標(biāo)題: Titlebook: Alternate Programmed Cell Death Signaling in Antiviral Host Defense; Edward S. Mocarski,Pratyusha Mandal Book 2023 Springer Nature Switzer [打印本頁(yè)] 作者: BREED 時(shí)間: 2025-3-21 20:02
書目名稱Alternate Programmed Cell Death Signaling in Antiviral Host Defense影響因子(影響力)
書目名稱Alternate Programmed Cell Death Signaling in Antiviral Host Defense影響因子(影響力)學(xué)科排名
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書目名稱Alternate Programmed Cell Death Signaling in Antiviral Host Defense網(wǎng)絡(luò)公開度學(xué)科排名
書目名稱Alternate Programmed Cell Death Signaling in Antiviral Host Defense被引頻次
書目名稱Alternate Programmed Cell Death Signaling in Antiviral Host Defense被引頻次學(xué)科排名
書目名稱Alternate Programmed Cell Death Signaling in Antiviral Host Defense年度引用
書目名稱Alternate Programmed Cell Death Signaling in Antiviral Host Defense年度引用學(xué)科排名
書目名稱Alternate Programmed Cell Death Signaling in Antiviral Host Defense讀者反饋
書目名稱Alternate Programmed Cell Death Signaling in Antiviral Host Defense讀者反饋學(xué)科排名
作者: uncertain 時(shí)間: 2025-3-21 20:57
ZBP1/DAI-Dependent Cell Death Pathways in Influenza A Virus Immunity and Pathogenesis,but a subset of strains infects other avian as well as mammalian species, including humans. In aquatic birds, infection is largely restricted to the gastrointestinal tract and spread is through feces, while in humans and other mammals, respiratory epithelial cells are the primary sites supporting pr作者: 相容 時(shí)間: 2025-3-22 01:28 作者: 皺痕 時(shí)間: 2025-3-22 07:34 作者: 魔鬼在游行 時(shí)間: 2025-3-22 10:48 作者: LANCE 時(shí)間: 2025-3-22 14:58
Cell Killing by Reovirus: Mechanisms and Consequences,yocytes contributes to the pathogenesis of reovirus-induced encephalitis and myocarditis in a newborn mouse model. Thus, reovirus-induced cell death has been used to investigate the basis of viral disease. Depending on the cell type, infection of host cells by reovirus results in one of two forms of作者: 損壞 時(shí)間: 2025-3-22 17:42 作者: conscience 時(shí)間: 2025-3-22 22:16 作者: 寒冷 時(shí)間: 2025-3-23 05:26 作者: Anticoagulants 時(shí)間: 2025-3-23 06:54
Seth Goldstein MD, MPhil,F. Dylan Stewart MDtogether with other released components. Pyroptosis, like other forms of programmed cell death, helps eliminate infected cells and thereby restricts the replicative niche, undermining survival and proliferation of intracellular pathogens. This includes viruses as well as bacteria, where ample eviden作者: Celiac-Plexus 時(shí)間: 2025-3-23 11:55
Kevin M. Riggle MD,Kenneth W. Gow MD-containing proteins such as RIPK1. To ensure lifelong infection in the host, HSV carries out sophisticated molecular?strategies to evade host cell death responses during viral infection. HSV-1 is a well-characterized pathogen that encodes potent viral inhibitors that modulate both caspase activatio作者: BOOST 時(shí)間: 2025-3-23 14:08
Guy F. Brisseau MD, MEd, FAAP, FACS, FRCS(C) or variola virus manifests through subversion of MLKL activation. Recently described viral mimics of MLKL in other chordopoxviruses inhibit all three modes of necroptotic cell death. As with inhibition of apoptosis, the evolution of potentially redundant viral mechanisms to inhibit programmed necro作者: Lineage 時(shí)間: 2025-3-23 20:53 作者: Control-Group 時(shí)間: 2025-3-24 00:18
0070-217X oded inhibitor was shown to prevent the recruitment of RIPK3 (RIP3). This transformative evidence identified a novel pathway acting independent of TNF, interferon or RIPK1 that can stop virus from infecting its978-3-031-45280-2978-3-031-45278-9Series ISSN 0070-217X Series E-ISSN 2196-9965 作者: 軟膏 時(shí)間: 2025-3-24 04:55 作者: Congestion 時(shí)間: 2025-3-24 08:34 作者: 圓錐體 時(shí)間: 2025-3-24 11:41 作者: Mri485 時(shí)間: 2025-3-24 16:15 作者: 障礙物 時(shí)間: 2025-3-24 21:37
