標(biāo)題: Titlebook: Cardiovascular Disease; Cellular and Molecul Linda L. Gallo Book 1995 Springer Science+Business Media New York 1995 Lipid.Oxidation.cardiov [打印本頁] 作者: 黑暗社會(huì) 時(shí)間: 2025-3-21 19:20
書目名稱Cardiovascular Disease影響因子(影響力)
作者: Postmenopause 時(shí)間: 2025-3-21 21:27
https://doi.org/10.1007/978-3-642-84708-0ural stability of lipoproteins but also in their metabolism has led to the separation of HDL into further subpopulations according to their apolipoprotein composition rather than their physicochemical properties.作者: Minatory 時(shí)間: 2025-3-22 03:25 作者: 人造 時(shí)間: 2025-3-22 08:32 作者: NOCT 時(shí)間: 2025-3-22 11:39 作者: MAOIS 時(shí)間: 2025-3-22 16:29 作者: MAOIS 時(shí)間: 2025-3-22 20:02 作者: 沒有貧窮 時(shí)間: 2025-3-22 21:32
Antioxidants and Endothelial Expression of Vcam-1: A Molecular Paradigm for Atherosclerosislammatory and growth responses, represents an intriguing paradigm for understanding atherogenesis as well as for the development of novel therapeutic treatment regimens, drug design and diagnostic assessment of disease state.作者: adulterant 時(shí)間: 2025-3-23 05:01
Proteinases and Restenosis: Matrix Metalloproteinase and their Inhibitor and Activatorion. The breakdown of this highly organized extracellular environment is controlled, for the most part, by a gene family of matrix metalloproteinases (MMPs) which collectively can degrade virtually all components of connective tissues..作者: GLUE 時(shí)間: 2025-3-23 05:33 作者: 惹人反感 時(shí)間: 2025-3-23 11:53 作者: phlegm 時(shí)間: 2025-3-23 14:24
Algebraic Model of Hadronic Structure,be mediated by binding of HDL to a specific cell-surface receptor. Through this receptor interaction, HDL may continually deplete peripheral cells of excess cholesterol, thereby preventing the buildup of cholesterol in artery wall cells that is an early feature of the developing atherosclerotic lesion.作者: Nomogram 時(shí)間: 2025-3-23 20:58 作者: ASSAY 時(shí)間: 2025-3-23 22:59 作者: Ptsd429 時(shí)間: 2025-3-24 06:02 作者: 輕而薄 時(shí)間: 2025-3-24 07:16
The Defect in HDL3 Mediated Efflux of Newly Synthesized Cholesterol is Associated with Impaired Actiisk and plasma levels of HDL-cholesterol.. In addition to unspecific cholesterol desorption from the plasma membrane., depletion of cholesterol may be achieved by binding of HDL apolipoproteins to specific surface receptors and subsequent induction of cholesterol transport from internal stores to the plasma membrane .作者: ineluctable 時(shí)間: 2025-3-24 13:00
Book 1995 countries to review and update research on cardiovascular disease. This group satisfied the symposium goals of formulating a more comprehensive and integrated picture of the events contributing to atherosclerosis and of exploring modified gene expression as an to understanding the causes of atheros作者: 東西 時(shí)間: 2025-3-24 15:15 作者: vocation 時(shí)間: 2025-3-24 22:30 作者: 諄諄教誨 時(shí)間: 2025-3-25 00:48 作者: 獨(dú)白 時(shí)間: 2025-3-25 04:06 作者: capsule 時(shí)間: 2025-3-25 10:49 作者: Spirometry 時(shí)間: 2025-3-25 12:01
The Regulation of Normal and Pathological Angiogenesis by Vascular Endothelial Growth Factor the treatment of such disorders. Conversely, disorders characterized by inadequate tissue perfusion such as obstructive atherosclerosis and diabetes are expected to benefit from agents able to promote endothelial cell growth and neovascularization作者: Cocker 時(shí)間: 2025-3-25 16:25
New Developments in Activation Analysision. The breakdown of this highly organized extracellular environment is controlled, for the most part, by a gene family of matrix metalloproteinases (MMPs) which collectively can degrade virtually all components of connective tissues..作者: CAMEO 時(shí)間: 2025-3-25 22:50 作者: 名義上 時(shí)間: 2025-3-26 03:15 作者: 伙伴 時(shí)間: 2025-3-26 06:50
Triglyceride-Rich Lipoprotein Metabolism and DiabetesThe most frequent form of hyperlipidemia in diabetes is hypertriglyceridemia.. Therefore, this paper will focus on triglyceride-rich lipoprotein metabolism. The major metabolic effects of diabetes stem from abnormalities in insulin. Hence, the chapter will deal with the effect of insulin on triglyceride-rich lipoprotein metabolism.作者: 動(dòng)物 時(shí)間: 2025-3-26 08:32
