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標題: Titlebook: Animal Models of Neurological Disease, I; Neurodegenerative Di Alan A. Boulton,Glen B. Baker,Roger F. Butterworth Book 1992 Springer Scienc [打印本頁]

作者: Arthur    時間: 2025-3-21 19:38
書目名稱Animal Models of Neurological Disease, I影響因子(影響力)




書目名稱Animal Models of Neurological Disease, I影響因子(影響力)學科排名




書目名稱Animal Models of Neurological Disease, I網絡公開度




書目名稱Animal Models of Neurological Disease, I網絡公開度學科排名




書目名稱Animal Models of Neurological Disease, I被引頻次




書目名稱Animal Models of Neurological Disease, I被引頻次學科排名




書目名稱Animal Models of Neurological Disease, I年度引用




書目名稱Animal Models of Neurological Disease, I年度引用學科排名




書目名稱Animal Models of Neurological Disease, I讀者反饋




書目名稱Animal Models of Neurological Disease, I讀者反饋學科排名





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Alan A. Boulton,Glen B. Baker,Roger F. Butterworth
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Tracing Housing Choices Over Timeease to date. We will review these models,which have evolved over a period of over fifty years and have provided us with powerful tools to understand the pathophysiology of Parkinson’s disease and to test new therapeutic approaches.
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Ethics of Animal Models of Neurological Diseases,ed. And in the animal room or laboratory, they are directly involved in ministering (providing veterinary care) to the animals being used, a vital aspect of which is the prevention and relief of pain and suffering. Veterinarians take the role of the “animal’s advocate” in this whole process.
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,Animal Models of Alzheimer’s Disease,d with AD; however, each model has been useful for the investigation of specific aspects of the disease. This chapter will outline the many experimental animal models of AD and compare the advantages and disadvantages of specific methodological approaches.
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0893-2336 iatric disorders. Because of increasing public concern over the ethical treatment of animals in research, we felt it incumbent upon us to include this general preface to these volumes in order to indicate why we think further research using animals is necessary and why animal models of psychi- ric a
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,Animal Models of Huntington’s Disease, in the US is approx 50/1,000,000 (.; .). Although HD may occur during the juvenile years (.); the manifestation of the disorder typically occurs in middle life, about 35–45 years of age. From the time of onset, an intractable course of mental deterioration and progressive motor abnormalities begins with death usually occurring within 15 years.
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,Rodent Models of Parkinson’s Disease,on of dopamine (DA) and its metabolites is the main neurochemical change in the disease, although other neurotransmitters such as norepinephrine (NE) and serotonin (5-hydroxy-tryptamine, 5-HT) are also reduced (.; .; .; .).
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Sample Selection and Endogeneity, in the US is approx 50/1,000,000 (.; .). Although HD may occur during the juvenile years (.); the manifestation of the disorder typically occurs in middle life, about 35–45 years of age. From the time of onset, an intractable course of mental deterioration and progressive motor abnormalities begins with death usually occurring within 15 years.
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Test of Hypotheses with Panel Data, either because of vulnerable periods in development leading to maturation of myelinating cells, including the myelination process, or because of unique aspects of myelin composition. . concerns general considerations in the selection and evaluation of an animal model.
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Econometric Analysis of Regional EconomyThe cerebellum is composed of the cerebellar cortex, internal white matter, and deep cerebellar nuclei. These nuclei are the fastigial, interpositus, and dentate nuclei; they mediate most of the output of the cerebellum. This output is directed primarily to motor regions of the brain stem and cerebral cortex.
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Animal Models of Neurological Disease, I978-1-59259-626-3Series ISSN 0893-2336 Series E-ISSN 1940-6045
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Gebhard Kirchg?ssner,Jürgen Woltersopagation of nerve impulses—known as saltatory conduction—without depolarization of adjacent nerve fibers (. chapter by Miller). The myelin sheath is a prerequisite for the development of complex nervous function, as indicated by the appearance of increasing amounts of compact myelin in the nervous
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,Animal Models of Huntington’s Disease, the disease was first described by George Huntington in 1872 as consisting of a progressive dementia coupled with bizarre uncontrollable movements and abnormal postures. HD is found in nearly all ethnic and racial groups with slight variations in prevalence rates. Overall, the prevalence rate of HD
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