標(biāo)題: Titlebook: Cardiac Cytoarchitecture; How to Maintain a Wo Elisabeth Ehler Book 2015 Springer International Publishing Switzerland 2015 Actin and Myosi [打印本頁(yè)] 作者: Menthol 時(shí)間: 2025-3-21 19:20
書目名稱Cardiac Cytoarchitecture影響因子(影響力)
作者: 漂浮 時(shí)間: 2025-3-21 20:43
Ca2+ Regulation of the Cardiac Thin Filament,ltimately allow the force generation required for the maintenance of normal cardiac pump function. This chapter seeks to delve into the biochemistry and biophysics that dictate the cellular physiology of the myofilaments.作者: Basal-Ganglia 時(shí)間: 2025-3-22 00:22 作者: 可商量 時(shí)間: 2025-3-22 04:40 作者: excrete 時(shí)間: 2025-3-22 09:50 作者: 協(xié)奏曲 時(shí)間: 2025-3-22 14:08 作者: 協(xié)奏曲 時(shí)間: 2025-3-22 17:53 作者: FEAS 時(shí)間: 2025-3-22 22:48 作者: Efflorescent 時(shí)間: 2025-3-23 02:18 作者: 輕率看法 時(shí)間: 2025-3-23 05:48
https://doi.org/10.1007/978-3-540-68430-5 the ID is presented. Because of its complex nature, it is not surprising that the ID has been implicated in a number of heart diseases and malfunctions. Changes in its structure and composition associated with these heart problems are described with special regard to the role of the ID in dilated cardiomyopathy.作者: 大酒杯 時(shí)間: 2025-3-23 11:31 作者: 使閉塞 時(shí)間: 2025-3-23 15:24 作者: anachronistic 時(shí)間: 2025-3-23 18:59 作者: Jejune 時(shí)間: 2025-3-23 23:05 作者: 誘拐 時(shí)間: 2025-3-24 03:08
https://doi.org/10.1007/978-3-540-68430-5isease such as hypertrophic cardiomyopathy, dilated cardiomyopathy or arrhythmogenic right ventricular cardiomyopathy, these structures can be compromised in their composition and thus function. The aim of this chapter is to discuss how cardiac cytoarchitecture is established during development and how it is altered in disease.作者: 敲竹杠 時(shí)間: 2025-3-24 10:07
Nonlinear Magnetoelastic Interactions,ltimately allow the force generation required for the maintenance of normal cardiac pump function. This chapter seeks to delve into the biochemistry and biophysics that dictate the cellular physiology of the myofilaments.作者: 他一致 時(shí)間: 2025-3-24 14:24
Nonlinear Theory of Shallow Shellsin modulating cardiomyocyte stiffness. This review provides an overview on the current knowledge of signaling pathways and kinases involved in phosphorylating I-band titin in healthy and diseased hearts and further highlights some recent findings that suggest posttranslational adaptation of titin stiffness in response to oxidative stress.作者: cartilage 時(shí)間: 2025-3-24 17:01
Nonlinear Theory of Shallow Shellsons of the AMPK pathway, in particular those relevant for the heart. Activation of the kinase is involved in the myocardial response to ischemia, pressure overload, and heart failure. Pharmacological activation of AMPK may prove to be a useful therapeutic strategy in the treatment of these pathologies.作者: anticipate 時(shí)間: 2025-3-24 20:37
https://doi.org/10.1007/978-3-540-68430-5rt, and the intercalated discs, a special type of cell–cell contact that serves for mechanical and electrochemical connection between individual cardiomyocytes, are multiprotein complexes that must be assembled in a regular fashion during development to guarantee a fully functional heart. In heart d作者: Processes 時(shí)間: 2025-3-25 01:07 作者: 可商量 時(shí)間: 2025-3-25 05:27 作者: gastritis 時(shí)間: 2025-3-25 07:52 作者: crease 時(shí)間: 2025-3-25 14:21 作者: LIEN 時(shí)間: 2025-3-25 17:50 作者: 背書 時(shí)間: 2025-3-25 23:06 作者: 冷峻 時(shí)間: 2025-3-26 02:52
Nonlinear Theory of Shallow Shellsg filament mechanism is the basis of contractility. Furthermore, sarcomeres contain a multitude of regulatory proteins, which are not directly involved in force generation. Many of them interact with key parts of intracellular signaling cascades. Thus, sarcomeres are increasingly noted as central hu作者: 安慰 時(shí)間: 2025-3-26 05:58 作者: 鍵琴 時(shí)間: 2025-3-26 10:43 作者: 偽書 時(shí)間: 2025-3-26 15:03 作者: Vsd168 時(shí)間: 2025-3-26 20:12
General Mathematical Questions, is critical, as increased protein synthesis is linked to hypertrophy of the heart, whereas increased degradation is usually associated with atrophy. Hypertrophy and atrophy of the heart are just two examples of cardiomyopathies, where the cellular equilibrium between synthesis and degradation is ou作者: Alopecia-Areata 時(shí)間: 2025-3-26 21:36
Elisabeth EhlerGives a concise and comprehensive overview of cardiac cytoarchitecture.Treats the different subcellular compartments of the cardiomyocyte.Addresses development, disease and basic cell biological quest作者: reptile 時(shí)間: 2025-3-27 04:28
http://image.papertrans.cn/c/image/221749.jpg作者: 向外供接觸 時(shí)間: 2025-3-27 08:16 作者: Generalize 時(shí)間: 2025-3-27 11:08
978-3-319-34653-3Springer International Publishing Switzerland 2015作者: maverick 時(shí)間: 2025-3-27 14:32 作者: Subdue 時(shí)間: 2025-3-27 18:46 作者: 重疊 時(shí)間: 2025-3-27 23:51
The M-Band: Not Just Inert Glue but Playing an Active Role in the Middle of the Sarcomere,ering active contractile force, the M-band is the origin of signalling processes that can go all the way to the nucleus to affect gene transcription. The mechanical deformations of the M-band filaments during muscle contraction allow the integration of smart molecular sensors to monitor the activity作者: 混雜人 時(shí)間: 2025-3-28 04:37 作者: 無(wú)動(dòng)于衷 時(shí)間: 2025-3-28 06:23
The Nuclear Envelope in Cardiac Health and Disease,ns to these components result in disease phenotypes, which vary in severity and tissue specificity and are often broadly termed ‘premature ageing disorders’. One of the key phenotypes frequently present is cardiomyopathy. As a result, the NE is becoming increasingly relevant in the context of cardia作者: 樂章 時(shí)間: 2025-3-28 13:04
https://doi.org/10.1007/978-3-540-68430-5thick myosin filaments and thin actin filaments that slide past each other and shorten the sarcomere in response to an action potential. Because all sarcomeres within a cell are aligned, and all cells in a tissue are aligned, the amount of uniaxial force generated by the tissue as a whole is maximiz作者: Harass 時(shí)間: 2025-3-28 17:31
Nonlinear Electroelastic Interactions,other proteins have been proposed to contribute. For instance, tropomyosin plays a vital role in stabilizing the thin filament. In addition, cyclase-associated protein 2 (CAP2) has been proposed to regulate thin filament length by severing filamentous actin and sequestering globular actin. Actin-mon作者: 影響 時(shí)間: 2025-3-28 21:03 作者: 蒙太奇 時(shí)間: 2025-3-29 00:08 作者: 冒煙 時(shí)間: 2025-3-29 04:25 作者: Notorious 時(shí)間: 2025-3-29 10:47 作者: Adherent 時(shí)間: 2025-3-29 12:47
