標(biāo)題: Titlebook: Cellular Effects of Heavy Metals; Gaspar Banfalvi Book 2011 Springer Science+Business Media B.V. 2011 accumulation.apoptosis.carcinogenesi [打印本頁(yè)] 作者: Inspection 時(shí)間: 2025-3-21 17:04
書目名稱Cellular Effects of Heavy Metals影響因子(影響力)
書目名稱Cellular Effects of Heavy Metals影響因子(影響力)學(xué)科排名
書目名稱Cellular Effects of Heavy Metals網(wǎng)絡(luò)公開度
書目名稱Cellular Effects of Heavy Metals網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Cellular Effects of Heavy Metals被引頻次
書目名稱Cellular Effects of Heavy Metals被引頻次學(xué)科排名
書目名稱Cellular Effects of Heavy Metals年度引用
書目名稱Cellular Effects of Heavy Metals年度引用學(xué)科排名
書目名稱Cellular Effects of Heavy Metals讀者反饋
書目名稱Cellular Effects of Heavy Metals讀者反饋學(xué)科排名
作者: 難解 時(shí)間: 2025-3-21 20:58 作者: N防腐劑 時(shí)間: 2025-3-22 02:21 作者: slow-wave-sleep 時(shí)間: 2025-3-22 06:07
as a Model Organism for Elucidating Arsenic Tolerance Mechanisms of key yeast proteins involved in arsenite accumulation and efflux, arsenate reduction, and the use of complementation assays where a yeast protein is replaced by a homologous protein from another organism, has accelerated the identification of arsenic tolerance genes in fungi, plants, animals, and作者: 激怒某人 時(shí)間: 2025-3-22 09:11
Heavy Metal Toxicity in an Insect Cell Line (Methyl-HgCl, HgCl2, CdCl2 and CuSO4)proliferation was drastically inhibited by CdCl. and MeHgCl while HgCl. showed only a moderate effect. Acute toxicity tests showed that membrane integrity was severely affected in cells treated with low doses of HgCl. and MeHgCl. Uptake of both mercury species occurred via simple diffusion while cad作者: Criteria 時(shí)間: 2025-3-22 13:45
Cellular Changes in Mammalian Cells Induced by Cadmiumreplication, DNA repair) and morphological (chromatin) changes in CHO and murine preB cells. Low Cd concentration (0.5–1?μM) interferes with both replicative and repair DNA synthesis. The replicative DNA synthesis is gradually inhibited, but this inhibition is less than the increment of repair DNA s作者: Criteria 時(shí)間: 2025-3-22 17:13 作者: Flinch 時(shí)間: 2025-3-22 23:35 作者: 培養(yǎng) 時(shí)間: 2025-3-23 05:05 作者: vibrant 時(shí)間: 2025-3-23 07:33
Heavy Metal-Induced Carcinogenicity: Depleted Uranium and Heavy-Metal Tungsten Alloy whose toxicological properties may not as yet be well-understood. This chapter reviews what is currently known about two recent additions to many nations’ arsenals: depleted uranium and heavy-metal tungsten alloy. The toxicological and genotoxic properties of these materials, derived from both . an作者: Haphazard 時(shí)間: 2025-3-23 12:16 作者: Phonophobia 時(shí)間: 2025-3-23 16:43 作者: condone 時(shí)間: 2025-3-23 20:01 作者: CHAFE 時(shí)間: 2025-3-24 00:07
https://doi.org/10.1007/978-94-007-0428-2accumulation; apoptosis; carcinogenesis; genotoxicity; removal; ecotoxicology作者: LUCY 時(shí)間: 2025-3-24 05:26
978-94-007-9225-8Springer Science+Business Media B.V. 2011作者: 遭受 時(shí)間: 2025-3-24 10:27
Gaspar BanfalviDescribes the biological importance of heavy metals.Shows how heavy metals enter cells.What happens inside the cells upon heavy metal uptake.What chemical reactions are induced by heavy metals.What me作者: cyanosis 時(shí)間: 2025-3-24 11:55 作者: chemical-peel 時(shí)間: 2025-3-24 17:31 作者: forestry 時(shí)間: 2025-3-24 19:11
https://doi.org/10.1007/978-3-322-99332-8l/metalloid tolerance of fungi. Yeast-based models are highly applicable when metal/metalloid transport and compartmentalization processes are mapped in other fungal species or higher eukaryotes but this approach has limitations, which necessitates further fungal models evolutionarily closer to heav作者: vasculitis 時(shí)間: 2025-3-24 23:52 作者: Inoperable 時(shí)間: 2025-3-25 04:09
