標(biāo)題: Titlebook: Chronic Pancreatitis; Research and Clinica H. G. Beger,M. Büchler,P. Malfertheiner Conference proceedings 1990 Springer-Verlag Berlin Heide [打印本頁] 作者: aggression 時(shí)間: 2025-3-21 18:45
書目名稱Chronic Pancreatitis影響因子(影響力)
書目名稱Chronic Pancreatitis影響因子(影響力)學(xué)科排名
書目名稱Chronic Pancreatitis網(wǎng)絡(luò)公開度
書目名稱Chronic Pancreatitis網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Chronic Pancreatitis被引頻次
書目名稱Chronic Pancreatitis被引頻次學(xué)科排名
書目名稱Chronic Pancreatitis年度引用
書目名稱Chronic Pancreatitis年度引用學(xué)科排名
書目名稱Chronic Pancreatitis讀者反饋
書目名稱Chronic Pancreatitis讀者反饋學(xué)科排名
作者: Fillet,Filet 時(shí)間: 2025-3-21 22:40
Incidence and Prevalence of Chronic Pancreatitis] and 5% [2]. The clinical picture was only sporadically mentioned in the medical literature up to 1946, when Comfort et al. [3] presented the first detailed clinical material of chronic pancreatitis, comprising 29 cases. Since that time, studies on etiology, clinical picture, medical and surgical t作者: 主動(dòng) 時(shí)間: 2025-3-22 00:40
Role of Nutrition in Chronic Pancreatitisical stimuli, the most prominent of which are nutritional in origin. Normal pancreatic function and structure are altered by a number of nutritional factors which include changes in intake of major dietary components (proteins, carbohydrates, and fats) as well as vitamins and other micronutrients an作者: Dorsal-Kyphosis 時(shí)間: 2025-3-22 05:08 作者: Sad570 時(shí)間: 2025-3-22 09:08 作者: Hyperplasia 時(shí)間: 2025-3-22 14:44 作者: Hyperplasia 時(shí)間: 2025-3-22 20:58
Focal Necrosis: Primary Event in the Pathogenesis of Chronic Pancreatitis?20]. Yet its significance for the pathogenesis of chronic pancreatitis has been neglected for many years, owing to the widely accepted view that acute and chronic pancreatitis differ in etiology and pathogenesis [18]. Here we will briefly review the pathology of focal necrosis and pseudocysts in chr作者: 細(xì)節(jié) 時(shí)間: 2025-3-22 21:46 作者: Intersect 時(shí)間: 2025-3-23 01:45 作者: 喃喃訴苦 時(shí)間: 2025-3-23 07:55 作者: Adherent 時(shí)間: 2025-3-23 11:03
Chronic Pancreatitis: Search for Animal ModelsTable 1), chronic pancreatitis has not been consistently produced in experimental animals. Extensive overviews on the experimental models and concepts in acute pancreatitis [1–25, 26] have been published; international workshops on acute experimental pancreatitis have been held [24]. In addition, ex作者: Ledger 時(shí)間: 2025-3-23 14:15
Lysosomal Enzyme Activation of Digestive Enzymes During Chronic Pancreatitis?nea-pig cell [1]. Digestive enzymes which constitute most of the pancreatic proteins and lysosomal hydrolases are both synthesized by ribosomes attached to the rough endoplasmic reticulum. The nascent polypetide chains elongate within the cisternae of the reticulum, migrate through this compartment,作者: vascular 時(shí)間: 2025-3-23 20:23
Juristen um Luther in Wittenbergme of the loss comes about by a process which has been termed apoptosis because the apical part of the acinar cell is sloughed off. Some of the cells simply disintegrate and become part of the amorphous material which may be drained away by patent ducts or retained in the connective tissue space.作者: Vasoconstrictor 時(shí)間: 2025-3-23 23:57 作者: 外向者 時(shí)間: 2025-3-24 05:26 作者: Torrid 時(shí)間: 2025-3-24 08:31
Lysosomal Enzyme Activation of Digestive Enzymes During Chronic Pancreatitis?granules and when necessary are discharged into the acinar lumen by fusion-fission of the zymogen granule limiting membrane and the luminal plasmalemma whereas lysosomal hydrolases are usually targeted for inclusion into lysosomes.作者: Vulvodynia 時(shí)間: 2025-3-24 13:32 作者: crease 時(shí)間: 2025-3-24 16:25
