標(biāo)題: Titlebook: Calcium, Neuronal Function and Transmitter Release; Proceedings of the S Rami Rahamimoff,Bernard Katz Conference proceedings 1986 Martinus [打印本頁] 作者: malcontented 時(shí)間: 2025-3-21 18:49
書目名稱Calcium, Neuronal Function and Transmitter Release影響因子(影響力)
書目名稱Calcium, Neuronal Function and Transmitter Release影響因子(影響力)學(xué)科排名
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書目名稱Calcium, Neuronal Function and Transmitter Release被引頻次
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書目名稱Calcium, Neuronal Function and Transmitter Release讀者反饋
書目名稱Calcium, Neuronal Function and Transmitter Release讀者反饋學(xué)科排名
作者: Directed 時(shí)間: 2025-3-21 22:38 作者: browbeat 時(shí)間: 2025-3-22 00:53
Modulation Of Calcium Channels in Cardiac Cells by Neurotransmitters channels. Adrenergic neurotransmitter effects on Ca currents, I. in cardiac cells have served as prime examples of modulation of voltagegated ion channels. Recently it has become possible to analyze molecular events Involved in Ca channel modulation. Ca channels are activated by changes in membrane作者: Inflamed 時(shí)間: 2025-3-22 07:04 作者: 可憎 時(shí)間: 2025-3-22 10:38 作者: fertilizer 時(shí)間: 2025-3-22 13:23 作者: fertilizer 時(shí)間: 2025-3-22 18:15 作者: Contend 時(shí)間: 2025-3-23 00:12
Persistent Inactivation of Potassium Currents by Elevation of Intracellular Calciumtoreceptor, remain reduced for days after associatively conditioning the nudibranch mollusc .. Because intracellular calcium, Ca.. (as measured by differential absorption spectrophotometry of calcium complexed with the dye arsenazo III), undergoes prolonged elevation during the acquisition of the co作者: Goblet-Cells 時(shí)間: 2025-3-23 02:40
Some Consequences of Intracellular Calcium Binding on Phasic Synaptic Transmitter Releasear junction. Katz & Miledi (1) showed that external calcium was necessary at the time of the first nerve impulse and suggested that calcium entered the terminal and, by binding to some receptor site at the presynaptic terminal, released transmitter. The “residual calcium” hypothesis for facilitation作者: HARP 時(shí)間: 2025-3-23 09:20
Calcium Cooperativity in Calcium Entry and Calcium Action, and its Implications with Regard to Facilrnal medium via voltage-regulated cation-selective channels (reviews, 1, 2, 3). At the vertebrate neuromuscular junction this dependence is manifest in essentially the same way for release provoked by nerve impulses (i.e., end-plate potentials, EPPs) or release manifest as a raised frequency of mini作者: DIKE 時(shí)間: 2025-3-23 09:54
Resting Calcium Levels and Evoked Release at the Neuromuscular Junctionnated subsequent work on this problem. We have modified their model by adding one additional parameter: the level of Ca in the resting nerve terminal. This single addition substantially alters the behavior of the model and makes it necessary to reinterpret many previous measurements. We have present作者: 與野獸博斗者 時(shí)間: 2025-3-23 14:29 作者: 水汽 時(shí)間: 2025-3-23 19:43
Calcium Independent Quantal Transmitter Release at the Neuromuscular Junctionarization-transmitter secretion coupling to occur (29). Nerve impulse-evoked multi-quantal release of acetylcholine (ACh), giving rise to the end-plate potential (e.p.p.), as well as the spontaneous quantal secretion of ACh, causing the miniature end-plate potentials (m.e.p.ps), depend on extra- and作者: Pseudoephedrine 時(shí)間: 2025-3-24 00:47
On the Voltage Dependence of Neurotransmitter Releaseellular Ca. concentration (3,4) resulting from terminal depolarization and inflow of calcium. Indeed, this is considered the only role of membrane depolarization; no additional effects are presumed in any of the processes that follow the entry of calcium ions and lead to the fusion of synaptic vesic作者: fatuity 時(shí)間: 2025-3-24 03:36