https://doi.org/10.1007/978-3-322-99355-7 of key yeast proteins involved in arsenite accumulation and efflux, arsenate reduction, and the use of complementation assays where a yeast protein is replaced by a homologous protein from another organism, has accelerated the identification of arsenic tolerance genes in fungi, plants, animals, and作者: 闖入 時(shí)間: 2025-3-25 09:00
Entscheidungs- und Organisationstheorieproliferation was drastically inhibited by CdCl. and MeHgCl while HgCl. showed only a moderate effect. Acute toxicity tests showed that membrane integrity was severely affected in cells treated with low doses of HgCl. and MeHgCl. Uptake of both mercury species occurred via simple diffusion while cad作者: Insulin 時(shí)間: 2025-3-25 14:59
Darstellungsmethoden im überblickreplication, DNA repair) and morphological (chromatin) changes in CHO and murine preB cells. Low Cd concentration (0.5–1?μM) interferes with both replicative and repair DNA synthesis. The replicative DNA synthesis is gradually inhibited, but this inhibition is less than the increment of repair DNA s作者: Allowance 時(shí)間: 2025-3-25 16:56
Meilensteine der Organometallchemies on chromatin organization are reported relative to dose and duration of Ni(II) incubation, with gradation of apoptotic to necrotic changes manifested as the dosage increases. Ni(II) generated citotoxic effects in K562 cells at low concentration with an estimated IC50 value of 2?μM. Chromatotoxicit作者: ostracize 時(shí)間: 2025-3-25 21:41
Peter Hauptmanns,Bettina Landers chapter is placed on large-scale chromatin structures of fungal nuclei, isolated from protoplasts of the fission yeast .and visualized for the first time by fluorescence microscopy. Although, the pattern of chromosome condensation was similar to mammalian cells, but less compact and resembled more作者: RAG 時(shí)間: 2025-3-26 01:16 作者: 逃避責(zé)任 時(shí)間: 2025-3-26 06:06 作者: forthy 時(shí)間: 2025-3-26 09:17
Peter Hauptmanns,Bettina LanderCarcinogenic metals, e.g., Be, Cd, Cr, Co, Ni, and the metalloid As, are capable of generating various kinds of active oxygen and other reactive species in direct redox reactions with O., O. ., H.O., organic peroxides, and other cellular or tissue substrates, and/or indirectly—by inducing inflammati作者: Meander 時(shí)間: 2025-3-26 13:23 作者: Coronation 時(shí)間: 2025-3-26 20:38 作者: CLASP 時(shí)間: 2025-3-26 22:30 作者: ANIM 時(shí)間: 2025-3-27 01:15
Darstellungsmethoden im überblick10–40-times higher in S phase upon Cd treatment. The oxidative DNA damage product 8-oxodeoxyguanosine was induced at low (0.5?μM) CdCl. concentration. Among the interphase chromatin damages the most characteristic feature was the appearance of holes and disruptions in nuclei which are regarded as typical diagnostic symptoms of cadmium toxicity.作者: 離開可分裂 時(shí)間: 2025-3-27 06:55
Cellular Changes in Mammalian Cells Induced by Cadmium10–40-times higher in S phase upon Cd treatment. The oxidative DNA damage product 8-oxodeoxyguanosine was induced at low (0.5?μM) CdCl. concentration. Among the interphase chromatin damages the most characteristic feature was the appearance of holes and disruptions in nuclei which are regarded as typical diagnostic symptoms of cadmium toxicity.作者: ingestion 時(shí)間: 2025-3-27 09:43
