標(biāo)題: Titlebook: Cellular Function and Metabolism; Yoshio Yazaki (Professor of Medicine),Seibu Mochiz Book 1993 Springer Science+Business Media Dordrecht 1 [打印本頁] 作者: FROM 時間: 2025-3-21 19:56
書目名稱Cellular Function and Metabolism影響因子(影響力)
書目名稱Cellular Function and Metabolism影響因子(影響力)學(xué)科排名
書目名稱Cellular Function and Metabolism網(wǎng)絡(luò)公開度
書目名稱Cellular Function and Metabolism網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Cellular Function and Metabolism被引頻次
書目名稱Cellular Function and Metabolism被引頻次學(xué)科排名
書目名稱Cellular Function and Metabolism年度引用
書目名稱Cellular Function and Metabolism年度引用學(xué)科排名
書目名稱Cellular Function and Metabolism讀者反饋
書目名稱Cellular Function and Metabolism讀者反饋學(xué)科排名
作者: 搖晃 時間: 2025-3-21 22:32
Control of Growth in Neonatal Pig Hearts,l maleate, or an angiotensin receptor antagonist, DuP 753. In the enalapril-treated heart, norepinephrine plus propranolol, but not angiotensin II, accelerated ribosome formation. Rapid growth of the left ventricle of pig heart during the first 10 days of life is due largely to eccentric hypertrophy. (Mol Cell Biochem .: 3-9, 1993)作者: 不如樂死去 時間: 2025-3-22 02:19 作者: outer-ear 時間: 2025-3-22 05:04 作者: PAD416 時間: 2025-3-22 12:48 作者: Transfusion 時間: 2025-3-22 13:12
Mitochondrial DNA Deletion in Human Myocardium,ted to the severity or prognosis of the various diseases, but they indicate that mutation of myocardial mitochondrial DNA can occur in other diseases besides cardiomyopathy, although the influence of aging could not be excluded. (Mol Cell Biochem .: 105–108, 1993)作者: Transfusion 時間: 2025-3-22 18:25 作者: 富饒 時間: 2025-3-23 01:03 作者: Obstreperous 時間: 2025-3-23 01:24
Mechanistic Effects of Kijimicin on Inhibition of Human Immunodeficiency Virus Replication,ach step of HIV replication. The infectivity of progeny viral particles was reduced by Kijimicin treatment. This decrease may be due to incompletely glycosylated forms of gpl20. (Mol Cell Biochem .: 35’41, 1993)作者: Embolic-Stroke 時間: 2025-3-23 08:59 作者: 啜泣 時間: 2025-3-23 12:22
Book 1993iceventsinvolvedin thesynthesis and hydrolysisofATPaswellasforthesynthesisof proteins and phospholipidsareessentialforcellular health.The regulation of cellfunctionisgenerally achieved throughparticipation of a wide varietyofhormones and different signaltransduction mechanisms for theactivation/deac作者: 抗生素 時間: 2025-3-23 15:11 作者: Expediency 時間: 2025-3-23 20:52
Developments in Molecular and Cellular Biochemistryhttp://image.papertrans.cn/c/image/223027.jpg作者: Inclement 時間: 2025-3-23 22:17
,Humanistische Organisationsans?tze,n and protein synthesis cannot be further accelerated during . perfusion with agents that increase cyclic AMP, that bind to α.-adrenergic receptors or that bind to angiotensin II receptors. Growth of the heart . can be restrained by treatment with an angiotensin-converting enzyme inhibitor, enalapri作者: interlude 時間: 2025-3-24 04:01 作者: 招惹 時間: 2025-3-24 10:11
,Humanistische Organisationsans?tze, verify the role of the cardiac growth factor and its purification from hypertrophied hearts. For this propose the hypertrophied hearts of renovascular hypertensive rats were used. The purification was made by using an isoelectric focusing chromatography and the HPLC method. We examined the cardiac 作者: 慢跑鞋 時間: 2025-3-24 10:43 作者: GEN 時間: 2025-3-24 15:39 作者: hypertension 時間: 2025-3-24 19:51
,Humanistische Organisationsans?tze,pplement. We examined Kijimicin for its HIV inhibitory activity. The compound exhibited concentration-dependent inhibition of HIV replication ih primary infected cultures of human T-lymphoblastoid H9 cells. Substantial inhibition of viral replication was observed at concentrations of Kijimicin that 作者: 產(chǎn)生 時間: 2025-3-25 00:04
https://doi.org/10.1007/978-3-531-90185-5(SHR) rats. Hyperthyroidism was induced by daily injections of T. (0.2 mg/kg s.c.) for 14 days. The hearts were then isolated and perfused in the Langendorff mode. ATP, phosphocreatine (PCr), and inorganic phosphate (Pi) were measured continuously by means of .P-nuclear magnetic resonance (NMR) spec作者: 憎惡 時間: 2025-3-25 03:32