A Possible Involvement of the Na-Ca Exchanger in Regulation of Transmitter Release at the Frog Neuronson, 1957; Dodge & Rahamimoff, 1967; Katz & Miledi, 1969; Miledi, 1973), the involvement of sodium ions in regulation of the release process — although evident since 1952 (Fatt & Katz, 1952) — is still not completely understood. Sodium ions, although cannot replace calcium in the release process it作者: 損壞 時(shí)間: 2025-3-24 08:04
Optical Studies of Excitation and Secretion in Vertebrate Nerve Terminalsrminals in the neurohypophyses of amphibia and mammals. Ca. and Na. contributions to the action potential are demonstrated and, in the mouse, variations in light scattering are detected following membrane potential changes known to trigger the release of peptide hormones. Calcium antagonists such as作者: 圍巾 時(shí)間: 2025-3-24 14:27 作者: 確定無疑 時(shí)間: 2025-3-24 16:40
A Channel View at Ca Inactivationess is greatly reduced when outward currents are minimized. Tail currents measured at return potentials where outward currents should be negligible have envelopes that follow the rise and fall of I. and the result is readily taken as evidence of inactivation of I. (9, see also 10).作者: 抱狗不敢前 時(shí)間: 2025-3-24 19:27
Some Consequences of Intracellular Calcium Binding on Phasic Synaptic Transmitter Release evolved from their suggestion (2) that a residuum of “active calcium” remains within the terminal following an action potential for several milliseconds and is able to enhance the amount of transmitter released by succeeding stimuli.作者: grudging 時(shí)間: 2025-3-25 01:46
https://doi.org/10.1007/978-3-322-97250-7f release results from an increase in intracellular Ca. concentrations near critical sites. Correspondingly, termination of release must result from a drop in Ca. concentration near these critical sites to levels insufficient to support release (5,6,7).作者: 波動(dòng) 時(shí)間: 2025-3-25 05:51
https://doi.org/10.1007/978-3-531-91917-1n, 1968; Colomo & Rahamimoff, 1968; Lev Tov & Rahamimoff, 1980). Both the influx of calcium ions into the depolarized neuronal axon and their extrusion from the axoplasm, are affected by extracellular sodium.作者: glacial 時(shí)間: 2025-3-25 11:26
On the Voltage Dependence of Neurotransmitter Releasef release results from an increase in intracellular Ca. concentrations near critical sites. Correspondingly, termination of release must result from a drop in Ca. concentration near these critical sites to levels insufficient to support release (5,6,7).作者: Yag-Capsulotomy 時(shí)間: 2025-3-25 15:13 作者: Antimicrobial 時(shí)間: 2025-3-25 18:54 作者: BOAST 時(shí)間: 2025-3-25 22:56 作者: 含水層 時(shí)間: 2025-3-26 03:00
Wilfried von Bredow,Thomas J?gere potential (e.p.p.), as well as the spontaneous quantal secretion of ACh, causing the miniature end-plate potentials (m.e.p.ps), depend on extra- and intracellular calcium concentrations (see 20; 28; 40).作者: 嘲弄 時(shí)間: 2025-3-26 04:29
Resting Calcium Levels and Evoked Release at the Neuromuscular Junction This single addition substantially alters the behavior of the model and makes it necessary to reinterpret many previous measurements. We have presented the mathematics for the model elsewhere (2, 3). Here we will give a more general description of our model, its application to experiments, and some directions for future research.作者: 征服 時(shí)間: 2025-3-26 10:33
Calcium Independent Quantal Transmitter Release at the Neuromuscular Junctione potential (e.p.p.), as well as the spontaneous quantal secretion of ACh, causing the miniature end-plate potentials (m.e.p.ps), depend on extra- and intracellular calcium concentrations (see 20; 28; 40).作者: FAZE 時(shí)間: 2025-3-26 14:59 作者: Influx 時(shí)間: 2025-3-26 17:06 作者: POINT 時(shí)間: 2025-3-27 00:18 作者: MUMP 時(shí)間: 2025-3-27 02:59
https://doi.org/10.1007/978-3-658-19608-0 properties similar to an intact cell, are more permeable to K than to Na (1) and release neurotransmitters in media containing calcium — provided depolarizing potassium concentrations or other depolarizing agents are present (1, 2, 3).作者: 悠然 時(shí)間: 2025-3-27 09:21 作者: Paradox 時(shí)間: 2025-3-27 12:56 作者: sterilization 時(shí)間: 2025-3-27 13:41 作者: 沒收 時(shí)間: 2025-3-27 21:18