Manipulation of Host Cell Death Pathways by Herpes Simplex Virus,-containing proteins such as RIPK1. To ensure lifelong infection in the host, HSV carries out sophisticated molecular?strategies to evade host cell death responses during viral infection. HSV-1 is a well-characterized pathogen that encodes potent viral inhibitors that modulate both caspase activatio作者: Traumatic-Grief 時(shí)間: 2025-3-25 02:02
Subversion of Programed Cell Death by Poxviruses, or variola virus manifests through subversion of MLKL activation. Recently described viral mimics of MLKL in other chordopoxviruses inhibit all three modes of necroptotic cell death. As with inhibition of apoptosis, the evolution of potentially redundant viral mechanisms to inhibit programmed necro作者: 沖擊力 時(shí)間: 2025-3-25 04:04 作者: LOPE 時(shí)間: 2025-3-25 10:15
Alternate Programmed Cell Death Signaling in Antiviral Host Defense作者: 錯(cuò) 時(shí)間: 2025-3-25 13:21 作者: 面包屑 時(shí)間: 2025-3-25 17:10 作者: 騷擾 時(shí)間: 2025-3-25 21:43
Book 2023es into the molecular functions and regulation of necroptosis and discusses how viruses induce and manipulate this potent innate cellular sensing system...Initially, understanding of necroptosis emerged from studies on tumor necrosis factor (TNF) signaling that showed the key role of receptor intera作者: LIMN 時(shí)間: 2025-3-26 01:59
0070-217X mammals.Sheds new light on additional features of the signa.This volume provides a comprehensive review of programmed cell death pathways and their fundamental role in antiviral host defense. The book deep-dives into the molecular functions and regulation of necroptosis and discusses how viruses in作者: harrow 時(shí)間: 2025-3-26 06:08 作者: 類人猿 時(shí)間: 2025-3-26 08:50
Current Topics in Microbiology and Immunologyhttp://image.papertrans.cn/a/image/154017.jpg作者: Deject 時(shí)間: 2025-3-26 15:11
Alternate Programmed Cell Death Signaling in Antiviral Host Defense978-3-031-45278-9Series ISSN 0070-217X Series E-ISSN 2196-9965 作者: Omnipotent 時(shí)間: 2025-3-26 19:42
Psychological and Clinical Issues,se. Evolution affords mammals with a wide variety of independent immune mechanisms to control or eliminate invading infectious agents. Many pathogens acquire functions to deflect these immune mechanisms and promote infection. Following successful invasion of a host, cell autonomous signaling pathway作者: 蝕刻術(shù) 時(shí)間: 2025-3-26 21:47 作者: 轉(zhuǎn)換 時(shí)間: 2025-3-27 04:30 作者: 闡釋 時(shí)間: 2025-3-27 06:39
Kevin M. Riggle MD,Kenneth W. Gow MDion in sensory neurons of the peripheral nervous system. HSV-1 causes oral cold sores, and HSV-2 causes genital lesions characterized by recurrence at the site of the initial infection. In multicellular organisms, cell death plays a pivotal role in host?defense by eliminating pathogen-infected cells作者: MORT 時(shí)間: 2025-3-27 10:33 作者: Agility 時(shí)間: 2025-3-27 14:19 作者: 滴注 時(shí)間: 2025-3-27 21:38
William G. Finn,Steven H. Kroftnd the molecular mechanisms that govern protective versus pathological neuroinflammatory responses to infection are a matter of intense investigation. Recent evidence suggests that necroptosis, an immunogenic form of programmed cell death, may contribute to the pathogenesis of viral encephalitis. Ho作者: AVOID 時(shí)間: 2025-3-28 01:21 作者: Fibrinogen 時(shí)間: 2025-3-28 03:34 作者: Annotate 時(shí)間: 2025-3-28 08:38
Book 2021iameter chimneys are introduced showing cold inflow phenomena that significantly reduced the performance of solar chimney. The cold inflow-free operation of solar chimneys restores the draft losses and enhances the performance of the solar chimneys. Numerical and experimental investigation results w
