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標(biāo)題: Titlebook: Clinical Cellular Immunology; Molecular and Therap Albert A. Luderer,Howard H. Weetall Book 1982 The HUMANA Press Inc. 1982 Antigen.Immune [打印本頁(yè)]

作者: Wilson    時(shí)間: 2025-3-21 16:58
書目名稱Clinical Cellular Immunology影響因子(影響力)




書目名稱Clinical Cellular Immunology影響因子(影響力)學(xué)科排名




書目名稱Clinical Cellular Immunology網(wǎng)絡(luò)公開度




書目名稱Clinical Cellular Immunology網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Clinical Cellular Immunology被引頻次




書目名稱Clinical Cellular Immunology被引頻次學(xué)科排名




書目名稱Clinical Cellular Immunology年度引用




書目名稱Clinical Cellular Immunology年度引用學(xué)科排名




書目名稱Clinical Cellular Immunology讀者反饋




書目名稱Clinical Cellular Immunology讀者反饋學(xué)科排名





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https://doi.org/10.1007/978-1-4842-1506-7wing infection by tubercle bacilli. Dienes many years later (1936) was the first to consider delayed hypersensitivity as an independent branch of the immunological response rather than a unique response to infectious microorganisms. He was successful in producing delayed hypersensitivity by injectin
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https://doi.org/10.1057/9780230508996 that dialyzable substances present in leukocytes have the ability to influence the expression of delayed hypersensitivity (Lawrence, 1955; Lawrence, 1963). In particular, Lawrence noted that a component of such dialyzates could apparently “transfer” specific immunologic sensitivity from a highly se
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The End of the War and Reconversion,One of the major problems in immunology has been to raise homogeneous antibodies of high concentration that are specific for a given antigen.
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Hybridization of Lymphocytes,One of the major problems in immunology has been to raise homogeneous antibodies of high concentration that are specific for a given antigen.
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Coating Thickness Calculation and Adhesionhe basic immunological phenomena that have led to the advances illustrated in the more clinically oriented chapters and to the concepts of clinical cellular immunology to which this book is addressed.
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https://doi.org/10.1007/978-1-4842-1506-7immunological response rather than a unique response to infectious microorganisms. He was successful in producing delayed hypersensitivity by injecting nonliving protein antigens into tuberculous foci.
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,Immunosuppressive Agents—Their Action on Inductive and Regulatory Pathways,
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Finite Element Methods for Rolling Contact,therapeutic agents, the introduction of new agents in recent years, and the development of combinations of drugs have made possible more prudent utilization of these agents for maximal therapeutic benefits with relatively minimal undesirable effects. Nevertheless, chemotherapy and extensive radiothe
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Immunosuppressive Agents,therapeutic agents, the introduction of new agents in recent years, and the development of combinations of drugs have made possible more prudent utilization of these agents for maximal therapeutic benefits with relatively minimal undesirable effects. Nevertheless, chemotherapy and extensive radiothe
作者: 褻瀆    時(shí)間: 2025-3-26 12:49
,The Immunobiology of Human Non-Hodgkin’s Lymphomas,s spectrum may permit a dissection of those elements that define pathophysiology and determine the clinical presentation of disease. These must include the cell type affected (T, B, null) and the roles of lymphocyte homing patterns and immunoregulatory influences.
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The T Cell Antigen Receptor,ssociated receptors found on the cell surface of lymphocytes. Certainly the interaction of antigen with both regulatory and effector thymus-derived lymphocytes (T cells) is a critical event in this process. According to clonal selection theories (Burnett, 1969), antigen initially interacts with a re
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Immunosuppressive Agents,he disease, and each has its benefits and risks. Although radiotherapy and chemotherapy have the advantage of reaching tumors and cells that are inaccessible to surgery (or, because of their location or wide dissemination, that cannot be surgically removed), these forms of treatment may exert damagi
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,The Immunobiology of Human Non-Hodgkin’s Lymphomas,ly expanding knowledge of immunocytes has been applied to this spectrum of diseases and has resulted in more scientifically accurate classifications. Of perhaps greater import, however, is the nascent realization that these diseases represent malignant counterparts of normal lymphocyte development,
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Cell-Mediated Immunity in Autoimmune Disease,wing infection by tubercle bacilli. Dienes many years later (1936) was the first to consider delayed hypersensitivity as an independent branch of the immunological response rather than a unique response to infectious microorganisms. He was successful in producing delayed hypersensitivity by injectin
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Cell-Mediated Immunity in Tumor Rejection,nt. Although much effort was initially expended concerning humoral immune mechanism in malignancy, many studies over the last decade or so have focused attention mainly on cell-mediated immune (CMI) interactions of the host with tumors. It is, however, quite apparent that such CMI reactions are high
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Immunologic Tests for Diagnosis and Monitoring of Defects in Cell-Mediated Immunity,agnosis and more precise definition of clinical immunodeficiencies. The definition of particular defects by immunological and biochemical means has in addition led to the development of rational therapeutic approaches, such as replacement therapy and immunomodulation (Fudenberg and Wybran, 1980). Ad
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