Serendiptic Modulation of Transmitter Release: Extracellular Calcium Inhomogeneity origins for the calcium inhomogeneity may be extracellular matrix components and calcium transporters situated in the synaptic membranes facing the cleft; changes in their distributions and densities may provide the nerve terminal with energetically economical mechanisms for self-regulating synaptic transmission.作者: Peak-Bone-Mass 時(shí)間: 2025-3-27 23:36 作者: 闡釋 時(shí)間: 2025-3-28 06:10 作者: NAIVE 時(shí)間: 2025-3-28 07:26 作者: Arthritis 時(shí)間: 2025-3-28 12:14
https://doi.org/10.1007/978-3-658-09888-9 activated by depolarization of the presynaptic membrane. Direct evidence for this has been obtained by Llinas et al (1), in the giant synapse of the squid. Little is known, however, about the relationship between membrane currents and transmitter release in vertebrate endings whose small size preve作者: 棲息地 時(shí)間: 2025-3-28 17:07
https://doi.org/10.1007/978-3-658-19608-0corded in the postsynaptic cell comes from experiments performed on the neuromuscular synapse. This preparation which is most suitable for electrophysiological studies is unfortunately not adequate for biochemical experiments. Therefore, we are using synaptosomes from brain (intact or following osmo作者: famine 時(shí)間: 2025-3-28 21:04 作者: 減弱不好 時(shí)間: 2025-3-29 01:28 作者: wangle 時(shí)間: 2025-3-29 03:07
https://doi.org/10.1007/978-3-658-17570-2toreceptor, remain reduced for days after associatively conditioning the nudibranch mollusc .. Because intracellular calcium, Ca.. (as measured by differential absorption spectrophotometry of calcium complexed with the dye arsenazo III), undergoes prolonged elevation during the acquisition of the co作者: 褪色 時(shí)間: 2025-3-29 07:49 作者: 農(nóng)學(xué) 時(shí)間: 2025-3-29 13:56
Die Wohnungsgemeinnützigkeit in ?sterreichrnal medium via voltage-regulated cation-selective channels (reviews, 1, 2, 3). At the vertebrate neuromuscular junction this dependence is manifest in essentially the same way for release provoked by nerve impulses (i.e., end-plate potentials, EPPs) or release manifest as a raised frequency of mini作者: minimal 時(shí)間: 2025-3-29 16:01
Soziale Ungleichheit von Jugendlichen,nated subsequent work on this problem. We have modified their model by adding one additional parameter: the level of Ca in the resting nerve terminal. This single addition substantially alters the behavior of the model and makes it necessary to reinterpret many previous measurements. We have present作者: Galactogogue 時(shí)間: 2025-3-29 22:04
https://doi.org/10.1007/978-3-642-91120-0 in the extracellular solution (Jenkinson, 1957; Dodge & Rahamimoff, 1967). When the calcium concentration in the bathing solution is clamped at a constant level by employing a suitable calcium buffer, the average endplate potential amplitude and quantal content are reduced relative to those measure作者: forager 時(shí)間: 2025-3-30 02:09
Wilfried von Bredow,Thomas J?gerarization-transmitter secretion coupling to occur (29). Nerve impulse-evoked multi-quantal release of acetylcholine (ACh), giving rise to the end-plate potential (e.p.p.), as well as the spontaneous quantal secretion of ACh, causing the miniature end-plate potentials (m.e.p.ps), depend on extra- and作者: 干涉 時(shí)間: 2025-3-30 07:30 作者: 賭博 時(shí)間: 2025-3-30 10:40 作者: Fortify 時(shí)間: 2025-3-30 15:30
,Heute übliche elektrische Bremsverfahren,rminals in the neurohypophyses of amphibia and mammals. Ca. and Na. contributions to the action potential are demonstrated and, in the mouse, variations in light scattering are detected following membrane potential changes known to trigger the release of peptide hormones. Calcium antagonists such as作者: 接合 時(shí)間: 2025-3-30 20:12
https://doi.org/10.1007/978-1-4613-2307-5biology; brain; neurobiology; neurons; physiology; receptor作者: craven 時(shí)間: 2025-3-30 21:39
