標題: Titlebook: Chemical Carcinogenesis and Mutagenesis II; Colin S. Cooper,Philip L. Grover Book 1990 Springer-Verlag Berlin Heidelberg 1990 Carcinom.DNA [打印本頁] 作者: Odious 時間: 2025-3-21 17:35
書目名稱Chemical Carcinogenesis and Mutagenesis II影響因子(影響力)
書目名稱Chemical Carcinogenesis and Mutagenesis II影響因子(影響力)學科排名
書目名稱Chemical Carcinogenesis and Mutagenesis II網(wǎng)絡(luò)公開度
書目名稱Chemical Carcinogenesis and Mutagenesis II網(wǎng)絡(luò)公開度學科排名
書目名稱Chemical Carcinogenesis and Mutagenesis II被引頻次
書目名稱Chemical Carcinogenesis and Mutagenesis II被引頻次學科排名
書目名稱Chemical Carcinogenesis and Mutagenesis II年度引用
書目名稱Chemical Carcinogenesis and Mutagenesis II年度引用學科排名
書目名稱Chemical Carcinogenesis and Mutagenesis II讀者反饋
書目名稱Chemical Carcinogenesis and Mutagenesis II讀者反饋學科排名
作者: BILIO 時間: 2025-3-21 23:02 作者: 剝削 時間: 2025-3-22 02:33 作者: 諷刺 時間: 2025-3-22 06:17 作者: 一起 時間: 2025-3-22 11:59
Basic Windows Forms Applicationsh DNA. This DNA damage then leads to the alteration of the activity of certain critical genes by inducing either mutations or an increased frequency of gene rearrangement which, in turn, results in abnormal regulation of gene expression. Although multiple mutations may be required to produce neoplas作者: 無瑕疵 時間: 2025-3-22 16:47
Advanced Windows Forms Applicationst at an early stage in the step-wise induction of malignant cancers, by favouring the cloncal expansion of initiated cells to benign lesions such as papillomas, nodules and polyps. Although mouse skin has been most extensively studied, a number of animal models have been developed in order to study 作者: 無瑕疵 時間: 2025-3-22 17:42
Pro Visual C++/CLI and the .NET 3.5 Platformreviewed in S. 1980). Epidemiological studies have suggested that specific, pharmcologically active agents present in the diet may reduce the relative risk of exposure to carcinogens. For example, an inverse correlation between dietary selenium levels and human cancer mortality has been noted (S. an作者: Leisureliness 時間: 2025-3-22 22:41
.NET Configuration File Programmingthe aetiology of the whole range of neoplastic diseases. It is equally clear that factors intrinsic to the patient or animal host also influence both the occurrence of cancer and the biological behaviour of the neoplasm. The principal intrinsic variables are age, hormone balance, immune response and作者: Creditee 時間: 2025-3-23 05:28 作者: Functional 時間: 2025-3-23 08:50 作者: averse 時間: 2025-3-23 11:37
System and Logical Datacenter Designerses which carry oncogenes. Initially these oncogenes were isolated from avian retroviruses. Cellular homologues of these genes, termed proto-oncogenes, have been identified in eukaryotes. The demonstration that the introduction of activated oncogenes can confer a tumorigenic phenotype in certain cell作者: mechanism 時間: 2025-3-23 16:06
Pro Visual Studio 2005 Team Systemnd tumour induction. The recent exciting advances in this field are rooted in earlier observations that identified genetic material as the probable target during carcinogenesis. Boverii, who in 1914 noted frequent karyotypic abnormalities in tumour cells, is usually credited as the first person to r作者: JOT 時間: 2025-3-23 18:04
https://doi.org/10.1007/978-1-4302-0171-7ve been extensively characterised, and the corresponding cDNAs have been cloned (Table 1). Several additional growth factors are known from their biological activities but have not yet been structurally characterised. The in vivo functions of growth factors are assumed to be the stimulation of fetal作者: vasculitis 時間: 2025-3-24 00:08 作者: Ligament 時間: 2025-3-24 05:12
Pro Visual Studio 2005 Team System morphology and by their ability to proliferate under conditions which do not support, or severely limit, the proliferation of their normal counterparts. The altered morphology is recognized by the pathologist in tumor biopsies and is used not only for diagnosis of malignancy but also for tumor stag作者: 獨白 時間: 2025-3-24 07:09
Chemical Carcinogenesis and Mutagenesis II978-3-642-74778-6Series ISSN 0171-2004 Series E-ISSN 1865-0325 作者: Musket 時間: 2025-3-24 10:40
https://doi.org/10.1007/978-3-642-74778-6Carcinom; DNA; Mutagene; Onkogene Viren; cancer; cancer research; carcinogenesis; cellular differentiation; 作者: 鋼筆尖 時間: 2025-3-24 16:08 作者: 無思維能力 時間: 2025-3-24 23:03
Handbook of Experimental Pharmacologyhttp://image.papertrans.cn/c/image/224298.jpg作者: ADORE 時間: 2025-3-25 00:47 作者: 單調(diào)女 時間: 2025-3-25 04:35
Pro Visual C++/CLI and the .NET 3.5 Platformver, spontaneous tumours or tumours induced by treatment with furan and furfuranal were analysed (R. et al. 1987). In the spontaneous tumours the activated H-. mutations were always at codon 61. However, 40%–60% of the . oncogenes detected in the furan- or furfural-induced tumours were at sites othe作者: Occlusion 時間: 2025-3-25 10:42 作者: entail 時間: 2025-3-25 15:24 作者: Dendritic-Cells 時間: 2025-3-25 17:30