Birgit Ulrike Münch,Andreas Tacke and chronic pancreatitis differ in etiology and pathogenesis [18]. Here we will briefly review the pathology of focal necrosis and pseudocysts in chronic pancreatitis and discuss their possible role in the pathogenesis of the disease.作者: Cougar 時(shí)間: 2025-3-24 22:04
https://doi.org/10.1007/978-3-030-81795-4 in acute pancreatitis [1–25, 26] have been published; international workshops on acute experimental pancreatitis have been held [24]. In addition, experimental models of exocrine pancreatic tumor have been developed [8]. As yet, however, no reproducible animal models of chronic pancreatitis have been reported.作者: Climate 時(shí)間: 2025-3-25 02:23 作者: 人類學(xué)家 時(shí)間: 2025-3-25 05:22
Focal Necrosis: Primary Event in the Pathogenesis of Chronic Pancreatitis? and chronic pancreatitis differ in etiology and pathogenesis [18]. Here we will briefly review the pathology of focal necrosis and pseudocysts in chronic pancreatitis and discuss their possible role in the pathogenesis of the disease.作者: Additive 時(shí)間: 2025-3-25 11:12 作者: 構(gòu)成 時(shí)間: 2025-3-25 12:26 作者: 博愛家 時(shí)間: 2025-3-25 19:25
Birgit Ulrike Münch,Andreas Tackeundergo a tonsillectomy or spontaneous abortion [5], just to name a few. Chronic pancreatitis also has been mentioned among these risk factors. The purpose of this report is to focus on whether there is any link between chronic pancreatitis and pancreatic cancer based on review of the epidemiologic data and clinical and experimental experiences.作者: 鋪?zhàn)?nbsp; 時(shí)間: 2025-3-25 22:38 作者: BOON 時(shí)間: 2025-3-26 00:39 作者: sinoatrial-node 時(shí)間: 2025-3-26 04:29
Chronic Pancreatitis with Inflammatory Mass in the Head of the Pancreas: A Special Entity?t lesion either in the right (head) or in the left part (body and tail) of the gland. For this reason, surgical techniques in chronic pancreatitis have been developed and applied either to resect the pancreatic head and preserve the body and tail or vice versa [4, 5].作者: Rankle 時(shí)間: 2025-3-26 10:07
Peptidergic Innervation in Chronic Pancreatitise tachykinin substance P (SP) may be involved in chronic inflammatory and painful disease of the gastrointestinal system is evidenced by a selective increase in the density of tachykinin receptors in the bowels of patients suffering from Crohn’s disease and ulcerative colitis [62, 63, 64].作者: Brain-Waves 時(shí)間: 2025-3-26 15:38 作者: incisive 時(shí)間: 2025-3-26 19:25
Juristen um Luther in Wittenberge tachykinin substance P (SP) may be involved in chronic inflammatory and painful disease of the gastrointestinal system is evidenced by a selective increase in the density of tachykinin receptors in the bowels of patients suffering from Crohn’s disease and ulcerative colitis [62, 63, 64].作者: Feckless 時(shí)間: 2025-3-27 00:02
Reform, Transformation and Growthbe done?” The second is to provide a lingua franca among those caring for the patients. No classification system is perfect, and any one simply reflects the state of the art at the time that it is promulgated.作者: 斜 時(shí)間: 2025-3-27 01:15 作者: maroon 時(shí)間: 2025-3-27 08:23
https://doi.org/10.1007/978-981-99-5712-5ical stimuli, the most prominent of which are nutritional in origin. Normal pancreatic function and structure are altered by a number of nutritional factors which include changes in intake of major dietary components (proteins, carbohydrates, and fats) as well as vitamins and other micronutrients an作者: Commodious 時(shí)間: 2025-3-27 13:07 作者: 單獨(dú) 時(shí)間: 2025-3-27 15:31