Gwumc Department of Biochemistry and Molecular Biology Annual Spring Symposiahttp://image.papertrans.cn/c/image/221920.jpg作者: Urgency 時(shí)間: 2025-3-26 13:48
Peculiar Processes in Glass Surface Analysisept that there are acute “triggering” risk factors for plaque disruption has been described in detail elsewhere (., ., .) and we will summarize the salient aspects of this approach to understanding coronary artery disease.作者: Gnrh670 時(shí)間: 2025-3-26 20:42 作者: 煩憂 時(shí)間: 2025-3-26 21:54
978-1-4613-5805-3Springer Science+Business Media New York 1995作者: AGATE 時(shí)間: 2025-3-27 05:10
Peculiar Processes in Glass Surface Analysisept that there are acute “triggering” risk factors for plaque disruption has been described in detail elsewhere (., ., .) and we will summarize the salient aspects of this approach to understanding coronary artery disease.作者: Ceramic 時(shí)間: 2025-3-27 07:33
https://doi.org/10.1007/3-540-55625-7tion and sudden cardiac death. These disparate syndromes occur along a clinical continuum linked by a common pathophysiologic event, intracoronary thrombosis (.–.). Thrombosis is initiated by rupture of atherosclerotic plaque and exposure of highly thrombogenic plaque constituents to coronary blood 作者: 使成整體 時(shí)間: 2025-3-27 13:29
New Developments in Activation Analysisesis, wound healing/repair, inflammation, embryonic development, post-partum involution of the uterus, bone and growth plate remodeling, ovulation, and the progression of certain diseases.. The process of connective tissue restructuring represents a balance between matrix production and its degradat作者: 是剝皮 時(shí)間: 2025-3-27 13:38 作者: 一再煩擾 時(shí)間: 2025-3-27 18:05
Positrons as Solid State Probesinto the femoral vein of a rabbit, the LDL was found trapped in the three dimensional matrix network.. The trapped LDL appeared to bind to the collagen fibers near the junction of connecting fibrils.. The bound LDL was no longer in solution and appeared to be trapped in a microenvironment that exclu作者: 受人支配 時(shí)間: 2025-3-27 23:57 作者: 前奏曲 時(shí)間: 2025-3-28 05:07
The Lebedev Physics Institute Series was first characterized as the “arginine-rich” peptide). Subsequently its properties have been virtually completely described.: it is a peptide composed of 299 amino acids with a molecular weight of 34,000 daltons. It contains 22 amino acid repeats, forming amphipathic helices. It is synthesized pr作者: 有限 時(shí)間: 2025-3-28 06:26 作者: Gnrh670 時(shí)間: 2025-3-28 10:58 作者: FIR 時(shí)間: 2025-3-28 17:46 作者: rods366 時(shí)間: 2025-3-28 21:16 作者: 無畏 時(shí)間: 2025-3-29 00:49
https://doi.org/10.1007/978-3-642-84708-0ed risk of premature coronary artery disease (CAD). have generated interest in the mechanism through which HDL prevents atherosclerosis. The HDL have been historically defined as lipoproteins with densities between 1.063 and 1.20 g/ml.. Human HDL consists of a collection of particles differing in si作者: Creatinine-Test 時(shí)間: 2025-3-29 05:17 作者: Abbreviate 時(shí)間: 2025-3-29 09:49 作者: 有惡臭 時(shí)間: 2025-3-29 11:38 作者: 長矛 時(shí)間: 2025-3-29 17:04
S. Guiducci,G. Martinelli,M. A. Preger and the migration and activation of macrophages are among the most important.. The responses of normal cells that lead to atherosclerosis are the consequences of directed signals that are initiated by cytokines that are upregulated at sites of developing lesions and by circulating plasma factors, s作者: 單純 時(shí)間: 2025-3-29 20:59
Electromagnetic sum rules in nuclei,ngiogenesis”, is prominent during embryonic development and somatic growth but in a normal adult it only takes place following injury or, in a cyclical fashion, in the endometrium and in the ovary (., .). Angiogenesis plays a significant role in the pathogenesis of a variety of disorders including c作者: 思想 時(shí)間: 2025-3-30 02:32 作者: 失誤 時(shí)間: 2025-3-30 05:08 作者: visceral-fat 時(shí)間: 2025-3-30 11:05
Apolipoprotein E: Paradoxes Aboundensity lipoproteins [IDL], and high density lipoproteins [HDL]) except low density lipoproteins (LDL) (though traces of apo E may be found in more buoyant LDL particles, especially in animal species other than the human). Like apo B, it serves as a ligand for the E/B (LDL) receptor, and its binding 作者: 單挑 時(shí)間: 2025-3-30 14:30