Advanced Windows Forms Applicationser (see Table 1), and certainly promotion in skin and liver is achieved by different groups of compounds. Consequently, we believe it would also be useful to consider whether the same, different or overlapping mechanisms are involved.作者: 2否定 時間: 2025-3-25 20:18 作者: Foment 時間: 2025-3-26 03:47
Team Foundation Version Controlcinogenesis (Fig. 1). Viral infections are widespread in humans, and viruses are factors in several types of human cancer, e.g., Burkitt’s lymphoma, nasopharyngeal cancer, liver cancer, T-cell leukemia, skin cancer, and cervical cancer (for review, see P. 1983). The purpose of our review is to summa作者: 不安 時間: 2025-3-26 06:34 作者: 駁船 時間: 2025-3-26 09:58 作者: 薄荷醇 時間: 2025-3-26 15:10 作者: 受人支配 時間: 2025-3-26 20:03
Pro Visual Studio 2005 Team System diagnosis or identification of the altered cells may be aided by the excess proliferation of these cells in relation to their normal counterparts. The altered morphology is an indication of changes in the synthesis of products which allow us to distinguish cell types. Typically, these are not produ作者: 得罪人 時間: 2025-3-27 00:34 作者: AFFIX 時間: 2025-3-27 03:58
Cancer-Prone Human Disorders with Defects in DNA Repairl as with a variety of other clinical abnormalities. The prototype disease in this category is xeroderma pigmentosum (XP) in which a clearly identifiable defect in the repair of UV damage in DNA is associated with a high frequency of UV-induced mutations in cultured cells and with a greatly increase作者: Pcos971 時間: 2025-3-27 07:59
DNA Repair and Carcinogenesis by Alkylating Agents a tumor. Numerous laboratory investigations, some of which are discussed in this chapter, are consistent with this hypothesis. There is also abundant evidence from epidemiological studies of the increased incidence of cancer in individuals suffering from heritable diseases involving defects in DNA 作者: AROMA 時間: 2025-3-27 12:00 作者: 微塵 時間: 2025-3-27 17:15
Inhibition of Chemical Carcinogenesisrs of chemical carcinogenesis in experimental animals. Interest in chemical inhibitors of carcinogenesis is twofold: (i) they provide valuable tools for studying the biochemical and molecular mechanism(s) of carcinogenesis; and (ii) understanding their chemical nature, mechanism(s), and specificity 作者: 包庇 時間: 2025-3-27 19:28 作者: ovation 時間: 2025-3-28 00:30 作者: 刺耳的聲音 時間: 2025-3-28 04:49 作者: flavonoids 時間: 2025-3-28 09:47
Signal Transduction in Proliferating Normal and Transformed Cellstion see W. et al. (1987). Following the addition of serum or defined growth factors, the quiescent cells are stimulated to enter the cell cycle. Using this experimental system, the early biochemical events occurring in the cell in response to growth stimulation can be investigated. Such studies hav作者: 正面 時間: 2025-3-28 12:34 作者: 難取悅 時間: 2025-3-28 17:23 作者: inconceivable 時間: 2025-3-28 22:05
https://doi.org/10.1007/978-1-4302-1054-2y in humans involve deficiencies in processing DNA lesions (see Chap. 4). Moreover, it is becoming quite clear that unrepaired lesions which lead to mutations play a role in the activation of proto-oncogenes (see Chap. 12).作者: 噴油井 時間: 2025-3-29 02:40 作者: 與野獸博斗者 時間: 2025-3-29 03:34 作者: 詢問 時間: 2025-3-29 10:51
Mechanisms of Repair in Mammalian Cellsy in humans involve deficiencies in processing DNA lesions (see Chap. 4). Moreover, it is becoming quite clear that unrepaired lesions which lead to mutations play a role in the activation of proto-oncogenes (see Chap. 12).作者: Engulf 時間: 2025-3-29 12:33
Recessive Oncogenes and Anti-Oncogenes are involved in tumour development. Indeed, there is now a growing body of evidence to suggest that other classes of genes known as anti-oncogenes or suppressor genes may also function in the progressive series of events which leads to tumorigenesis.作者: 北極熊 時間: 2025-3-29 19:35 作者: 輕快走過 時間: 2025-3-29 20:25 作者: STING 時間: 2025-3-30 03:53 作者: 易彎曲 時間: 2025-3-30 08:06 作者: irritation 時間: 2025-3-30 08:30 作者: intuition 時間: 2025-3-30 13:16
Use of Mutations in Bacteria as Indicators of Carcinogenic Potentialms from Boveri’s theory of the mutational origin of cancer (see W. 1974), which in turn was refined by B. (1928) and gained increasing acceptance with successive comparative studies of mutagens and carcinogens in the early 1950s.作者: Etching 時間: 2025-3-30 17:26 作者: 外面 時間: 2025-3-31 00:47
Use of Mutations in Bacteria as Indicators of Carcinogenic Potentialen and sulphur mustards induced gene mutations (and chromosome rearrangements) in the germ cells of . (A. and R. 1946). The rationale for such use stems from Boveri’s theory of the mutational origin of cancer (see W. 1974), which in turn was refined by B. (1928) and gained increasing acceptance with
