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標(biāo)題: Titlebook: Atlas of Atherosclerosis; Risk Factors and Tre Peter W. F. Wilson (Professor of Medicine, Directo Book 2003Latest edition Springer Science+ [打印本頁(yè)]

作者: ABS    時(shí)間: 2025-3-21 17:27
書(shū)目名稱Atlas of Atherosclerosis影響因子(影響力)




書(shū)目名稱Atlas of Atherosclerosis影響因子(影響力)學(xué)科排名




書(shū)目名稱Atlas of Atherosclerosis網(wǎng)絡(luò)公開(kāi)度




書(shū)目名稱Atlas of Atherosclerosis網(wǎng)絡(luò)公開(kāi)度學(xué)科排名




書(shū)目名稱Atlas of Atherosclerosis被引頻次




書(shū)目名稱Atlas of Atherosclerosis被引頻次學(xué)科排名




書(shū)目名稱Atlas of Atherosclerosis年度引用




書(shū)目名稱Atlas of Atherosclerosis年度引用學(xué)科排名




書(shū)目名稱Atlas of Atherosclerosis讀者反饋




書(shū)目名稱Atlas of Atherosclerosis讀者反饋學(xué)科排名





作者: inclusive    時(shí)間: 2025-3-21 21:21
The Endothelium in Atherothrombosis,ogic functions of the vasculature. In response to biochemical and mechanical stimuli, endothelial cells synthesize and elaborate a number of factors that modulate vascular tone, inflammation, thrombosis, and vascular growth. Normal endothelium provides a fluid antiatherogenic environment that inhibi
作者: 啜泣    時(shí)間: 2025-3-22 03:39
The Macrophage and The Atherosclerotic Process, disease. Angiography performed at the time of acute myocardial infarction demonstrated that culprit lesions frequently caused only mild to moderate stenoses, not high-grade lesions that nearly obstructed the coronary arteries. Arteriographic regression trials that used lipid interventions to lower
作者: 蝕刻    時(shí)間: 2025-3-22 08:10

作者: DUST    時(shí)間: 2025-3-22 11:34
Triglyceride-Rich Lipoproteins,sis and increased levels of specific TG-carrying lipoproteins is complex and convoluted. Severalprimary and secondary disorders cause plasma triglycerideelevations. An increase in the risk of arteriosclerotic disease isobserved in some of these disorders. Risk appears more stronglyassociated with co
作者: 黃瓜    時(shí)間: 2025-3-22 16:42

作者: bromide    時(shí)間: 2025-3-22 18:41

作者: 埋伏    時(shí)間: 2025-3-22 22:14
The Newer Coronary Risk Factors,ing considered. This chapter provides a sampling of some of these “newer” risk factors, particularly those that have a biologic basis. The major categories to be considered are hematologic, newer lipid particles and their metabolic considerations, vitamins and homocysteine, and aspects of glucose me
作者: LUDE    時(shí)間: 2025-3-23 03:36

