作者: 繁榮中國 時(shí)間: 2025-3-21 22:27
Clinical Featureslude rapidly or slowly developing processes, compressive or disruptive/distractive. Minor indirect trauma can also cause BCP [1, 2]. Stroke, either ischemic or hemorrhagic, is the commonest cause of BCP. The entire spectrum of causes of stroke can lead to CP. Several instances of rare etiologies are作者: 得意牛 時(shí)間: 2025-3-22 04:28
Somatosensory Findings to complete loss of all somatic sensibility in the painful region or a very painful hyperesthesia. However, pain distribution is usually well correlated with sensory abnormalities (Tables 4.1 and 4.2). Exceptional cases of somatotopic mismatch are on record: Birznieks et al. [30] performed detailed作者: 遭遇 時(shí)間: 2025-3-22 08:10
Central Pruritusequence of PNP). Pruritus can blend with pain or dysesthesias or present singly (note that pain-insensitive patients are also itch-insensitive: [38]). Most likely, patients may complain of itching, but the treating physician may dismiss it until scratching makes the problem visible. Pruritus tends t作者: beta-cells 時(shí)間: 2025-3-22 10:48 作者: somnambulism 時(shí)間: 2025-3-22 13:25 作者: somnambulism 時(shí)間: 2025-3-22 17:56 作者: 行為 時(shí)間: 2025-3-23 01:09
GABA Agonists–3, δ, ε, θ, π, and ρ1–3). GABA-A receptor assembly can be derived from a permutation and combination of two, three, four, or even five different subunits. Distribution of the major subunits in various regions of the brain varies. Importantly, GABA neurotransmission can be recoded to become excitato作者: 一再困擾 時(shí)間: 2025-3-23 03:04 作者: 大喘氣 時(shí)間: 2025-3-23 08:12 作者: Negotiate 時(shí)間: 2025-3-23 11:48
Brainstem Modulators forebrain (e.g., Meynert’s nucleus) nuclei. Fibers leaving these nuclei fan out both upward into the brain and downward to the cord. These regulate the function and activation state of the brain, both diffusely and focally. However, other mechanisms of action are possible and are differentiated acc作者: 含糊 時(shí)間: 2025-3-23 14:29 作者: falsehood 時(shí)間: 2025-3-23 18:12 作者: 知識(shí) 時(shí)間: 2025-3-23 23:06
https://doi.org/10.1007/978-3-319-56765-5Neuropathic Pain; Chronic Pain; Neuromodulation; Neuroimaging; Central Pain作者: MOCK 時(shí)間: 2025-3-24 05:07 作者: Injunction 時(shí)間: 2025-3-24 10:08
https://doi.org/10.1007/978-3-642-81763-2ling opinion of the day, introduced the concept of .. The patient was .. (Mrs. R.), aged 48, who developed . (violent pains and clear-cut hyperesthesia in the paretic limbs: right arm and leg), . (due to the terrible pains, suicide 1888). She was . (opioid) unresponsive. At autopsy, a thalamic infar作者: propose 時(shí)間: 2025-3-24 13:41
,Neue Verfahren zur Schmierstoffprüfung,lude rapidly or slowly developing processes, compressive or disruptive/distractive. Minor indirect trauma can also cause BCP [1, 2]. Stroke, either ischemic or hemorrhagic, is the commonest cause of BCP. The entire spectrum of causes of stroke can lead to CP. Several instances of rare etiologies are作者: 使害羞 時(shí)間: 2025-3-24 14:54
Notes on the Life History of Coho Salmon to complete loss of all somatic sensibility in the painful region or a very painful hyperesthesia. However, pain distribution is usually well correlated with sensory abnormalities (Tables 4.1 and 4.2). Exceptional cases of somatotopic mismatch are on record: Birznieks et al. [30] performed detailed作者: nocturia 時(shí)間: 2025-3-24 22:26
