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標(biāo)題: Titlebook: Biochemistry, Molecular Biology, and Physiology of Phospholipase A2 and Its Regulatory Factors; Anil B. Mukherjee Book 1990 The Editor(s) [打印本頁]

作者: 自由才謹(jǐn)慎    時間: 2025-3-21 19:55
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作者: 情感    時間: 2025-3-21 23:32

作者: surmount    時間: 2025-3-22 01:30

作者: preeclampsia    時間: 2025-3-22 04:54

作者: NOCT    時間: 2025-3-22 10:02

作者: MINT    時間: 2025-3-22 14:21

作者: RAFF    時間: 2025-3-22 20:04
Stimulation of Phospholipases A2 by Transglutaminases,m the enzymological point of view (1–4). Nevertheless, the physiological role(s) of these enzymes, particularly the intracellular TGs, are still poorly understood. TGs are a class of enzymes which catalyze a Ca.-dependent acyl-transfer reaction in which the γ-carboxamide group of a peptide-bound glu
作者: BANAL    時間: 2025-3-22 22:10
Functional Consequences of Phospholipase A2 Activation in Human Monocytes,etradecanoate phorbol-13-acetate, TPA), calcium ionophores (ionomycin), serum-treated zymosan (STZ) concanavalin A (Con A), and, to a minor degree, lipopolysaccharides (LPS). Protein Kinase C activation or increased intracellular Ca. are common features of the actions of most, if not all, of these s
作者: MINT    時間: 2025-3-23 01:52

作者: In-Situ    時間: 2025-3-23 08:45
Antiflammins Inhibit Synthesis of Platelet-Activating Factor and Intradermal Inflammatory Reactions. Lipocortins inhibit phospholipase A. (PLA.) activity in vitro with a mechanism still unclear (Davidson et al., 1987; Haigier et al., 1987). Furthermore, recombinant lipocortin I inhibits eicosanoid synthesis in vivo in perfused lungs (Cirino at al., 1987). Another steroid-induced protein with PLA.
作者: slow-wave-sleep    時間: 2025-3-23 13:38

作者: BLA    時間: 2025-3-23 14:36

作者: PLUMP    時間: 2025-3-23 18:45
Lipocortins as Antiphospholipase A2 and Anti-Inflammatory Proteins,ves a cascade of events leading to cellular activation, mediator formation, enzymatic cleavage… In such a complex network, involving so many different parameters, it seemed difficult to consider a common pathway. Anyhow, the empirical discovery of the anti-inflammatory action first of aspirin and th
作者: LATE    時間: 2025-3-24 01:59
Biology of Phospholipase Inhibitory Proteins, rheumatoid arthritis, lupus erythematosus and bronchial asthma. The mechanism of action is attributed to the anti-inflammatory action, immunosuppressive action and anti-edematous action.. Of these actions, the mode of anti-inflammation is now correlated to the inhibition of release of arachidonic a
作者: wangle    時間: 2025-3-24 02:59

作者: nautical    時間: 2025-3-24 07:49

作者: Atrium    時間: 2025-3-24 13:03
Soluble Phospholipase A2 in Human Pathology: Clinical-Laboratory Interface,s related to hemodynamic homeostasis, inflammatory reactions, phagocytic activity and others (1). Arachidonic acid cascade is initiated enzymatically by a group of phospholipases, phospholipase A. (PLA.) being one of the pivotal triggering enzymes. PLA. is located in at least 2 subcellular sites, as
作者: Facilities    時間: 2025-3-24 18:24
0065-2598 ticular. Progress in this field has been evident on many fronts, with novel information rapidly accumulating in the literature regarding the chemistry and molecular biology of this enzyme and its role in many important physiological processes. These include cellular signal transduction via the G-pro
作者: Mast-Cell    時間: 2025-3-24 22:49
https://doi.org/10.1007/978-3-642-60903-9 of the aggregated lipids. Our interests in phospholipase A. are focussed on the mechanism of the activation process and the physical basis of the role of lipid structure and/or dynamics in that process.
作者: Aqueous-Humor    時間: 2025-3-25 02:18
Molecular Aspects of Phospholipase A2 Activation, of the aggregated lipids. Our interests in phospholipase A. are focussed on the mechanism of the activation process and the physical basis of the role of lipid structure and/or dynamics in that process.
作者: enflame    時間: 2025-3-25 03:59