https://doi.org/10.1007/978-3-531-90185-5cytes this phenomenon has not been confirmed. The purpose of the present study was to confirm the effect of (1, 4, 5)-IP. on Ca. release from SR in cardiac myocytes. The effect of IP. on Ca. release from SR in hypertrophic cardiac cells was also determined..We examined the effects of IP. on Ca. rele作者: Trypsin 時間: 2025-3-25 10:37
https://doi.org/10.1007/978-3-531-90185-5le activation with different maneuvers was mapped with digital imaging microscopy. During 50 Hz electrical activation (tetanus), the aequorin light intensity from different locations in the muscle fiber rose with very similar time course. Caffeine (10 mM) application, on the other hand, caused aequo作者: 放肆的我 時間: 2025-3-25 13:11
Gert Pickel,Detlef Pollack,J?rg Jacobsrant, and can not provoke any heart-specific disease. Why the proteins can provoke such lethal autoimmune myocarditis has not been completely elucidated. Shortly after cardiac infection or myocardial ischemia, these proteins may work as the antigen for the autoimmune myocardites. First of all, the r作者: craving 時間: 2025-3-25 16:49
Gert Pickel,Detlef Pollack,J?rg Jacobsstigate whether a different origin of the myocardial sample or pretreatment with the cardioprotective agent 2, 3-butanedione-monoxime (BDM) influences the force-frequency- relationship in electrically driven muscle strips taken from failing and nonfailing human myocardium. With as well as without pr作者: 混雜人 時間: 2025-3-25 20:30 作者: Ballad 時間: 2025-3-26 00:11 作者: 避開 時間: 2025-3-26 04:23
https://doi.org/10.1007/978-3-531-90378-1s of aging on human mitochondrial DNA. 8-Hydroxy-deoxyguanosine, a product of free radical damage to deoxyguanosine, is reported to cause random point mutations. In human mitochondrial DNA, 8-hydroxy-deoxyguanosine increased exponentially with age, and the population of mitochondrial DNA with deleti作者: 印第安人 時間: 2025-3-26 09:33 作者: 昏暗 時間: 2025-3-26 15:03 作者: 全面 時間: 2025-3-26 16:49
https://doi.org/10.1007/978-3-531-90378-1f saturation transfer using .P-NMR during the preischemic and reperfusion periods. The hearts were made ischemic for 15 min, followed by 60 min of reperfusion. PRP was almost completely depressed, and recovered to 60% of the control level (preischemic period) after reperfusion. The ATP level during 作者: Confidential 時間: 2025-3-26 23:33 作者: ARBOR 時間: 2025-3-27 01:28
Poetische Orte und regionale Entwicklung,ical parameters. Ischemia was initiated by ligating the left anterior descending coronary artery. When the coronary artery was ligated for 3 min, the levels or glycogen, fructose-1, 6-diphosphate (FDP), adenosine triphosphate and creatinephosphate decreased, and the levels of glucose-6-phosphate (G6作者: penance 時間: 2025-3-27 06:50 作者: Jargon 時間: 2025-3-27 11:45 作者: tackle 時間: 2025-3-27 15:47 作者: Creatinine-Test 時間: 2025-3-27 19:40 作者: homocysteine 時間: 2025-3-27 23:43
https://doi.org/10.1007/978-3-531-90185-5 reduced in comparison with those of hypertrophied myocardium. These results indicate that regression of cardiac hypertrophy is not always associated with a decrease in the number of α1-adrenergic receptors and that the beneficial effects of enalapril in the hypertrophied heart in aortic banding ani作者: 單片眼鏡 時間: 2025-3-28 05:25 作者: puzzle 時間: 2025-3-28 09:17
https://doi.org/10.1007/978-3-531-90185-5od. PCr decreased at every Ca. elevation in both the untreated and hyperthyroid SHR hearts. The PCr/ATP ratio was not altered at any Ca. concentration neither in the untreated SHR nor in the hyperthyroid SHR hearts. The Ca.-induced stepwise elevation in LVDP was higher at any given PCr/Pi ratio in t作者: CUB 時間: 2025-3-28 10:45 作者: jovial 時間: 2025-3-28 18:38
Gert Pickel,Detlef Pollack,J?rg Jacobsts in the heart; on cardio-cytotoxity of the T-lymphocyte and on the precise contribution of cardiac dendritic cells, this autoimmune myocarditis will be more clarified. (Mol Cell Biochem .: 67–71, 1993)作者: 仔細(xì)檢查 時間: 2025-3-28 21:34
