標(biāo)題: Titlebook: Animal Models of Epilepsy; Methods and Innovati Scott C. Baraban Book 2009 The Editor(s) (if applicable) and The Author(s), under exclusive [打印本頁] 作者: 櫥柜 時(shí)間: 2025-3-21 18:01
書目名稱Animal Models of Epilepsy影響因子(影響力)
書目名稱Animal Models of Epilepsy影響因子(影響力)學(xué)科排名
書目名稱Animal Models of Epilepsy網(wǎng)絡(luò)公開度
書目名稱Animal Models of Epilepsy網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Animal Models of Epilepsy被引頻次
書目名稱Animal Models of Epilepsy被引頻次學(xué)科排名
書目名稱Animal Models of Epilepsy年度引用
書目名稱Animal Models of Epilepsy年度引用學(xué)科排名
書目名稱Animal Models of Epilepsy讀者反饋
書目名稱Animal Models of Epilepsy讀者反饋學(xué)科排名
作者: Daily-Value 時(shí)間: 2025-3-21 23:39
Val Roseg: A High Alpine Catchment,hat seizures elicited by acute convulsant treatment (i.e., pentylenetetrazole, PTZ) or knockdown of zebrafish homologs for human genetic epilepsy disorders (i.e., tuberous sclerosis complex, TSC) reliably evoke seizures in developing zebrafish. Using tectal or forebrain recordings, we also show that作者: delegate 時(shí)間: 2025-3-22 02:58
https://doi.org/10.1007/978-94-009-5524-0many neurological aspects of TSC. In this chapter I will review the clinical manifestations of TSC, molecular roles of the TSC genes, and current animal models of TSC. These and future models should facilitate the development of new and exciting discoveries that will have a meaningful impact on live作者: separate 時(shí)間: 2025-3-22 08:16 作者: 粗魯?shù)娜?nbsp; 時(shí)間: 2025-3-22 09:21
W. G. Beeftink,J. Rozema,A. H. L. Huiskes of BFNC has helped solidify the link between attenuated . . function and increased seizure susceptibility that results from . and . mutations. The mouse models described here have all significantly contributed to our understanding of how mutations in these genes might precipitate human epilepsy and作者: 憤怒歷史 時(shí)間: 2025-3-22 13:34
https://doi.org/10.1007/978-94-009-5524-0loss of specific subtypes of interneurons. Furthermore, electrophysiological studies have demonstrated that dentate gyrus granule cells from epileptic patients exhibit a functional reduction in inhibitory synaptic transmission (4). These clinical findings are consistent with extensive work in pharma作者: 松雞 時(shí)間: 2025-3-22 19:16
C. Ferrari,R. Gerdol,F. Piccolihe conventional means of recording epileptiform events has always been, and likely will continue to be, electrophysiological, but a careful calibration of imaging signals with respect to electrophysiological recordings can extend our data set immeasurably, providing new insights into old problems.作者: 外科醫(yī)生 時(shí)間: 2025-3-22 22:20 作者: dilute 時(shí)間: 2025-3-23 01:24
https://doi.org/10.3920/978-90-8686-744-8electrographic and behavioral seizure activity induced by peripheral kainic acid administration, prevents seizure-induced cell damage, and attenuates the seizure threshold in previously kindled animals. For NPY, AAV vector-mediated expression of prepro-neuropeptide Y delays seizure onset and increas作者: fidelity 時(shí)間: 2025-3-23 06:11
Tick – , interactions: burden or benefit? their role in epileptogenesis or associated cognitive dysfunction. Other research on neural stem or progenitor cells suggest that cell grafts may offer a strategy to repair epileptogenic damage or to deliver anticonvulsant treatments. Thus, both manipulating endogenous neural progenitors and neural作者: 分離 時(shí)間: 2025-3-23 12:34
The Nematode, ,, as an Emerging Model for Investigating Epilepsy,aracterizing the underlying defects associated with epilepsy via mammalian systems are not only laborious and time consuming but also costly. Because epilepsy represents a significant disease burden worldwide .1., there is a serious need to accelerate research by considering non-traditional approach作者: Solace 時(shí)間: 2025-3-23 17:57 作者: 榨取 時(shí)間: 2025-3-23 21:22
Modeling Tuberous Sclerosis Complex: Brain Development and Hyperexcitability,many neurological aspects of TSC. In this chapter I will review the clinical manifestations of TSC, molecular roles of the TSC genes, and current animal models of TSC. These and future models should facilitate the development of new and exciting discoveries that will have a meaningful impact on live作者: 閃光你我 時(shí)間: 2025-3-23 23:30
BK Potassium Channel Mutations Affecting Neuronal Function and Epilepsy,list of mutations causing epilepsy in humans and mice. BK channels have the specialized role of sharpening action potentials in many excitatory neurons. Surprisingly, functional analysis in heterologous expression systems indicates that some of these mutations are gain of function. This paradoxical 作者: landfill 時(shí)間: 2025-3-24 04:05 作者: cancellous-bone 時(shí)間: 2025-3-24 09:57 作者: Anticlimax 時(shí)間: 2025-3-24 14:28 作者: BILK 時(shí)間: 2025-3-24 17:39 作者: Arthropathy 時(shí)間: 2025-3-24 21:25 作者: 乏味 時(shí)間: 2025-3-25 02:52 作者: 鳴叫 時(shí)間: 2025-3-25 04:22
