn.ΔE, was shown to e
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978-1-4939-5701-9Springer Science+Business Media, LLC 2010
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,Das heutige Wissen um ?Schizophrenien“, the last decade) that methods have been developed to introduce plasmid DNA into . and most importantly to enable the generation of isogenic mutants in this emerging human pathogen. These methods are essential prerequisites for the effective study of gene function in this important bacterium.
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https://doi.org/10.1007/978-3-642-83960-3used for dissecting the cell surface and the biochemical and immunological procedures that are commonly used to analyse the properties of the proteins within the cell wall. A major consideration is the S-layer which in . shows considerable variation in sequence and between strains, a property which is also reflected in its antigenic properties.
作者: Grievance    時間: 2025-3-29 00:12
https://doi.org/10.1007/978-3-642-83960-3tion and associated disease pathogenesis; however, the role of other potential bacterial factors at present remains unknown. Early effects of . on host intestinal epithelia include modest induction of innate immune responses with progressive loss of intestinal epithelial cell barrier function and cell death.
作者: SHRIK    時間: 2025-3-29 06:49
https://doi.org/10.1007/978-3-662-09811-0nome strain . 630 for transposon mutagenesis a erythromycin-sensitive derivative . 630Δ. was constructed and the Tn. derivative, Tn.ΔE, was shown to enter the genome at multiple sites enabling the construction of a Tn. insertion library.
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作者: 下級    時間: 2025-3-29 15:12

作者: CUR    時間: 2025-3-29 15:40
Human Intestinal Epithelial Response(s) to ,tion and associated disease pathogenesis; however, the role of other potential bacterial factors at present remains unknown. Early effects of . on host intestinal epithelia include modest induction of innate immune responses with progressive loss of intestinal epithelial cell barrier function and cell death.




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