Arthur D. Hasler,Allan T. Scholzequence of PNP). Pruritus can blend with pain or dysesthesias or present singly (note that pain-insensitive patients are also itch-insensitive: [38]). Most likely, patients may complain of itching, but the treating physician may dismiss it until scratching makes the problem visible. Pruritus tends t作者: 省略 時(shí)間: 2025-3-25 00:25
https://doi.org/10.1007/978-3-642-82141-7Figs. 8.1 and 8.2), following the establishment of an intracortical S1 . (. (e.g., layer 4? (layer 2/3?) layer 5? layer 6? layer 4+corticothalamic outflow). The attendant sensory information decorrelation translates into different sensory percepts (pain, dysesthesias, paresthesias, pruritus).作者: Fresco 時(shí)間: 2025-3-25 07:19 作者: Detoxification 時(shí)間: 2025-3-25 10:06 作者: A精確的 時(shí)間: 2025-3-25 13:10 作者: 抱負(fù) 時(shí)間: 2025-3-25 17:21 作者: 燈泡 時(shí)間: 2025-3-25 20:25 作者: 爭吵加 時(shí)間: 2025-3-26 01:13
Properties of Message Addressing Schemes forebrain (e.g., Meynert’s nucleus) nuclei. Fibers leaving these nuclei fan out both upward into the brain and downward to the cord. These regulate the function and activation state of the brain, both diffusely and focally. However, other mechanisms of action are possible and are differentiated acc作者: 神圣將軍 時(shí)間: 2025-3-26 06:56 作者: Chemotherapy 時(shí)間: 2025-3-26 09:43 作者: 使厭惡 時(shí)間: 2025-3-26 13:33 作者: 放牧 時(shí)間: 2025-3-26 18:31 作者: 圓錐體 時(shí)間: 2025-3-26 23:34 作者: 成份 時(shí)間: 2025-3-27 04:58
EpidemiologyWhen one considers neuropathic pain globally, 6.9%–10% of the entire population is affected [1].作者: 極大痛苦 時(shí)間: 2025-3-27 05:55 作者: incisive 時(shí)間: 2025-3-27 12:44
DiagnosisCP is pain/dysesthesia/pruritus due to a CNS lesion along the spinothalamoparietal (STP) path. Thus, an appropriate lesion must be demonstrated in such a location. Bilateral pain and dysesthesia referred to the limbs, although usually pointing to a spinal cord lesion, may rarely be observed after unilateral brain lesions.作者: SKIFF 時(shí)間: 2025-3-27 15:26 作者: 停止償付 時(shí)間: 2025-3-27 20:24
Channel Blockersgeminal neuralgia with cocaine injections. Deisenhammer et al. [1] published the first Medline-indexed paper detailing the use of a Na+ channel blocker (carbamazepine) for CP (Tables 13.1, 13.2, 13.3, 13.4, 13.5, 13.6, 13.7, 13.8, 13.9, 13.10, 13.11, 13.12, 13.13, 13.14, 13.15, and 13.16).作者: 帶來墨水 時(shí)間: 2025-3-27 22:57
Opioids and Antagonists with aminergics) [1, 2]. The first patient in history to be diagnosed with CP was also opioid unresponsive [3]. Most chronic pain patients on long-term opioids report strong or very strong pain, highlighting their inefficacy [4, 5]. In short-term clinical trials of PNP, the NNT for strong opioids was 4.3 and for tramadol 4.7.作者: Simulate 時(shí)間: 2025-3-28 02:17
https://doi.org/10.1007/978-3-642-82141-7Figs. 8.1 and 8.2), following the establishment of an intracortical S1 . (. (e.g., layer 4? (layer 2/3?) layer 5? layer 6? layer 4+corticothalamic outflow). The attendant sensory information decorrelation translates into different sensory percepts (pain, dysesthesias, paresthesias, pruritus).作者: Musculoskeletal 時(shí)間: 2025-3-28 10:06