作者: 移動(dòng)    時(shí)間: 2025-3-23 08:31

作者: 珍奇    時(shí)間: 2025-3-23 09:53

作者: 無(wú)聊點(diǎn)好    時(shí)間: 2025-3-23 15:58

作者: 高度贊揚(yáng)    時(shí)間: 2025-3-23 19:02

作者: 恭維    時(shí)間: 2025-3-23 23:17

作者: compassion    時(shí)間: 2025-3-24 03:04
,Entwürfe und unausgeführte Pl?ne ?Warbeck?,ogic functions of the vasculature. In response to biochemical and mechanical stimuli, endothelial cells synthesize and elaborate a number of factors that modulate vascular tone, inflammation, thrombosis, and vascular growth. Normal endothelium provides a fluid antiatherogenic environment that inhibi
作者: Conduit    時(shí)間: 2025-3-24 09:30
,?über naive und sentimentalische Dichtung?, disease. Angiography performed at the time of acute myocardial infarction demonstrated that culprit lesions frequently caused only mild to moderate stenoses, not high-grade lesions that nearly obstructed the coronary arteries. Arteriographic regression trials that used lipid interventions to lower
作者: Muscularis    時(shí)間: 2025-3-24 10:59
https://doi.org/10.1007/978-3-476-04039-8n oil droplet formed from an outer layer of phospholipids, unesterified cholesterol, and proteins, with a core of neutral lipids, predominantly cholesterol ester and triglycerides [1]. Their main function is to transport lipids and lipid-soluble material throughout the body.
作者: 軟弱    時(shí)間: 2025-3-24 17:51
,Mannheim, Bauerbach, Dresden / 1782–1787,sis and increased levels of specific TG-carrying lipoproteins is complex and convoluted. Severalprimary and secondary disorders cause plasma triglycerideelevations. An increase in the risk of arteriosclerotic disease isobserved in some of these disorders. Risk appears more stronglyassociated with co
作者: 公司    時(shí)間: 2025-3-24 21:41
https://doi.org/10.1007/978-3-476-98904-8The association between diet and cardiovas-cular disease was established primarily by results from ecologic studies and studies of migrating populations. These studies have provided evidence that populations consuming diets high in saturated fat and cholesterol and low in fruits and vegetables have
作者: SOBER    時(shí)間: 2025-3-25 01:52
https://doi.org/10.1007/978-3-476-98904-8ention (no prior clinical event) and secondary prevention (prior clinical event) of coronary heart disease (CHD) is now firmly established [1–6]. The majority of evidence for clinical benefit exists for the prevention of CHD. However, these drugs are also used in patients with clinical evidence of c
作者: 結(jié)構(gòu)    時(shí)間: 2025-3-25 03:32

作者: 獨(dú)白    時(shí)間: 2025-3-25 10:54

作者: 漂浮    時(shí)間: 2025-3-25 15:28

作者: abject    時(shí)間: 2025-3-25 18:23
Os Magna Sonaturum Schillers Jugendlyrik,programs. Are resources better spent on treating cardiovascular disease or preventing it? Should programs be adopted that screen all persons for risk factors or should the efforts be targeted to certain patient groups? Should all risk factors receive equal attention or are resources better directed
作者: 彩色    時(shí)間: 2025-3-25 22:59
Peter W. F. Wilson (Professor of Medicine, DirectoEach chapter illustrates an important facet of diagnosing and treating this common and often fatal disease.Over 500 high quality images in step with the latest developments in medicine.World-renowned
作者: 曲解    時(shí)間: 2025-3-26 00:41
http://image.papertrans.cn/b/image/164075.jpg
作者: 效果    時(shí)間: 2025-3-26 04:38
Structure of the Plasma Lipoproteins,n oil droplet formed from an outer layer of phospholipids, unesterified cholesterol, and proteins, with a core of neutral lipids, predominantly cholesterol ester and triglycerides [1]. Their main function is to transport lipids and lipid-soluble material throughout the body.
作者: Commemorate    時(shí)間: 2025-3-26 08:33
The Newer Coronary Risk Factors,ing considered. This chapter provides a sampling of some of these “newer” risk factors, particularly those that have a biologic basis. The major categories to be considered are hematologic, newer lipid particles and their metabolic considerations, vitamins and homocysteine, and aspects of glucose metabolism.
作者: Nebulous    時(shí)間: 2025-3-26 16:27
https://doi.org/10.1007/978-1-4615-6484-3Lipoprotein; atherosclerosis; cardiovascular; high-density lipoprotein (HDL); vascular disease
作者: 較早    時(shí)間: 2025-3-26 17:10
Springer Science+Business Media New York 2003
作者: harpsichord    時(shí)間: 2025-3-26 23:23