.What chemical reactions are induced by heavy metals.What me.The term “heavy metals” is used as a group name of toxic metals and metalloids (semimetals) causing contaminations and ecotoxicity. In strict chemical sense the density of heavy metals is higher than 5 g/cm3. From biological point of作者: 演繹 時(shí)間: 2025-3-27 14:45 作者: 豐滿有漂亮 時(shí)間: 2025-3-27 20:38 作者: Ingest 時(shí)間: 2025-3-27 22:15 作者: myriad 時(shí)間: 2025-3-28 03:06 作者: ACRID 時(shí)間: 2025-3-28 07:16
Erhebung von Zeit- und Mengendatenbstrates and regulatory mechanisms of budding yeast SCF., fission yeast SCF., and the mammalian Cul3/Keap1 ligase. The similarities and differences of these ubiquitin ligases that coordinate the cadmium stress response in yeast and human will be highlighted to describe the different mechanisms for cadmium sensing and detoxification.作者: Subjugate 時(shí)間: 2025-3-28 12:09 作者: BROTH 時(shí)間: 2025-3-28 17:46
pects of environmental hazard, ecosystems and human health. Among the cellular responses of heavy metals irregularities in cellular mechanisms such as gene expression, protein folding, stress signaling pathways978-94-007-9225-8978-94-007-0428-2作者: 使習(xí)慣于 時(shí)間: 2025-3-28 20:57
Book 2011oxic, mutagenic and carcinogenic effects. The toxicity of several metals touch upon the aspects of environmental hazard, ecosystems and human health. Among the cellular responses of heavy metals irregularities in cellular mechanisms such as gene expression, protein folding, stress signaling pathways作者: 反復(fù)拉緊 時(shí)間: 2025-3-29 02:23 作者: 修飾語(yǔ) 時(shí)間: 2025-3-29 04:01
Toxic Metal/Metalloid Tolerance in Fungi—A Biotechnology-Oriented Approachracellular metal chelators, (v) overproduction of elements of the antioxidative defense system, (vi) genetic modification of the regulatory network of metal/metalloid stress defense, and (vii) interfering with the metal/metalloid-dependent initialization of apoptotic cell death. Owing to the wide-sp作者: 托人看管 時(shí)間: 2025-3-29 08:52
Interference of Chromium with Cellular Functions transport of Cr(VI) and Cr(III) through non-selective and selective ion channels and yeast chromium mutants that are able to take up very low concentrations of chromium. This uptake-reduction model helps to explain the generation of reactive oxygen species (ROS), which are primarily responsible for作者: 清洗 時(shí)間: 2025-3-29 15:26 作者: 分貝 時(shí)間: 2025-3-29 16:03
Chromatin Toxicity of Ni(II) Ions in K562 Erythroleukemia Cellsion 1?μM NiCl. generated larger apoptotic chromatin circles. At 5?μM NiCl. the slight expansion of nuclei and the continuous chromatin ribbon became visible. At 10?μM Ni(II) most of the nuclear material condensed prematurely, the rest remained in highly decondensed fibrillary stage. Necrotic changes作者: anus928 時(shí)間: 2025-3-29 20:30 作者: uncertain 時(shí)間: 2025-3-30 01:20 作者: 流眼淚 時(shí)間: 2025-3-30 07:07
Role of Oxidative Damage in Metal-Induced Carcinogenesisicity of DNA base products, strand breaks, apurinic sites, cross-links, and adducts typical for the attacking reactive species (oxygen-, carbon-, or sulfur-centered radicals), originating from oxidized amino acids and proteins, and 4-hydroxynonenal, a lipid oxidation product. Oxidative damage to nuc作者: 鉗子 時(shí)間: 2025-3-30 10:26
Non-native Proteins as Newly-Identified Targets of Heavy Metals and Metalloidsuantitative deficiency of the affected proteins and the formation of proteotoxic aggregates. Possibly, the toxic effects of heavy metals and metalloids arise not only from their interaction with specific, particularly susceptible native proteins but also from a general derailing of protein folding. 作者: figment 時(shí)間: 2025-3-30 16:19