https://doi.org/10.1007/978-3-531-90378-1e age of 3 months, p < 0.05; 0.44 vs. 0.88 μm/s at 6 months, p < 0.01; 0.44 vs. 0.67 μm/s at 9 months, p < 0.01; 0.35 vs. 0.52 μm/s at 18 months, p < 0.05). Both Ca.-activated ATPase activity and the percentage of α-myosin heavy chain were also lower in B than in F for each age group. When examined 作者: –scent 時間: 2025-3-29 02:59
https://doi.org/10.1007/978-3-531-90378-1, the decrease in the AAC content ratio was more marked for the animals with more severe myocardial damage. The H.-ATPase activities of mitochondrial membrane were higher in 10-week-old J-2-N hamsters than in control hamsters. These results suggest that the decrease of AAC in J-2-N hamster plays an 作者: 革新 時間: 2025-3-29 06:34 作者: alcohol-abuse 時間: 2025-3-29 07:54 作者: plasma-cells 時間: 2025-3-29 13:27 作者: 不近人情 時間: 2025-3-29 18:44
https://doi.org/10.1007/978-3-531-90378-1ion. Estimation of the metabolites: 6-keto-PGF. (6-KETO) and thromboxane B. (TXB.) was made from the perfusate of isolated Langendorff hearts of guinea-pigs pretreated with 50μg/kg 7-OXO, 24 and 48 h before preparation. Pretreatment alone produced a slight, but significant elevation of 6-KETO (from 作者: somnambulism 時間: 2025-3-29 20:59
Poetische Orte und regionale Entwicklung,onset of ischemia. Iloprost (0.1, 0.3, and 1μg-kg.) reduced the ischemia-induced decrease in energy charge potential to 94, 74, and 86%, respectively, the increase in ([G6P] + [F6P])/[FDP] to 38, 29, 32%, respectively, and the increase in [lactate]/[pyruvate] to 67, 45, 65%, respectively. These resu作者: 昏迷狀態(tài) 時間: 2025-3-30 02:16
Book 1993t with some of these issues from biochemical, molecularbiological,physiological,andpharmacologicalviewpoints. Specialemphasishas been laid on understanding heart function and metabolisminhealthanddiseaseingeneral,and cardiac hypertrophy, heartfailure,andischemicheart disease in particular. It is hop作者: Inertia 時間: 2025-3-30 06:34 作者: 負(fù)擔(dān) 時間: 2025-3-30 08:13
Role of Protein Kinase System in the Signal Transduction of Stretch-Mediated Protooncogene Expressiy via protein kinase C activation. Furthermore, we observed the activation of MAP kinase by myocytes stretching. This result suggests that MAP kinase activation induced by mechanical stimuli might increase the efficiency of protein synthesis on ribosomes induced by mechanical stimuli. (Mol Cell Bioc作者: 變量 時間: 2025-3-30 16:28
Growth Factor for Cardiac Hypertrophy,t growth activities were observed at these five proteins compared to the absence of the fractions. For the appearance of these growth effect, it is necessary that the structure of the protein is there fundamentally as a form with a molecular weight of 27 k dalton. After addition of these isolated fr作者: irreparable 時間: 2025-3-30 16:57
Effect of Angiotensin Coverting Enzyme Inhibitor on Regression in Cardiac Hypertrophy, reduced in comparison with those of hypertrophied myocardium. These results indicate that regression of cardiac hypertrophy is not always associated with a decrease in the number of α1-adrenergic receptors and that the beneficial effects of enalapril in the hypertrophied heart in aortic banding ani作者: 牛的細(xì)微差別 時間: 2025-3-30 22:24 作者: Interferons 時間: 2025-3-31 03:16
Function and Energy Metabolism of Isolated Hearts Obtained from Hyperthyroid Spontaneously Hypertenod. PCr decreased at every Ca. elevation in both the untreated and hyperthyroid SHR hearts. The PCr/ATP ratio was not altered at any Ca. concentration neither in the untreated SHR nor in the hyperthyroid SHR hearts. The Ca.-induced stepwise elevation in LVDP was higher at any given PCr/Pi ratio in t作者: 誰在削木頭 時間: 2025-3-31 05:33 作者: notice 時間: 2025-3-31 10:16 作者: 商品 時間: 2025-3-31 13:22 作者: 釋放 時間: 2025-3-31 19:42 作者: 懶惰人民 時間: 2025-4-1 01:40 作者: 財主 時間: 2025-4-1 01:54
Ischemic changes in myocardial ionic eontents of the isolated perfused rat hearts as studied by NMRbserved in association with a second increase in Pi. This was accompnanied by an abrupt rise of the ventricular end-diastolic pressure. As there was a depletion of ATP at this time, the increase in intracellular Na. and associated decrease in intracellular K. may be explained by inhibition of the Na
