標(biāo)題: Titlebook: Anti-Inflammatory Drugs in Asthma; Anthony P. Sampson,Martin K. Church Book 1999 Springer Basel AG 1999 Atmen.allergy.asthma.cytokine.rese [打印本頁] 作者: Traction 時(shí)間: 2025-3-21 20:08
書目名稱Anti-Inflammatory Drugs in Asthma影響因子(影響力)
書目名稱Anti-Inflammatory Drugs in Asthma影響因子(影響力)學(xué)科排名
書目名稱Anti-Inflammatory Drugs in Asthma網(wǎng)絡(luò)公開度
書目名稱Anti-Inflammatory Drugs in Asthma網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Anti-Inflammatory Drugs in Asthma被引頻次
書目名稱Anti-Inflammatory Drugs in Asthma被引頻次學(xué)科排名
書目名稱Anti-Inflammatory Drugs in Asthma年度引用
書目名稱Anti-Inflammatory Drugs in Asthma年度引用學(xué)科排名
書目名稱Anti-Inflammatory Drugs in Asthma讀者反饋
書目名稱Anti-Inflammatory Drugs in Asthma讀者反饋學(xué)科排名
作者: MELON 時(shí)間: 2025-3-21 23:30 作者: GRAVE 時(shí)間: 2025-3-22 03:17
,β2-Agonists, a coupling G-protein and adenylate cyclase, the effector enzyme [3] (Fig. 1). Cyclic AMP is able to modulate a number of processes which are important in governing the contractile state of the cell [4].作者: Anemia 時(shí)間: 2025-3-22 07:33
Anti-IgE Agents,to have a crucial role in the generation and persistence of symptoms in allergic diseases. The development of drugs that interfere with IgE production and function may therefore represent a more specific approach to treat these pathological conditions [1, 2].作者: 疼死我了 時(shí)間: 2025-3-22 10:41 作者: 物種起源 時(shí)間: 2025-3-22 13:22 作者: ICLE 時(shí)間: 2025-3-22 17:20 作者: interrogate 時(shí)間: 2025-3-23 01:09
Marta Mierendorff,Heinrich Tostorphonuclear cells, mast cells, and macrophages are present in an activated state and release proinflammatory mediators, cytokines and growth factors. From inflammation, a process of healing and repair may follow and this has been postulated to lead to remodeling of the airways.作者: decipher 時(shí)間: 2025-3-23 01:25
https://doi.org/10.1007/978-3-663-02006-6e numerous in the bronchial mucosa [2], submucosa and alveolar walls [3], and are even found free in the lumen [4]. The activation of mast cells by allergen or by other stimuli is associated with the rapid release of a range of potent mediators of inflammation and bronchoconstriction.作者: 車床 時(shí)間: 2025-3-23 08:10 作者: LATER 時(shí)間: 2025-3-23 11:36 作者: 蜿蜒而流 時(shí)間: 2025-3-23 15:49
Mast Cell Proteases as New Targets for Therapeutic Intervention in Asthma,e numerous in the bronchial mucosa [2], submucosa and alveolar walls [3], and are even found free in the lumen [4]. The activation of mast cells by allergen or by other stimuli is associated with the rapid release of a range of potent mediators of inflammation and bronchoconstriction.作者: ALIEN 時(shí)間: 2025-3-23 18:00
Einführung in die Kultur- und Kunst?konomiereased hospital admissions [2]. The reasons for this are unclear, but environmental factors such as indoor and outdoor air pollution and changes in lifestyle are considered to be amongst the contributing factors.作者: foliage 時(shí)間: 2025-3-24 00:14 作者: 搬運(yùn)工 時(shí)間: 2025-3-24 02:34 作者: 揮舞 時(shí)間: 2025-3-24 08:17
978-3-0348-9757-0Springer Basel AG 1999作者: HUMP 時(shí)間: 2025-3-24 14:27
Anti-Inflammatory Drugs in Asthma978-3-0348-8751-9Series ISSN 1422-7746 Series E-ISSN 2296-4525 作者: deriver 時(shí)間: 2025-3-24 17:30 作者: Allodynia 時(shí)間: 2025-3-24 21:05 作者: BRAND 時(shí)間: 2025-3-24 23:33 作者: 發(fā)怨言 時(shí)間: 2025-3-25 05:19 作者: 橫條 時(shí)間: 2025-3-25 10:40 作者: GENRE 時(shí)間: 2025-3-25 12:48
Marta Mierendorff,Heinrich Tostating and orchestrating this inflammatory response. Many types of cells are involved, the airway epithelium is shed; eosinophils, T lymphocytes, polymorphonuclear cells, mast cells, and macrophages are present in an activated state and release proinflammatory mediators, cytokines and growth factors.作者: 動(dòng)物 時(shí)間: 2025-3-25 17:36
Marta Mierendorff,Heinrich Tostulate allergen-specific B cells to produce IgE molecules via the release of IL-4/IL-13. The receptors for IgE are found on a multitude of different cells and two different types have been identified: the high-affinity receptor (FcεRI) on mast cells, basophils, and antigen-presenting cells, and the l作者: Immunoglobulin 時(shí)間: 2025-3-25 20:48 作者: 內(nèi)向者 時(shí)間: 2025-3-26 00:51