https://doi.org/10.1007/978-3-8349-8109-7ronic pancreatitis; thus, nutritional, metabolic, and mechanical disturbances, as well as a hereditary disposition, have been implicated:.However, not all of these etiologies are proven, and some are unlikely or controversial.作者: Fatten 時(shí)間: 2025-3-27 18:23
https://doi.org/10.1007/978-3-8349-8109-7of patients (5%–10%), obviously in an early phase of the development of the disease, who suffer from focal lesions of chronic pancreatitis. Mainly data from surgical literature have shown that patients with chronic pancreatitis undergoing surgery for pain or complications often exhibit a preponderan作者: FLAGR 時(shí)間: 2025-3-28 00:29 作者: Proclaim 時(shí)間: 2025-3-28 03:58
Juristen um Luther in Wittenbergparenchyma. Acinar cells in affected areas contain fewer or no zymogen granules; the cells decrease in height. The result of this reversion is tubular complexes, which are derived from a combination of acini and small ducts, but which have the appearance of a collection of ducts [5]. These areas are作者: outer-ear 時(shí)間: 2025-3-28 07:24 作者: FADE 時(shí)間: 2025-3-28 12:10 作者: Cardioplegia 時(shí)間: 2025-3-28 16:19
https://doi.org/10.1007/978-3-030-81795-4Table 1), chronic pancreatitis has not been consistently produced in experimental animals. Extensive overviews on the experimental models and concepts in acute pancreatitis [1–25, 26] have been published; international workshops on acute experimental pancreatitis have been held [24]. In addition, ex作者: corpuscle 時(shí)間: 2025-3-28 20:33 作者: Suggestions 時(shí)間: 2025-3-29 02:44 作者: gusher 時(shí)間: 2025-3-29 06:00
Role of Nutrition in Chronic Pancreatitisical stimuli, the most prominent of which are nutritional in origin. Normal pancreatic function and structure are altered by a number of nutritional factors which include changes in intake of major dietary components (proteins, carbohydrates, and fats) as well as vitamins and other micronutrients and trace elements [1].作者: ligature 時(shí)間: 2025-3-29 07:26 作者: Antagonism 時(shí)間: 2025-3-29 11:58 作者: 清楚說話 時(shí)間: 2025-3-29 18:22 作者: Heterodoxy 時(shí)間: 2025-3-29 21:15 作者: Lacunar-Stroke 時(shí)間: 2025-3-30 03:54
https://doi.org/10.1007/978-3-8349-8109-7ronic pancreatitis; thus, nutritional, metabolic, and mechanical disturbances, as well as a hereditary disposition, have been implicated:.However, not all of these etiologies are proven, and some are unlikely or controversial.作者: antidepressant 時(shí)間: 2025-3-30 06:27
https://doi.org/10.1007/978-3-642-75319-0Chronic pancreatitis; Exocrine pancreatic insufficiency; Pancreatic duct stones; Pancreatic pseudo cyst作者: N斯巴達(dá)人 時(shí)間: 2025-3-30 11:40 作者: 移植 時(shí)間: 2025-3-30 13:08 作者: extinguish 時(shí)間: 2025-3-30 16:49 作者: genuine 時(shí)間: 2025-3-30 22:07
Early Modern Literature in HistoryEthanol is rapidly absorbed and metabolized in the liver by a complex mixture of isoenzymes (alcohol dehydrogenase) that oxidize it to acetaldehyde. Acetaldehyde may be formed in the pancreas or released from albumin complexes with subsequent preferential binding to target cells including pancreatic acinar and ductal cells.作者: incisive 時(shí)間: 2025-3-31 01:29
Natural History of Chronic (Progressive) Pancreatitis: A Life ExperienceThe controversies on diagnosis and therapy of chronic pancreatitis (CP) are as marked today as 30 years ago despite many international meetings of classification [1], a marked improvement of diagnostic technics, and, the large and increasing amount of literature.作者: 變量 時(shí)間: 2025-3-31 06:39 作者: 溫和女孩 時(shí)間: 2025-3-31 12:02 作者: Musket 時(shí)間: 2025-3-31 14:06 作者: 死貓他燒焦 時(shí)間: 2025-3-31 18:15
Exocrine and Endocrine Morphologic Changes in Chronic Pancreatitis