Cholesterol Efflux from Cells in Culture: Studies with Lipid-Free Acceptors, Reconstituted Particlesthe release of radiolabeled cholesterol from the cells. In the studies using lipid-free peptides or reconstituted particles the release of the labeled cholesterol reflects the net movement of cholesterol from cells to acceptors, since the acceptors were initially free of cholesterol and no significa作者: 轉(zhuǎn)換 時(shí)間: 2025-3-30 20:01
Identification of FGF-1-Inducible Genes by Differential Displayas also been detected in human platelets. and T lymphocytes.. Direct evidence for the involvement of FGF-2 in rat balloon injury-induced medial smooth muscle cell proliferation was reported by Lindner and Reidy.. Therefore, it is possible that inhibition of FGF expression or action may be of therape作者: 致敬 時(shí)間: 2025-3-30 22:14
Mechanisms of Potentiation of Pdgf in Atherosclerosisoma.. Its protein product, the homodimeric PDGF A-chain, is a chemoattractant for monocytes, fibroblasts, and smooth muscle cells and a mitogen for fibroblasts and smooth muscle cells . In addition, PDGF A upregulates its own transcription (and thus is autoregulatoryll) and transcription of a number作者: debris 時(shí)間: 2025-3-31 04:02 作者: 召集 時(shí)間: 2025-3-31 08:52
Book 1995ivided into eight sections which reflect the focus of the plenary sessions. Part I focuses on the pathophysiology of atherosclerotic plaques and predicts that the nature of the fibrous cap of atheroma determines plaque disruption and clinical events. Papers in Part II deal with atherogenic lipoprote作者: 方便 時(shí)間: 2025-3-31 12:05
olume is divided into eight sections which reflect the focus of the plenary sessions. Part I focuses on the pathophysiology of atherosclerotic plaques and predicts that the nature of the fibrous cap of atheroma determines plaque disruption and clinical events. Papers in Part II deal with atherogenic lipoprote978-1-4613-5805-3978-1-4615-1959-1作者: CAMP 時(shí)間: 2025-3-31 16:03 作者: 哎呦 時(shí)間: 2025-3-31 20:41
The Lebedev Physics Institute Seriesensity lipoproteins [IDL], and high density lipoproteins [HDL]) except low density lipoproteins (LDL) (though traces of apo E may be found in more buoyant LDL particles, especially in animal species other than the human). Like apo B, it serves as a ligand for the E/B (LDL) receptor, and its binding 作者: 狗窩 時(shí)間: 2025-4-1 01:15 作者: 入會(huì) 時(shí)間: 2025-4-1 05:16
S. Guiducci,G. Martinelli,M. A. Pregeras also been detected in human platelets. and T lymphocytes.. Direct evidence for the involvement of FGF-2 in rat balloon injury-induced medial smooth muscle cell proliferation was reported by Lindner and Reidy.. Therefore, it is possible that inhibition of FGF expression or action may be of therape作者: 獨(dú)裁政府 時(shí)間: 2025-4-1 07:04
S. Guiducci,G. Martinelli,M. A. Pregeroma.. Its protein product, the homodimeric PDGF A-chain, is a chemoattractant for monocytes, fibroblasts, and smooth muscle cells and a mitogen for fibroblasts and smooth muscle cells . In addition, PDGF A upregulates its own transcription (and thus is autoregulatoryll) and transcription of a number作者: 大漩渦 時(shí)間: 2025-4-1 11:50
Nuclear Physics with Polarized ParticlesCs and SMCs in atherosclerotic plaques.. These similarities have led to the suggestion that SMCs in atherosclerosis and hypertension may recapitulate certain aspects of earlier developmental events.. To permit a critical analysis of this possibility, it is crucial that we obtain a better understandi作者: 主講人 時(shí)間: 2025-4-1 18:18
Lipid Accumulation and Plaque Disruption: Processes Triggering Clinical Instability in Coronary Diseept that there are acute “triggering” risk factors for plaque disruption has been described in detail elsewhere (., ., .) and we will summarize the salient aspects of this approach to understanding coronary artery disease.作者: 失望昨天 時(shí)間: 2025-4-1 21:20
Expression of 92 KDA Gelatinase in Human Atherosclerotic Lesions Following Recent Plaque Rupturetion and sudden cardiac death. These disparate syndromes occur along a clinical continuum linked by a common pathophysiologic event, intracoronary thrombosis (.–.). Thrombosis is initiated by rupture of atherosclerotic plaque and exposure of highly thrombogenic plaque constituents to coronary blood