https://doi.org/10.1007/978-3-663-02006-6selectively inhibit the migration of these inflammatory cells into the lung. In asthma, airway CD4. Th2 type lymphocytes, mast cells and eosinophils appear to be primarily effector cells that underlie the clinical manifestations of disease [1]. The cellular and molecular mechanisms involved in the r作者: 有雜色 時(shí)間: 2025-3-26 06:10 作者: Aerophagia 時(shí)間: 2025-3-26 08:28 作者: phytochemicals 時(shí)間: 2025-3-26 13:40 作者: 未完成 時(shí)間: 2025-3-26 17:21
Phosphodiesterases in Asthma,ylaxis, e.g. receptor desensitization and post-receptor events. In particular, it was repeatedly demonstrated that continuous use of inhaled β.-agonists is associated with an impairment of their acute protective effects against bronchoconstrictive stimuli [1–4]. Continuous inhalation of β.-agonists 作者: LURE 時(shí)間: 2025-3-26 21:16 作者: Culpable 時(shí)間: 2025-3-27 03:09
Cytokine and Adhesion Molecule Antagonists,r extravasation and localisation to the site of inflammation [2, 3]. At the initiation of inflammation, cytokines and chemokines play key roles in propagating the inflammatory response by eliciting signals that activate adhesion-systems, induce the secretion of other cytokines/chemokines from the va作者: Optometrist 時(shí)間: 2025-3-27 06:17 作者: 斜坡 時(shí)間: 2025-3-27 12:01 作者: aggravate 時(shí)間: 2025-3-27 17:18
Einführung in die Kulturwissenschafteng. exercise), and thirdly, pathohistological parameters, particularly eosinophilic airway inflammation, resulting in structural changes within the airways (“airway remodelling”) [2, 3]. Asthma may occur in varying degrees of severity ranging from intermittent to severe persistent forms [1].作者: NADIR 時(shí)間: 2025-3-27 20:10
https://doi.org/10.1007/978-3-663-02006-6r extravasation and localisation to the site of inflammation [2, 3]. At the initiation of inflammation, cytokines and chemokines play key roles in propagating the inflammatory response by eliciting signals that activate adhesion-systems, induce the secretion of other cytokines/chemokines from the va作者: Fortuitous 時(shí)間: 2025-3-28 01:23 作者: 異端邪說下 時(shí)間: 2025-3-28 04:03
Anti-Inflammatory Drugs in Asthma: The Pathophysiology of Asthma,y induced. The underlying pathophysiological feature of asthma is increased airway responsiveness which develops on a basis of diffuse bronchial inflammation. The prevalence of asthma is increasing worldwide despite improved treatment which has resulted from a more comprehensive understanding of its作者: PAN 時(shí)間: 2025-3-28 07:23
Corticosteroids,Inhaled glucocorticoids have revolutionised asthma treatment and have now become the mainstay of therapy for patients with chronic disease [1]. There has recently been an enormous increase in our understanding of the molecular mechanisms whereby glucocorticoids suppress inflammation in asthma and th作者: Nefarious 時(shí)間: 2025-3-28 13:40 作者: 上下連貫 時(shí)間: 2025-3-28 18:11 作者: Heresy 時(shí)間: 2025-3-28 21:53
Anti-Leukotriene Therapy for Asthma,s), and characterised by a range of abnormalities on various levels. Early definitions of asthma stressed the aspects of variable lung function, reversible spontaneously or with treatment, but more recent definitions also place emphasis on the underlying airway inflammation that is thought to promot作者: 包租車船 時(shí)間: 2025-3-28 23:12
Immunomodulators,ating and orchestrating this inflammatory response. Many types of cells are involved, the airway epithelium is shed; eosinophils, T lymphocytes, polymorphonuclear cells, mast cells, and macrophages are present in an activated state and release proinflammatory mediators, cytokines and growth factors.作者: 大包裹 時(shí)間: 2025-3-29 05:48
Anti-IgE Agents,ulate allergen-specific B cells to produce IgE molecules via the release of IL-4/IL-13. The receptors for IgE are found on a multitude of different cells and two different types have been identified: the high-affinity receptor (FcεRI) on mast cells, basophils, and antigen-presenting cells, and the l作者: pancreas 時(shí)間: 2025-3-29 11:04
Mast Cell Proteases as New Targets for Therapeutic Intervention in Asthma,roughout the body, but are particularly prevalent in tissues which form an interface with the external environment. In the lower airways mast cells are numerous in the bronchial mucosa [2], submucosa and alveolar walls [3], and are even found free in the lumen [4]. The activation of mast cells by al