Springer Series in Solid-State Sciencesgeminal neuralgia with cocaine injections. Deisenhammer et al. [1] published the first Medline-indexed paper detailing the use of a Na+ channel blocker (carbamazepine) for CP (Tables 13.1, 13.2, 13.3, 13.4, 13.5, 13.6, 13.7, 13.8, 13.9, 13.10, 13.11, 13.12, 13.13, 13.14, 13.15, and 13.16).作者: 小樣他閑聊 時(shí)間: 2025-3-28 10:56
J. Hogg,O. M. Nierstrasz,D. Tsichritzis with aminergics) [1, 2]. The first patient in history to be diagnosed with CP was also opioid unresponsive [3]. Most chronic pain patients on long-term opioids report strong or very strong pain, highlighting their inefficacy [4, 5]. In short-term clinical trials of PNP, the NNT for strong opioids was 4.3 and for tramadol 4.7.作者: Paraplegia 時(shí)間: 2025-3-28 14:41 作者: Scintigraphy 時(shí)間: 2025-3-28 20:46 作者: LEVY 時(shí)間: 2025-3-28 23:51
Notes on the Life History of Coho Salmonnsory maps. The pain may also occur in patients with brain lesions who have recovered from clinically detectable sensory loss, and it may persist in time; in this case, a crude sensory examination, weeks or months after the lesion, reveals no sensory deficit.作者: 熱心助人 時(shí)間: 2025-3-29 05:06
Somatosensory Findingsnsory maps. The pain may also occur in patients with brain lesions who have recovered from clinically detectable sensory loss, and it may persist in time; in this case, a crude sensory examination, weeks or months after the lesion, reveals no sensory deficit.作者: 愛社交 時(shí)間: 2025-3-29 08:32
Book 2018Latest editionresented here are based on the authors’ twenty years of treating patients and conducting research in the field. The book will be an invaluable guide for neurologists, neurosurgeons, anesthesiologists, pain therapists as well as physiotherapists..作者: Camouflage 時(shí)間: 2025-3-29 12:58 作者: ARCH 時(shí)間: 2025-3-29 17:33
Placebo Modulation of Pain chronic pain a disease of salience): different placebos have different mechanisms of action that could in turn influence outcomes [2]. The problem of why placebo or nocebo effects exist in the first place remains unanswered.作者: 消散 時(shí)間: 2025-3-29 23:40
Book 2018Latest editiongh-end pain centers. As a matter of fact, central pain, classified as a form of neuropathic pain, remains too often a scourge for those affected due to the ignorance of pain therapists worldwide and enduring misconceptions at the academic level. By weighing up the relevant evidence, the authors aim 作者: Mundane 時(shí)間: 2025-3-30 01:00 作者: Irremediable 時(shí)間: 2025-3-30 04:53 作者: zonules 時(shí)間: 2025-3-30 12:03
lines based on critical, unbiased assessments of the latest .This book sheds new light on central pain, a field that is largely obscured by lack of knowledge among pain professionals at all levels, including high-end pain centers. As a matter of fact, central pain, classified as a form of neuropathi作者: Grandstand 時(shí)間: 2025-3-30 15:02 作者: MEET 時(shí)間: 2025-3-30 19:22
Properties of Message Addressing Schemeshe function and activation state of the brain, both diffusely and focally. However, other mechanisms of action are possible and are differentiated according to individual drugs (Tables 14.1, 14.2, 14.3, 14.4, 14.5, 14.6, 14.7, 14.8, 14.9, 14.10, and 14.11).作者: 先鋒派 時(shí)間: 2025-3-30 20:59
GABA Agonistsnits. Distribution of the major subunits in various regions of the brain varies. Importantly, GABA neurotransmission can be recoded to become excitatory under certain conditions (Tables 11.1, 11.2, 11.3, 11.4, 11.5, 11.6, and 11.7).