作者: 帶來(lái)的感覺(jué)    時(shí)間: 2025-3-27 02:34
Low-density Lipoproteins in Atherogenesis,It is well established that high levels of plasma cholesterol, particularly those associated with low-density lipoprotein (LDL), increase the risk of developing atherosclerosis. It is also clear that lowering plasma cholesterol levels can arrest or even reverse the progression of the disease [1,2].
作者: Peculate    時(shí)間: 2025-3-27 08:12
s that are not have shown that fatty streak lesions are related to the lipoprotein cholesterol alter- very common in young adults. Recent ations, but stem from the pleiotropic effects research has extended thes978-1-4615-6484-3
作者: 出血    時(shí)間: 2025-3-27 12:30
Book 2003Latest editionct that therapy for as accident victims in more recent times dyslipidemia often has effects that are not have shown that fatty streak lesions are related to the lipoprotein cholesterol alter- very common in young adults. Recent ations, but stem from the pleiotropic effects research has extended thes
作者: 放逐    時(shí)間: 2025-3-27 13:37

作者: 燈絲    時(shí)間: 2025-3-27 20:07
Gender Differences in Coronary Risk Factors and Risk Interventions,nent, with increased case fatality rates and greater morbidity of women after both MI [2–4] and myocardial revascu- larization procedures [5]. Since 1984, more women than men die of cardiovascular disease in the United States. This underscores the need to undertake preventive interventions across the lifespan [4].
作者: 是突襲    時(shí)間: 2025-3-28 00:48

作者: 膽小懦夫    時(shí)間: 2025-3-28 03:24

作者: 新手    時(shí)間: 2025-3-28 10:14
Diabetes Mellitus and Vascular Disease Risk,nces between NIDDM and IDDM, the chronic sequelae of both forms of diabetes are the same, and vascular disease, particularly coronary heart disease (CHD), is the major cause of morbidity and mortality in diabetic patients.
作者: Deduct    時(shí)間: 2025-3-28 14:22

作者: 滋養(yǎng)    時(shí)間: 2025-3-28 16:17

作者: 草本植物    時(shí)間: 2025-3-28 18:59

作者: 豐滿中國(guó)    時(shí)間: 2025-3-28 23:59

作者: maverick    時(shí)間: 2025-3-29 03:35
,Entwürfe und unausgeführte Pl?ne ?Warbeck?,tions when homeostatic mechanisms are altered, the phenotypic changes that occur in endothelial cells support a vasospastic, prothrombotic, and proinflammatory milieu, and play a central role in the pathophysiology and clinical manifestations of cardiovascular disease [1].
作者: Alpha-Cells    時(shí)間: 2025-3-29 08:31

作者: LEVY    時(shí)間: 2025-3-29 15:15

作者: 里程碑    時(shí)間: 2025-3-29 16:35
The Endothelium in Atherothrombosis,tions when homeostatic mechanisms are altered, the phenotypic changes that occur in endothelial cells support a vasospastic, prothrombotic, and proinflammatory milieu, and play a central role in the pathophysiology and clinical manifestations of cardiovascular disease [1].
作者: Admire    時(shí)間: 2025-3-29 21:16
Triglyceride-Rich Lipoproteins,G-rich lipoproteins, commonly referred to as “remnant” lipoproteins. In addition, theassociation of hypertriglyceridemia with other risk factors, suchas diabetes mellitus and high blood pressure, causes linkage toatherosclerosis in epidemiologic studies to be less clear-cut.
作者: 鐵砧    時(shí)間: 2025-3-30 03:19

作者: 去掉    時(shí)間: 2025-3-30 04:31

作者: Legion    時(shí)間: 2025-3-30 08:16
Friedrich Nietzsches Naturbeflissenheit,o 1) proliferation of smooth muscle cells; 2) synthesis of connective tissue matrix; 3) focal accumulation of monocytes/macrophages; 4) lymphocytic infiltration; and 5) variable intracellular and extracellular lipid accumulation and eventually stenotic lesions [1].




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