0893-2336 nd Innovations. is an ideal guide for scientists who wish to expand our understanding of the pathogenesis of this neurological disorder..978-1-4939-6114-6978-1-60327-263-6Series ISSN 0893-2336 Series E-ISSN 1940-6045 作者: GROSS 時(shí)間: 2025-3-25 08:07 作者: FLAT 時(shí)間: 2025-3-25 13:00 作者: 有說服力 時(shí)間: 2025-3-25 19:36 作者: FIN 時(shí)間: 2025-3-25 21:25 作者: 通知 時(shí)間: 2025-3-26 01:12 作者: 廢止 時(shí)間: 2025-3-26 06:36 作者: Inscrutable 時(shí)間: 2025-3-26 10:06
https://doi.org/10.1007/978-94-009-5524-0nd often very resistant to standard medical therapies. In addition, patients with TSC also frequently suffer from autism as well as mental retardation and various psychiatric disorders. The causative . and . genes were identified in the 1990s. Despite tremendous growth in our knowledge of the . gene作者: STALL 時(shí)間: 2025-3-26 12:53
https://doi.org/10.1007/978-94-009-5524-0studies are useful in observing the physiological events that may occur during epileptogenesis, the great complexity of changes that ensue between first seizure and epilepsy makes it difficult to ascribe cause-and-effect to any single protein. In contrast, spontaneous mutations or mutations using ge作者: arthroplasty 時(shí)間: 2025-3-26 18:15
W. G. Beeftink,J. Rozema,A. H. L. Huiskese the neuronal M-type potassium channel (M channel). While numerous studies have provided evidence for the inhibitory role of normally functioning M channels in key brain structures related to seizures and epileptogenesis, the BFNC sequelae from mutation to seizure and ultimately to remission is lik作者: Ossification 時(shí)間: 2025-3-26 23:07 作者: Arthritis 時(shí)間: 2025-3-27 04:29
C. Ferrari,R. Gerdol,F. Piccoliqual measure. We can now manipulate biological systems, both genetically and otherwise, to introduce fluorescent markers, literally adding colour to our preparations. These advances have occurred in parallel with remarkable developments in microscopy technology, with novel means of illumination and 作者: wangle 時(shí)間: 2025-3-27 08:34 作者: 品嘗你的人 時(shí)間: 2025-3-27 11:50
Tick – , interactions: burden or benefit? rearrangements much like the events that can lead to acquired temporal lobe epilepsy. Organotypic hippocampal slice cultures can be maintained for months in vitro and exhibit a latent period followed by onset of electrographic seizures involving the dentate granule cells, which is a hallmark of epi作者: Mri485 時(shí)間: 2025-3-27 17:06
Willem Takken,Constantianus J. M. Koenraadt monitoring. Though the duration of monitoring may vary from brief experiments over minutes to months of continuous recording, all recording of this nature requires a similar set of hardware and software tools. This chapter reviews the basic setup and requirements for successful continuous EEG monit作者: 太空 時(shí)間: 2025-3-27 20:58 作者: Detain 時(shí)間: 2025-3-27 22:31 作者: Incompetent 時(shí)間: 2025-3-28 02:30 作者: prostatitis 時(shí)間: 2025-3-28 06:15
Neuromethodshttp://image.papertrans.cn/a/image/157642.jpg作者: saturated-fat 時(shí)間: 2025-3-28 12:27
https://doi.org/10.1007/978-1-60327-263-6Drosophila; Mouse models; Neurological disorders; Seizure; Zebrafish; development作者: MAOIS 時(shí)間: 2025-3-28 16:23 作者: 倒轉(zhuǎn) 時(shí)間: 2025-3-28 20:04 作者: antedate 時(shí)間: 2025-3-29 01:50 作者: Transfusion 時(shí)間: 2025-3-29 03:05
The Albino , Tadpole as a Novel Model of Developmental Seizures,sequelae within the intact developing brain. This system allows in vivo imaging of neuronal circuit activity with single-cell resolution, as well as acute and long-term imaging of neuronal growth and synapse formation, within the intact unanesthetized brain. Mounting evidence supports a strong role 作者: medieval 時(shí)間: 2025-3-29 09:23 作者: Default 時(shí)間: 2025-3-29 14:00 作者: investigate 時(shí)間: 2025-3-29 16:36
BK Potassium Channel Mutations Affecting Neuronal Function and Epilepsy,studies are useful in observing the physiological events that may occur during epileptogenesis, the great complexity of changes that ensue between first seizure and epilepsy makes it difficult to ascribe cause-and-effect to any single protein. In contrast, spontaneous mutations or mutations using ge作者: nephritis 時(shí)間: 2025-3-29 21:52
Mouse Models of Benign Familial Neonatal Convulsions (BFNC): Mutations in , Genes,e the neuronal M-type potassium channel (M channel). While numerous studies have provided evidence for the inhibitory role of normally functioning M channels in key brain structures related to seizures and epileptogenesis, the BFNC sequelae from mutation to seizure and ultimately to remission is lik作者: Mri485 時(shí)間: 2025-3-30 01:18