作者: Aphorism    時間: 2025-3-25 10:29
https://doi.org/10.1007/978-3-663-08665-9als (4–9). More recently, we detected UG in the circulation of the rabbit (10). The source of this protein in circulation seems to be the tracheobronchial epithelium and/or the progesterone-induced uterine endometrium (10).
作者: fetter    時間: 2025-3-25 15:23
Digitalisierung und Patientensicherheitstructure and enzymology of PLA., based on the paradigm of the pancreatic and snake venom enzymes, much remains to be learned regarding different cellular forms of PLA., their physiological roles, and how PLA. activity is regulated in cells.
作者: correspondent    時間: 2025-3-25 16:23
Probing the Mechanism of Pancreatic Phospholipase A2 with the Aid of Recombinant DNA Techniques,the digestive enzymes. To the cellular PLAs have been assigned regulatory functions (for a review see Waite, 1987), although certain cellular PLAs also exhibit a digestive role in the break-down of phagocytized materials (Elsbach and Weiss, 1988).
作者: Tonometry    時間: 2025-3-25 21:57
Inhibition of Phospholipase A2 by Uteroglobin and Antiflammin Peptides,als (4–9). More recently, we detected UG in the circulation of the rabbit (10). The source of this protein in circulation seems to be the tracheobronchial epithelium and/or the progesterone-induced uterine endometrium (10).
作者: Ingrained    時間: 2025-3-26 01:37

作者: 過去分詞    時間: 2025-3-26 06:00

作者: Ischemia    時間: 2025-3-26 11:39
Wulf-Dietrich Leber,Charlotte Vogt membrane bound and as cytosolic enzymes (2). Whereas the former acts intracellularly, the latter has the potential to be released from the cell upon a variety of stimuli (3). In humans, such soluble extracellular enzyme of pancreatic origin has indeed been found in the blood (4).
作者: 猛然一拉    時間: 2025-3-26 12:50

作者: 祖先    時間: 2025-3-26 20:18
MedR Schriftenreihe Medizinrechtes that hydrolyze the four ester bonds found in phospholipids. These enzymes are the phospholipases A., A., C, and D (2). Phospholipases A. and A. are responsible for cleaving the fatty acid ester bonds while phospholipases C and D cleave at the headgroup phosphoester bonds.
作者: 反抗者    時間: 2025-3-26 21:24

作者: IRS    時間: 2025-3-27 04:56

作者: FRET    時間: 2025-3-27 08:24
Soluble Phospholipase A2 in Human Pathology: Clinical-Laboratory Interface, membrane bound and as cytosolic enzymes (2). Whereas the former acts intracellularly, the latter has the potential to be released from the cell upon a variety of stimuli (3). In humans, such soluble extracellular enzyme of pancreatic origin has indeed been found in the blood (4).
作者: PATHY    時間: 2025-3-27 12:30

作者: 下垂    時間: 2025-3-27 16:22
Book 1990g endogenous and synthetic inhibitors and activators of this enzyme. To review these important areas in PLA2 research we invited scientists who made significant contributions in this field. The papers in this volume are organized to emphasize the recent advances in several areas of investigation, in
作者: Fortuitous    時間: 2025-3-27 20:06
A Novel Bifunctional Mechanism of Surface Recognition by Phospholipase A2,holipids lend themselves ideally to such studies in that they form a variety of stable and structurally well-defined surfaces in micelles, reverse micelles, monolayers, planar bilayers, and curved vesicles with which to study catalysis at the boundary between two phases.
作者: hemoglobin    時間: 2025-3-27 22:25
Stimulation of Phospholipases A2 by Transglutaminases, of inter or intramolecular covalent crosslinks. These enzymes have been detected both intra and extracellularly in higher animals including man. The best characterized extracellular TG is variously known as fibrin-stabilizing factor, Laki-Lorand factor or coagulation Factor XIII.
作者: Ophthalmologist    時間: 2025-3-28 05:55
Functional Consequences of Phospholipase A2 Activation in Human Monocytes,e agents..Release of arachidonic acid by the action of PLA. or other phospholipid hydrolyzing enzymes leads directly to the formation of cyclooxygenase products. In the presence of markedly elevated calcium concentrations, 5-lipoxygenase (LO) is activated as well, leading to the formation and releas
作者: 騙子    時間: 2025-3-28 08:54

作者: absorbed    時間: 2025-3-28 11:11

作者: Notify    時間: 2025-3-28 18:13
Lipocortins as Antiphospholipase A2 and Anti-Inflammatory Proteins,olipase A2 activation and/or with the subsequent formation of its derivatives. Phospholipase A2 is a cellular enzyme which, upon activation, cleaves phospholipids in the sn-2 position, giving rise to arachidonic acid, the usual unsaturated fatty acid located at this position in mammalian cells and t
作者: 尾隨    時間: 2025-3-28 19:01
Biology of Phospholipase Inhibitory Proteins,at the resulting complex is transferred into the nucleus to activate the specific genes, after all, inducing the synthesis of specific proteins.. Although the key enzymes and major sources of arachidonic acid still remain to be established in a quantitative aspect, phospholipase A. (PLA.) and alkylp
作者: 熱心    時間: 2025-3-29 02:38
Inhibition Of Human Phospholipases A, by ,-Unsaturated Fatty Acids and Oligomers Of Prostaglandin Buid, endometrium, degenerative disc, and snake venom was inhibited by PGB. with IC.S ranging from 0.5–7.0 μM. Inhibition was independent of substrate phospholipid concentration over a 24-fold range (5–120 μM) and PGB. quenched the tryptophan fluorescence of snake venom phospholipase A. in a dose-dep
作者: 改革運(yùn)動    時間: 2025-3-29 06:05

作者: 托運(yùn)    時間: 2025-3-29 07:40

作者: Limerick    時間: 2025-3-29 15:15
U. J. Winter,K. Blümling,G. V. Sabin of inter or intramolecular covalent crosslinks. These enzymes have been detected both intra and extracellularly in higher animals including man. The best characterized extracellular TG is variously known as fibrin-stabilizing factor, Laki-Lorand factor or coagulation Factor XIII.
作者: Aqueous-Humor    時間: 2025-3-29 18:19
https://doi.org/10.1007/978-3-663-08665-9e agents..Release of arachidonic acid by the action of PLA. or other phospholipid hydrolyzing enzymes leads directly to the formation of cyclooxygenase products. In the presence of markedly elevated calcium concentrations, 5-lipoxygenase (LO) is activated as well, leading to the formation and releas
作者: 流動才波動    時間: 2025-3-29 22:25

作者: 冒失    時間: 2025-3-30 03:41
https://doi.org/10.1007/978-3-662-58225-1iacylglycerol, from which arachidonate is subsequently released by a diacylglycerol lipase (Bell et al., 1979). However, it has become clear that in many cells arachidonate release can be readily separated from phosphatidylinositol metabolism (e.g., Burch et al., 1986; Burch and Axelrod, 1987; Slivk
作者: FLIP    時間: 2025-3-30 04:10

作者: Bravado    時間: 2025-3-30 08:20
Digitalisierung und Investitionsfinanzierungat the resulting complex is transferred into the nucleus to activate the specific genes, after all, inducing the synthesis of specific proteins.. Although the key enzymes and major sources of arachidonic acid still remain to be established in a quantitative aspect, phospholipase A. (PLA.) and alkylp
作者: HAWK    時間: 2025-3-30 15:25
Christopher Hermann,Nadia Mussauid, endometrium, degenerative disc, and snake venom was inhibited by PGB. with IC.S ranging from 0.5–7.0 μM. Inhibition was independent of substrate phospholipid concentration over a 24-fold range (5–120 μM) and PGB. quenched the tryptophan fluorescence of snake venom phospholipase A. in a dose-dep
作者: 亂砍    時間: 2025-3-30 18:23

作者: 現(xiàn)存    時間: 2025-3-30 20:48
0065-2598 who made significant contributions in this field. The papers in this volume are organized to emphasize the recent advances in several areas of investigation, in978-1-4612-7910-5978-1-4613-0651-1Series ISSN 0065-2598 Series E-ISSN 2214-8019
作者: 強(qiáng)化    時間: 2025-3-31 04:24
https://doi.org/10.1007/978-1-4613-0651-1DNA; Glutamin; Peptide; biochemistry; enzymes; molecular aspects; molecular biology; physiology; signal tran




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